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1.
2.
D. Habedank I. Reindl G. Vietzke U. Bauer A. Sperfeld S. Gl?ser K. D. Wernecke F. X. Kleber 《European journal of applied physiology and occupational physiology》1998,77(5):421-426
The ventilatory equivalent for CO2 defines ventilatory efficiency largely independent of metabolism. An impairment of ventilatory efficiency may be caused by
an increase in either anatomical or physiological dead space, the latter being the most important mechanism in the hyperpnoea
of heart failure, pulmonary embolism, pulmonary hypertension and the former in restrictive lung disease. However, normal values
for ventilatory efficiency have not yet been established. We investigated 101 (56 men) healthy volunteers, aged 16–75 years,
measuring ventilation and gas exchange at rest (n = 64) and on exercise (modified Naughton protocol, n = 101). Age and sex dependent normal values for ventilatory efficiency at rest defined as the ratio ventilation:carbon dioxide
output (V˙
E:V˙CO2), exercise ventilatory efficiency during exercise, defined as the slope of the linear relationship between ventilation and
carbon dioxide output (V˙
E vs V˙CO2 slope), oxygen uptake at the anaerobic threshold and at maximum (V˙O2AT,V˙O2max, respectively) and breathing reserve were established. Ventilatory efficiency at rest was largely independent of age, but
was smaller in the men than in the women [V˙
E:V˙CO2 50.5 (SD 8.8) vs 57.6 (SD 12.6) P<0.05]. Ventilatory efficiency during exercise declined significantly with age and was smaller in the men than in the women
(men: (V˙
E vs V˙CO2 slope = 0.13 × age + 19.9; women: V˙
E vs V˙CO2 slope = 0.12 × age + 24.4). The V˙O2AT and V˙O2max were 23 (SD 5) and 39 (SD 7) ml O2 · kg · min−1 in the men and 18 (SD 4) and 32 (SD 7) in the women, respectively, and declined significantly with age. The V˙O2AT was reached at 58 (SD 9)% V˙O2max. Breathing reserve at the end of exercise was 41% and was independent of sex and age. It was concluded from this study that
ventilatory efficiency as well as peak oxygen uptake are age and sex dependent in adults.
Accepted: 11 June 1997 相似文献
3.
Thierry Weissland P. Pelayo Jacques Vanvelcenaher Ga?lle Marais Jean-Marc Lavoie Hervé Robin 《European journal of applied physiology and occupational physiology》1997,76(5):428-433
The aims of the present study were: first, to assess the interindividual variations of a spontaneously chosen crank rate
(SCCR) in relation to the power developed during an incremental upper body exercise on an arm ergometer set at a constant
power regime, and second, to compare heart rate (HR) responses, expired minute ventilation (V˙
E) and oxygen consumption (V˙O2) when the pedal rates were chosen spontaneously (TSCCR) or set at ±10% of the freely chosen rates (T+10% and T−10%, respectively). The mean pedal rate values were linearly related (P < 0.01) with the power developed during arm cranking (r = 0.96), although large variations of pedalling rate strategies were observed between subjects. Maximal power (MP) and time
to exhaustion values were significantly higher (P < 0.05) during TSCCR than during T+10% and T−10%. Peak V˙O2 values were significantly higher (P < 0.05) in T+10% than in TSCCR and T−10%. The increase in HR, V˙
E, and V˙O2 mean values, in relation to the increase in the power developed, was significantly higher (P < 0.05) when the pedal rate was set at plus 10% of the SCCR (T±10%) than in the two other conditions. The findings of the present study suggest that the use of an electromagnetically braked
ergometer, which automatically adjusts the resistance component to maintain a constant work rate, should be used in order
to achieve the highest MP values during an incremental upper body exercise. A 10% increase of the SCCR should be used in order
to provide the highest peak V˙O2 value.
Accepted: 5 May 1997 相似文献
4.
