峰革赛螨幼若螨描述（蜱螨亚纲：革螨股：胭螨科）

在所查到的献中，峰革赛螨Gamasellus montanus(Willmann，1936)仅描述了雌和雄螨(BpereToBa和Щep5aK，1977)，本描述其后若螨、前若螨和幼螨。中测量单位为μm，括号内为测量均值。     

东北常革螨后若螨描述（蜱螨亚纲：寄螨科）

东北常革螨过去仅描述雌螨和雄螨，本描述后若螨。中测量单位为μm。     

武汉革螨的新纪录及亲缘寄螨前若螨描述（蜱螨亚纲：革螨亚目：寄螨科）

记录在武汉采到的富生寄螨、亲缘寄螨和1种未定的厚厉螨,并描述亲缘寄螨前若螨。     

叶氏广厉螨雄螨与若螨描述(蜱螨亚纲：革螨股：厉螨科)

本文描述叶氏广厉螨Cosmolaelaps yeruzyuae Ma,1995雄螨、后若螨及前若螨。     

维螨属一新种：蜱螨亚纲：革螨股

本文记述维螨属１新种：汤旺河维螨Ｖｅｉｇａｉａｔａｎｇｗａｎｇｈｅｎｓｉｓｓｐ．ｎｏｖ。     

曲美绥螨雄螨和洮儿河美绥螨前若螨描述（蜱螨亚纲：革螨股：美绥螨科）

描述曲美绥螨Ａｍｅｒｏｓｅｉｕｓ ｃｕｒｖａｔｕｓ Ｇｕ．Ｗａｎｇ ｅｔ Ｂａｉ,１９８９雄螨和洮儿河美绥螨Ａｍｅｒｏｓｅｉｕｓ ｔａｏｅｒｈｅｎｓｉｓ,Ｍａ,１９９５前若螨。     

背刻螨属—新种及洮儿河美绥螨雄螨、后若螨描述（蜱螨亚纲：革螨股：美绥螨科）

报道背刻螨属在中国的发现。并描述1新种：吉林背刻螨Epicriopsis jilinensis，sp.nov.。同时描述洮儿河美绥螨Ameroseius taoerhensis Ma，1995雄螨及后若螨。模式标本存在于吉林省白城市全国鼠疫布氏菌病防治基地。     

寄螨属四种前若螨描述（蜱螨亚纲：革螨股）

本文描述寄螨属4种前著螨;邓氏寄螨Parasitus tengkuofani Ma,1995,王氏寄螨parasitus wangdunqingi Ma,1995,温氏寄螨parasitus wentinghuani Ma,1996,二刺寄螨parasitus bis pinatus Ma,1996.     

中国北方寄螨属亲缘种团的螨类（蜱螨亚纲：革螨股：寄螨科）

中国北方寄螨属亲缘种团已知有5种螨,即（1）季氏寄螨Parasitus tichomirovi Davydova,1971,(2)王氏寄螨Parasitus wangdunqingi Ma,1995,(3)短毛寄螨Parasitus brachychaetus Ma,1986,(4)乳突寄螨Parasitus mammilatus (Berlese,1904),(5)毛螨螨Parasitus setosus Oudemans et Voigts,1904.     

新革螨属二新种记述：蜱螨亚纲：革螨股：寄螨科

记述新革螨属２新种：异形新革螨Ｎｅｏｇａｍａｓｕｓａｎｏｍａｌｕｓｓｐ．ｎｏｖ．和皱形新革螨Ｎｅｏｇａｍａｓｕｓｃｒｉｓｐｕｓｓｐ．ｎｏｖ。     

Quadruplexes of human telomere DNA analogs designed to contain G:A:G:A,G:G:A:A,and A:A:A:A tetrads

Replacement of two to four guanines by adenines in the human telomere DNA repeat dG₃(TTAG₃)₃ did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na⁺ and K⁺ solutions. Thermodynamic data showed that, in Na⁺ solution, the dG₃(TTAG₃)₃ quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K⁺ solution, the highest destabilization was caused by the 4A tetrad. In K⁺, only the unmodified dG₃(TTAG₃)₃ quadruplex rearranged into a K⁺‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.