Greg J. Wilson Andrew D. Walshe Mark R. Fisher 《European journal of applied physiology and occupational physiology》1997,75(5):455-461
The aims of the present study were: (1) to assess aerobic metabolism in paraplegic (P) athletes (spinal lesion level, T4–L3)
by means of peak oxygen uptake (V˙O2peak) and ventilatory threshold (VT), and (2) to determine the nature of exercise limitation in these athletes by means of cardioventilatory
responses at peak exercise. Eight P athletes underwent conventional spirographic measurements and then performed an incremental
wheelchair exercise on an adapted treadmill. Ventilatory data were collected every minute using an automated metabolic system:
ventilation (l · min−1), oxygen uptake (V˙O2, l · min−1, ml · min−1 · kg−1), carbon dioxide production (V˙CO2, ml · min−1), respiratory exchange ratio, breathing frequency and tidal volume. Heart rate (HR, beats · min−1) was collected with the aid of a standard electrocardiogram. V˙O2peak was determined using conventional criteria. VT was determined by the breakpoint in the V˙CO2−V˙O2 relationship, and is expressed as the absolute VT (V˙O2, ml · min−1 · kg−1) and relative VT (percentage of V˙O2peak). Spirometric values and cardioventilatory responses at rest and at peak exercise allowed the measurement of ventilatory
reserve (VR), heart rate reserve (HRr), heart rate response (HRR), and O2 pulse (O2 P). Results showed a V˙O2peak value of 40.6 (2.5) ml · min−1 · kg−1, an absolute VT detected at 23.1 (1.5) ml · min−1 · kg−1
V˙O2 and a relative VT at 56.4 (2.2)% V˙O2peak. HRr [15.8 (3.2) beats · min−1], HRR [48.6 (4.3) beat · l−1], and O2 P [0.23 (0.02) ml · kg−1 · beat−1] were normal, whereas VR at peak exercise [42.7 (2.4)%] was increased. As wheelchair exercise excluded the use of an able-bodied
(AB) control group, we compared our V˙O2peak and VT results with those for other P subjects and AB controls reported in the literature, and we compared our cardioventilatory
responses with those for respiratory and cardiac patients. The low V˙O2peak values obtained compared with subject values obtained during an arm-crank exercise may be due to a reduced active muscle
mass. Absolute VT was somewhat comparable to that of AB subjects, mainly due to the similar muscle mass involved in wheelchair
and arm-crank exercise by P and AB subjects, respectively. The increased VR, as reported in patients with chronic heart failure,
suggested that P athletes exhibited cardiac limitation at peak exercise, and this contributed to the lower V˙O2peak measured in these subjects.
Accepted: 22 April 1997 相似文献
5.
Hiroshi Takaki Kenji Sunagawa Masaru Sugimachi Yasushi Hara Toru Kawada Takashi Kurita Yoichi Goto 《European journal of applied physiology and occupational physiology》1998,78(4):333-339
The transient response of oxygen uptake (V˙O2) to submaximal exercise, known to be abnormal in patients with cardiovascular disorders, can be useful in assessing the functional
status of the cardiocirculatory system, however, a method for evaluating it accurately has not yet been established. As an
alternative approach to the conventional test at constant exercise intensity, we applied a random stimulus technique that
has been shown to provide relatively noise immune responses of system being investigated. In 27 patients with heart failure
and 24 age-matched control subjects, we imposed cycle exercise at 50 W intermittently according to a pseudo-random binary
(exercise-rest) sequence, while measuring breath-by-breath V˙O2. After determining the transfer function relating exercise intensity (W˙) to V˙O2 and attenuating the high frequency ranges (>6 exercise-rest cycles · min−1), we computed the high resolution band-limited (0–6 cycles · min−1) V˙O2 response (0–120 s) to a hypothetical step exercise. The V˙O2 response showed a longer time constant in the patients than in the control subjects [47 (SD 37) and 31 (SD 8) s, respectively,
P < 0.05]. Furthermore, the amplitude of the V˙O2 response after the initial response was shown to be significantly smaller in the patients than in the control subjects [176
(SD 50) and 267 (SD 54) ml · min−1 at 120 s]. The average amplitude over 120 s correlated well with peak V˙O2 (r = 0.73) and ΔV˙O2/ΔW˙ (r = 0.70), both of which are well-established indexes of exercise tolerance. The data indicated that our band-limited V˙O2
step response using random exercise was more markedly attenuated and delayed in the patients with heart failure than in the normal controls
and that it could be useful in quantifying the overall functional status of the cardiocirculatory system.
Accepted: 6 January 1998 相似文献
6.
Jerzy A. Zoladz Krzysztof Duda Joanna Majerczak 《European journal of applied physiology and occupational physiology》1998,77(5):445-451
A group of 12 healthy non-smoking men [mean age 22.3 (SD 1.1) years], performed an incremental exercise test. The test started
at 30 W, followed by increases in power output (P) of 30 W every 3 min, until exhaustion. Blood samples were taken from an antecubital vein for determination of plasma concentration
lactate [La−]pl and acid-base balance variables. Below the lactate threshold (LT) defined in this study as the highest P above which a sustained increase in [La−]pl was observed (at least 0.5 mmol · l−1 within 3 min), the pulmonary oxygen uptake (V˙O2) measured breath-by-breath, showed a linear relationship with P. However, at P above LT [in this study 135 (SD 30) W] there was an additional accumulating increase in V˙O2 above that expected from the increase in P alone. The magnitude of this effect was illustrated by the difference in the final P observed at maximal oxygen uptake (V˙O2max) during the incremental exercise test (P
max,obs at V˙O2max) and the expected power output at V˙O2max(P
max,exp at V˙O2max) predicted from the linear V˙O2-P relationship derived from the data collected below LT. The P
max,obs at V˙O2max amounting to 270 (SD 19) W was 65.1 (SD 35) W (19%) lower (P<0.01) than the P
max,exp at V˙O2max
. The mean value of V˙O2max reached at P
max,obs amounted to 3555 (SD 226) ml · min−1 which was 572 (SD 269) ml · min−1 higher (P<0.01) than the V˙O2 expected at this P, calculated from the linear relationship between V˙O2 and P derived from the data collected below LT. This fall in locomotory efficiency expressed by the additional increase in V˙O2, amounting to 572 (SD 269) ml O2 · min−1, was accompanied by a significant increase in [La−]pl amounting to 7.04 (SD 2.2) mmol · l−1, a significant increase in blood hydrogen ion concentration ([H+]b) to 7.4 (SD 3) nmol · l−1 and a significant fall in blood bicarbonate concentration to 5.78 (SD 1.7) mmol · l−1, in relation to the values measured at the P of the LT. We also correlated the individual values of the additional V˙O2 with the increases (Δ) in variables [La−]pl and Δ[H+]b. The Δ values for [La−]pl and Δ[H+]b were expressed as the differences between values reached at the P
max,obs at V˙O2max and the values at LT. No significant correlations between the additional V˙O2 and Δ[La−]pl on [H+]b were found. In conclusion, when performing an incremental exercise test, exceeding P corresponding to LT was accompanied by a significant additional increase in V˙O2 above that expected from the linear relationship between V˙O2 and P occurring at lower P. However, the magnitude of the additional increase in V˙O2 did not correlate with the magnitude of the increases in [La−]pl and [H+]b reached in the final stages of the incremental test.
Accepted: 30 October 1997 相似文献
7.
Jerzy A. Zoladz Zbigniew Szkutnik Joanna Majerczak Krzysztof Duda 《European journal of applied physiology and occupational physiology》1998,78(4):369-377
The purpose of this study was to develop a method to determine the power output at which oxygen uptake (V˙O2) during an incremental exercise test begins to rise non-linearly. A group of 26 healthy non-smoking men [mean age 22.1 (SD
1.4) years, body mass 73.6 (SD 7.4) kg, height 179.4 (SD 7.5) cm, maximal oxygen uptake (V˙O2max) 3.726 (SD 0.363) l · min−1], experienced in laboratory tests, were the subjects in this study. They performed an incremental exercise test on a cycle
ergometer at a pedalling rate of 70 rev · min−1. The test started at a power output of 30 W, followed by increases amounting to 30 W every 3 min. At 5 min prior to the first
exercise intensity, at the end of each stage of exercise protocol, blood samples (1 ml each) were taken from an antecubital
vein. The samples were analysed for plasma lactate concentration [La]pl, partial pressure of O2 and CO2 and hydrogen ion concentration [H+]b. The lactate threshold (LT) in this study was defined as the highest power output above which [La−]pl showed a sustained increase of more than 0.5 mmol · l−1 · step−1. The V˙O2 was measured breath-by-breath. In the analysis of the change point (CP) of V˙O2 during the incremental exercise test, a two-phase model was assumed for the 3rd-min-data of each step of the test: X
i
=at
i
+b+ɛ
i
for i=1,2,…,T, and E(X
i
)>at
i
+b for i =T+1,…,n, where X
1, … , X
n
are independent and ɛ
i
∼N(0,σ2). In the first phase, a linear relationship between V˙O2 and power output was assumed, whereas in the second phase an additional increase in V˙O2 above the values expected from the linear model was allowed. The power output at which the first phase ended was called the
change point in oxygen uptake (CP-V˙O2). The identification of the model consisted of two steps: testing for the existence of CP and estimating its location. Both
procedures were based on suitably normalised recursive residuals. We showed that in 25 out of 26 subjects it was possible
to determine the CP-O2 as described in our model. The power output at CP-V˙O2 amounted to 136.8 (SD 31.3) W. It was only 11 W – non significantly – higher than the power output corresponding to LT. The
V˙O2 at CP-V˙O2 amounted to 1.828 (SD 0.356) l · min−1 was [48.9 (SD 7.9)% V˙O2
max
]. The [La−]pl at CP-V˙O2, amounting to 2.57 (SD 0.69) mmol · l−1 was significantly elevated (P<0.01) above the resting level [1.85 (SD 0.46) mmol · l−1], however the [H+]b at CP-V˙O2 amounting to 45.1 (SD 3.0) nmol · l−1, was not significantly different from the values at rest which amounted to 44.14 (SD 2.79) nmol · l−1. An increase of power output of 30 W above CP-V˙O2 was accompanied by a significant increase in [H+]b above the resting level (P=0.03).
Accepted: 25 March 1998 相似文献
8.
This study investigated the effects on running economy (RE) of ingesting either no fluid or an electrolyte solution with
or without 6% carbohydrate (counterbalanced design) during 60-min running bouts at 80% maximal oxygen consumption (V˙O2max). Tests were undertaken in either a thermoneutral (22–23°C; 56–62% relative humidity, RH) or a hot and humid natural environment
(Singapore: 25–35°C; 66–77% RH). The subjects were 15 young adult male Singaporeans [V˙O2max = 55.5 (4.4 SD) ml kg−1 min−1]. The RE was measured at 3 m s−1 [65 (6)% V˙O2max] before (RE1) and after each prolonged run (RE2). Fluids were administered every 2 min, at an individual rate determined
from prior tests, to maintain body mass (group mean = 17.4 ml min−1). The V˙O2 during RE2 was higher (P < 0.05) than that during the RE1 test for all treatments, with no differences between treatments (ANOVA). The mean increase
in V˙O2 from RE1 to RE2 ranged from 3.4 to 4.7 ml kg−1 min−1 across treatments. In conclusion, the deterioration in RE at 3 m s−1 (65% V˙O2max) after 60 min of running at 80% V˙O2max appears to occur independently of whether fluid is ingested and regardless of whether the fluid contains carbohydrates or
electrolytes, in both a thermoneutral and in a hot, humid environment.
Accepted: 30 October 1997 相似文献
9.
A. S. Weller C. E. Millard P. L. Greenhaff I. A. Macdonald 《European journal of applied physiology and occupational physiology》1998,77(3):217-223
In a previous study, rectal temperature (T
re) was found to be lower, and oxygen consumption (V˙O2) and the respiratory exchange ratio (R) were higher in a cold (+5°C), wet and windy environment (COLD), compared with a thermoneutral environment during intermittent
walking at ≈30% of peak V˙O2 (Weller AS, Millard CE, Stroud MA et al. Am J Physiol 272:R226–R233, 1997). The aim of the present study was to establish
whether these cold-induced responses are influenced by prior fasting, as impaired thermoregulation has been demonstrated in
cold-exposed, resting men following a 48-h fast. To address this question, eight men attempted a 360-min intermittent (15 min
rest, 45 min exercise) walking protocol under COLD conditions on two occasions. In one condition, the subjects started the
exercise protocol ≈120 min after a standard meal (FED/COLD), whereas in the other the subjects had fasted for 36 h (FASTED/COLD).
The first two exercise periods were conducted at a higher intensity (HIGHER, 6 km · h−1 and 10% incline), than the four subsequent exercise periods (LOW, 5 km · h−1 and 0% incline). There was no difference in the time endured in FED/COLD and FASTED/COLD. In FASTED/COLD com pared with FED/COLD,
R was lower during HIGHER and LOW, and T
re was lower during LOW, whereas there was no difference in V˙O2, mean skin temperature and heart rate. Therefore, although the 36-h fast impaired temperature regulation during intermittent
low-intensity exercise in the cold, wet and windy environment, it was unlikely to have been the principal factor limiting
exercise performance under these experimental conditions.
Accepted: 26 August 1997 相似文献
10.
Fifteen young adult Singaporean male physical education students maximum oxygen consumption [(V˙O2max) = 56 (4.7) ml · kg−1 · min−1] performed three prolonged runs in a counterbalanced design. The running bouts varied in time (40 vs 60 min) and intensity
(70% vs 80% V˙O2
max
). Each prolonged run was separated by 7 days. The running economy (RE) at 10.8 km · h−1 during 10-min running bouts was measured before (RE1) and after (RE2) each prolonged run. A control study involved monitoring
RE at 10.8 km · h−1 before and after 60 min rest. There were no differences between RE1 and RE2 values during the control run. However, there
were differences between RE1 and RE2 values when separated by a prolonged run. For example, the mean (SD) changes in oxygen
consumption (ml · kg−1 · min−1) values were 38.2 (2.5) versus 40.1 (2.6) (40 min at 80% V˙O2
max
), 38.9 (2.8) versus 41.5 (2.6) (60 min at 70% V˙O2
max
), and 39.0 (3.1) versus 42.7 (2.9) (60 min at 80% V˙O2
max
; P < 0.01). The results of this investigation support the hypothesis that RE deteriorates during prolonged running, and that
the magnitude of the deterioration in RE increases with both increasing exercise intensity and duration.
Accepted: 14 July 1997 相似文献
11.
M. Faina V. Billat R. Squadrone M. De Angelis J. P. Koralsztein A. Dal Monte 《European journal of applied physiology and occupational physiology》1997,76(1):13-20
Using 23 elite male athletes (8 cyclists, 7 kayakists, and 8 swimmers), the contribution of the anaerobic energy system to
the time to exhaustion (t
lim) at the minimal exercise intensity (speed or power) at which maximal oxygen uptake (V˙O2
max) occurs (I
V˙O2
max) was assessed by analysing the relationship between the t
lim and the accumulated oxygen deficit (AOD). After 10-min warming up at 60% of V˙O2
max, the exercise intensity was increased so that each subject reached his I
V˙O2max
in 30 s and then continued at that level until he was exhausted. Pre-tests included a continuous incremental test with 2 min
steps for determining the I
V˙O2max
and a series of 5-min submaximal intensities to collect the data that would allow the estimation of the energy expenditure
at I
V˙O2max
. The AOD for the t
lim exercise was calculated as the difference between the above estimation and the accumulated oxygen uptake. The mean percentage
value of energy expenditure covered by anaerobic metabolism was 15.2 [(SD 6)%, range 8.9–24.1] with significant differences
between swimmers and kayakists (16.8% vs 11.5%, P≤0.05) and cyclists and kayakists (16.4% vs 11.5%, P≤0.05). Absolute AOD values ranged from 26.4 ml · kg−1 to 83.6 ml · kg−1 with a mean value of 45.9 (SD 18) ml · kg−1. Considering all the subjects, the t
lim was found to have a positive and significant correlation with AOD (r = 0.62, P≤0.05), and a negative and significant correlation with V˙O2
max (r = −0.46, P≤0.05). The data would suggest that the contribution of anaerobic processes during exercise performed at I
V˙O2max
should not be ignored when t
lim is used as a supplementary parameter to evaluate specific adaptation of athletes.
Accepted: 17 December 1996 相似文献
12.
M. Sandsund M. Sue-Chu J. Helgerud R. E. Reinertsen L. Bjermer 《European journal of applied physiology and occupational physiology》1998,77(4):297-304
The effects of whole-body exposure to ambient temperatures of −15°C and 23°C on selected performance-related physiological
variables were investigated in elite nonasthmatic cross-country skiers. At an ambient temperature of −15°C we also studied
the effects of the selective β2-adrenergic agonist Salbutamol (0.4 mg × 3) which was administered 10 min before the exercise test. Eight male cross-country
skiers with known maximal oxygen uptakes (V˙O2
max
) of more than 70 ml · kg−1 · min−1 participated in the study. Oxygen uptake (V˙O2), heart rate (f
c), blood lactate concentration ([La−]b) and time to exhaustion were measured during controlled submaximal and maximal running on a treadmill in a climatic chamber.
Lung function measured as forced expiratory volume in 1 s (FEV1) was recorded immediately before the warm-up period and at the conclusion of the exercise protocol. Submaximal V˙O2 and [La−]b at the two highest submaximal exercise intensities were significantly higher at −15°C than at 23°C. Time to exhaustion was
significantly shorter in the cold environment. However, no differences in V˙O2
max
or f
c were observed. Our results would suggest that exercise stress is higher at submaximal exercise intensities in a cold environment
and support the contention that aerobic capacity is not altered by cold exposure. Furthermore, we found that after Salbutamol
inhalation FEV1 was significantly higher than after placebo administration. However, the inhaled β2-agonist Salbutamol did not influence submaximal and maximal V˙O2, f
c, [La−]b or time to exhaustion in the elite, nonasthmatic cross-country skiers we studied. Thus, these results did not demonstrate
any ergogenic effect of the β2-agonist used.
Accepted: 18 August 1997 相似文献
13.
H. Hebestreit O. Bar-Or 《European journal of applied physiology and occupational physiology》1998,78(1):7-12
Heart rate (HR) monitoring is commonly used to assess 24-h energy expenditure (EE) in children but it has been found to overestimate
the true values. One reason for this may be the effect of climatic heat stress on HR. An equation has been previously developed
to adjust HR measured during continuous exercise for the influence of climate. Since play in children is rarely of a continuous
pattern, one objective of this study was to compare the effects of climatic heat stress on the HR response to intermittent
and to continuous exercise. A second objective was to determine whether the previously developed equation is suitable for
intermittent exercise. A group of 12 boys and 8 girls (aged 8–11 years) cycled in a climatic chamber. The exercise consisted
of continuous cycling for 5 min at 35%, 55%, and 75% of peak oxygen up take (random order) followed by alternating cycling
at the same resistance and cadence (30 s) and rest (30 s) for 3 additional min. The oxygen uptake (V˙O2) and HR were determined for 2 min at the end of continuous cycling and for 2 min during intermittent cycling. Climatic conditions
(randomly assigned) were dry bulb temperature T
db 22°C, 50% relative humidity (rh); T
db 28°C, 55% rh; T
db 32°C, 52% rh; or T
db 35°C, 58% rh. The difference between HR measured at a given T
db (HRmeas) and HR at 22°C and at the same V˙O2 was then calculated (ΔHR). The ΔHR increased linearly with increasing temperature but was not related to V˙O2 or to exercise type. However, a small but significant difference was found if the published equation was used with data from
intermittent exercise. The accuracy of the existing equation adjusting HRmeas for the influence of T
db (HRcorr) could be improved to HRcorr= HRmeas · (1.18308−(0.0083218 · T
db)). In conclusion, the effects of climatic heat stress on HR were similar in continuous and intermittent exercise, and HR
can be adjusted for the influence of climate in groups of pre- and early pubertal children during rest, intermittent and continuous
exercise at ambient temperatures between 22°C and 35°C, thereby reducing the error in predicting EE from HR.
Accepted: 13 January 1998 相似文献
14.
H. Arabi H. Vandewalle P. Pitor J. de Lattre H. Monod 《European journal of applied physiology and occupational physiology》1997,76(2):122-127
The present experiment was designed to study the importance of strength and muscle mass as factors limiting maximal oxygen
uptake (V˙O2
max
) in wheelchair subjects. Thirteen paraplegic subjects [mean age 29.8 (8.7) years] were studied during continuous incremental
exercises until exhaustion on an arm-cranking ergometer (AC), a wheelchair ergometer (WE) and motor-driven treadmill (TM).
Lean arm volume (LAV) was estimated using an anthropometric method based upon the measurement of various circumferences of
the arm and forearm. Maximal strength (MVF) was measured while pushing on the rim of the wheelchair for three positions of
the hand on the rim (−30°, 0° and +30°). The results indicate that paraplegic subjects reached a similar V˙O2
max
[1.23 (0.34) l · min−1, 1.25 (0.38) l · min−1, 1.22 (0.18) l · min−1 for AC, TM and WE, respectively] and V˙O2
max
/body mass [19.7 (5.2) ml · min−1 · kg−1, 19.5 (6.14) ml · min−1 · kg−1, 19.18 (4.27) ml · min−1 · kg−1 for AC, TM and WE, respectively on the three ergometers. Maximal heart rate f
c
max
during the last minute of AC (173 (17) beats · min−1], TM [168 (14) beats · min−1], and WE [165 (16) beats · min−1], were correlated, but f
c
max
was significantly higher for AC than for TM (P<0.03). There were significant correlations between MVF and LAV (P<0.001) and between the MVF data obtained at different angles of the hand on the rim [311.9 (90.1) N, 313.2 (81.2) N, 257.1
(71) N, at −30°, 0° and +30°, respectively]. There was no correlation between V˙O2
max
and LAV or MVF. The relatively low values of f
c
max
suggest that V˙O2
max
was, at least in part, limited by local aerobic factors instead of central cardiovascular factors. On the other hand, the
lack of a significant correlation between V˙O2
max
and MVF or muscle mass was not in favour of muscle strength being the main factor limiting V˙O2
max
in our subjects.
Accepted: 31 January 1997 相似文献
15.
Guido Ferretti Massimo Girardis Christian Moia Guglielmo Antonutto 《European journal of applied physiology and occupational physiology》1998,78(5):398-402
The hypothesis was tested that prolonged bed rest impairs O2 transport during exercise, which implies a lowering of cardiac output Q˙
c and O2 delivery (aO2). The following parameters were determined in five males at rest and at the steady-state of the 100-W exercise before (B)
and after (A) 42-day bed rest with head-down tilt at −6°: O2 consumption (V˙O2), by a standard open-circuit method; Q˙
c, by the pressure pulse contour method, heart rate ( f
c), stroke volume (Q
h), arterial O2 saturation, blood haemoglobin concentration ([Hb]), arterial O2 concentration (C
aO2), and Q˙
aO2. The V˙O2 was the same in A and in B, as was the resting f
c. The f
c at 100 W was higher in A than in B (+17.5%). The Q
h was markedly reduced (−27.7% and −22.2% at rest and 100 W, respectively). The Q˙
c was lower in A than in B [−27.6% and −7.8% (NS) at rest and 100 W, respectively]. The C
aO2 was lower in A than in B because of the reduction in [Hb]. Thus also Q˙
aO2 was lower in A than in B (−32.0% and −11.9% at rest and at 100 W, respectively). The present results would suggest a down-regulation
of the O2 transport system after bed rest.
Accepted: 22 April 1998 相似文献
16.
David V. B. James Jonathan H. Doust 《European journal of applied physiology and occupational physiology》1998,77(6):551-555
Eight male endurance runners [mean ± (SD): age 25 (6) years; height 1.79 (0.06) m; body mass 70.5 (6.0) kg; % body fat 12.5
(3.2); maximal oxygen consumption (V˙O2max 62.9 (1.7) ml · kg−1 · min−1] performed an interval training session, preceded immediately by test 1, followed after 1 h by test 2, and after 72 h by
test 3. The training session was six 800-m intervals at 1 km · h−1 below the velocity achieved at V˙O2max with 3 min of recovery between each interval. Tests 1, 2 and 3 were identical, and included collection of expired gas, measurement
of ventilatory frequency (f
v
), heart rate (f
c), rate of perceived exertion (RPE), and blood lactate concentration ([La−]B) during the final 5 min of 15 min of running at 50% of the velocity achieved at V˙O2max (50% −V˙O2max).␣Oxygen uptake (V˙O2), ventilation (V˙
E
), and respiratory exchange ratio (R) were subsequently determined from duplicate expired gas collections. Body mass and plasma volume changes were measured preceding
and immediately following the training session, and before tests 1–3. Subjects ingested water immediately following the training
session, the volume of which was determined from the loss of body mass during the session. Repeated measures analysis of variance
with multiple comparison (Tukey) was used to test differences between results. No significant differences in body mass or
plasma volume existed between the three test stages, indicating that the differences recorded for the measured parameters
could not be attributed to changes in body mass or plasma volume between tests, and that rehydration after the interval training
session was successful. A significant (P < 0.05) increase was found from test 1 to test 2 [mean (SD)] for V˙O2 [2.128 (0.147) to 2.200 (0.140) 1 · min−1], f
c [125 (17) to 132 (16) beats · min−1], and RPE [9 (2) to 11 (2)]. A significant (P < 0.05) decrease was found for submaximal R [0.89 (0.03) to 0.85 (0.04)]. These results suggest that alterations in V˙O2 during moderate-intensity, constant-velocity running do occur following heavy-intensity endurance running training, and that
this is due to factors in addition to changed substrate metabolism towards greater fat utilisation, which could explain only
31% of the increase in V˙O2.
Accepted: 8 December 1997 相似文献
17.
Nigel A. S. Taylor Mark J. Patterson James D. Cotter Duncan J. Macfarlane 《European journal of applied physiology and occupational physiology》1997,76(4):380-386
The influence of artificially induced anaemia on thermal strain was evaluated in trained males. Heat stress trials (38.6°C,
water vapour pressure 2.74 kPa) performed at the same absolute work rates [20 min of seated rest, 20 min of cycling at 30%
peak aerobic power (V˙O2peak), and 20 min cycling at 45% V˙O2peak] were completed before (HST1) and 3–5 days after 3 units of whole blood were withdrawn (HST2). Mild anaemia did not elevate
thermal strain between trials, with auditory canal temperatures terminating at 38.5°C [(0.16), HST1] and 38.6°C [(0.13), HST2;
P > 0.05]. Given that blood withdrawal reduced aerobic power by 16%, this observation deviates from the close association often
observed between core temperature and relative exercise intensity. During HST2, the absolute and integrated forearm sweat
rate (m˙
sw) exceeded control levels during exercise (P < 0.05), while a suppression of forehead m˙
sw occurred (P < 0.05). These observations are consistent with a possible peripheral redistribution of sweat secretion. It was concluded
that this level of artificially induced anaemia did not impact upon heat strain during a 60-min heat stress test.
Accepted: 17 April 1997 相似文献
18.
Stephen S. Cheung Tom M. McLellan 《European journal of applied physiology and occupational physiology》1998,78(1):50-58
The purpose of the present study was to determine the separate and combined effects of a short-term aerobic training program
and hypohydration on tolerance during light exercise while wearing nuclear, biological, and chemical protective clothing in
the heat (40°C, 30% relative humidity). Males of moderate fitness [<50 ml · kg−1 · min−1 maximal O2 consumption (V˙O2
max
)] were tested while euhydrated or hypohydrated by ≈2% of body weight through exercise and fluid restriction the day preceding
the trials. Tests were conducted before and after either a 2-week program of daily aerobic training (1 h treadmill exercise
at 65% V˙O2
max
for 12 days; n = 8) or a control period (n = 7), which had no effect on any measured variable. The training increased V˙O2
max
by 6.5%, while heart rate (f
c) and the rectal temperature (T
re) rise decreased during exercise in a thermoneutral environment. In the heat, training resulted in a decreased skin temperature
and increased sweat rate, but did not affect f
c, T
re or tolerance time (TT). In both training and control groups, hypohydration significantly increased T
re and f
c and decreased the TT. It was concluded that the short-term aerobic training program had no benefit on exercise-heat tolerance
in this uncompensable heat stress environment.
Accepted: 12 November 1997 相似文献
19.
D. L. Thompson K. M. Townsend R. Boughey K. Patterson D. R. Bassett Jr 《European journal of applied physiology and occupational physiology》1998,78(1):43-49
Substrate utilization during and after low- and moderate-intensity exercise of similar caloric expenditure was compared.
Ten active males [age: 26.9 (4.8) years; height: 181.1 (4.8) cm; Mass: 75.7 (8.8) kg; maximum O2 consumption (V˙O2
max
): 51.2 (4.8) ml · kg−1 · min−1] cycled at 33% and 66% V˙O2
max
on separate days for 90 and 45 min, respectively. After exercise, subjects rested in a recumbent position for 6 h. Two h
post-exercise, subjects ate a standard meal of 66% carbohydrate (CHO), 11% protein, and 23% fat. Near-continuous indirect
calorimetry and measurement of urinary nitrogen excretion were used to determine substrate utilization. Total caloric expenditure
was similar for the two trials; however, significantly (P<0.05) more fat [42.4 (3.6) g versus 24.0 (12.2) g] and less CHO [142.5 (28.5) g versus 188.8 (45.2) g] was utilized as a
substrate during the low-intensity compared to the moderate-intensity trial. Protein utilization was similar for the two trials.
The difference in substrate use can be attributed to the exercise period because over twice as much fat was utilized during
low-intensity [30.0 (11.0) g] compared to moderate-intensity exercise [13.6 (6.6) g]. Significantly more (P<0.05) CHO was utilized during the moderate-intensity [106.0 (27.8) g] compared to the low-intensity exercise [68.7 (20.0) g].
Substrate use during the recovery period was not significantly different. We conclude that low-intensity, long-duration exercise
results in a greater total fat oxidation than does moderate intensity exercise of similar caloric expenditure. Dietary-induced
thermogenesis was not different for the two trials.
Accepted: 3 November 1997 相似文献
20.
J. Hall I. A. Macdonald P. J. Maddison J. P. O''Hare 《European journal of applied physiology and occupational physiology》1998,77(3):278-284
This study compared the cardiorespiratory responses of eight healthy women (mean age 30.25 years) to submaximal exercise
on land (LTm) and water treadmills (WTm) in chest-deep water (Aquaciser). In addition, the effects of two different water
temperatures were examined (28 and 36°C). Each exercise test consisted of three consecutive 5-min bouts at 3.5, 4.5 and 5.5 km · h−1. Oxygen consumption (V˙O2) and heart rate (HR), measured using open-circuit spirometry and telemetry, respectively, increased linearly with increasing
speed both in water and on land. At 3.5 km · h−1
V˙O2 was similar across procedures [χ = 0.6 (0.05) l · min−1]. At 4.5 and 5.5 km · h−1
V˙O2 was significantly higher in water than on land, but there was no temperature effect (WTm: 0.9 and 1.4, respectively; LTm:
0.8 and 0.9 l · min−1, respectively). HR was significantly higher in WTm at 36°C compared to WTm at 28°C at all speeds, and compared to LTm at
4.5 and 5.5 km · h−1 (P ≤ 0.003). The HR-V˙O2 relationship showed that at a V˙O2 of 0.9 l · min−1, HR was higher in water at 36°C (115 beats · min−1) than either on land (100 beats · min−1) or in water at 28°C (99 beats · min−1). The Borg scale of perceived exertion showed that walking in water at 4.5 and 5.5 km · h−1 was significantly harder than on land (WTm: 11.4 and 14, respectively; LTm: 9.9 and 11, respectively; P ≤ 0.001). These cardiorespiratory changes occurred despite a slower cadence in water (the mean difference at all speeds was
27 steps/min). Thus, walking in chest-deep water yields higher energy costs than walking at similar speeds on land. This data
has implications for therapists working in hydrotherapy pools.
Accepted: 3 September 1997 相似文献