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1.
马立名 《蛛形学报》1997,6(2):140-142
首次描述甘肃常革螨Vulgarogamasus gansuensis Ma,1987雄螨,并报道中国1新纪录,即达氏异肢螨Poecilochirus davydovae Hyatt,1980。  相似文献   

2.
球革螨科一新属记述(蜱螨亚纲:革螨股)   总被引:3,自引:0,他引:3  
刘井元  马立名 《昆虫学报》2001,44(3):356-359
记述了球革螨科一新属Mirabulbus gen. nov.,包括2种:秦巴异球螨M. qinbaensis sp. nov.和亚东异球螨M.yadongensis (Ma et Wang,1997)。  相似文献   

3.
马立名 《蛛形学报》2004,13(1):23-27
记述革伊螨属1新种:钩形革伊螨Gamasiphis aduncus sp.nov.,并补充新美革伊螨Gamasiphis novipulchellus Ma et Yin, 1998特征.  相似文献   

4.
记述革板螨属1新种:拟绒革板螨Gamasholaspis imiteothenomydis,sp.nov.,并描述绒鼠革板螨Gamasholaspis eothenomydis雄螨、后若螨和前若螨。  相似文献   

5.
记述革赛螨属一新种:细毛革赛螨Gamasellus leptinochaetus sp.nov.和囊螨属一新种:拟异囊螨Asca subidiobasis sp.nov.,并对异基囊螨Asca idiobasis Gu et Guo,1997,安氏囊螨Asca anwenjui Ma,2003和拟巨囊螨Asca submajor Ma,2003进行补充描述.  相似文献   

6.
中国异肢螨属一新纪录和美绥螨属一新纪录(蜱螨亚纲:革螨股)马立名(吉林省地方病第一防治研究所白城137000)ANEWRECORDOFPOECILOCHIRUSANDANEWRECORDOFAMEROSEIUSFROMCHINA(ACARI:GAMA...  相似文献   

7.
记述派伦螨属1新种:遵义派伦螨Parholaspulus zunyiensis sp.nov.和革板螨属1新种:江西革板螨Gamasholaspis jiangxiensissp.nov.,并描述都匀革板螨Gamasholaspis duyunensis Chen,Guo et Gu,1994雄螨.  相似文献   

8.
血革螨属一新种:寄螨目:血革螨科   总被引:2,自引:2,他引:0  
本文介绍了血革螨属一新种:贡山血革螨 Haemogamasus gongshanensis sp.nov.,它采自多齿鼩鼹 Nasillus gracilis Thomas 体上。该文对新种的形态特征作了详细描述,并与近似种云龙血革螨 Haemogamasus yunlongensis Gu et Fang进行了比较。  相似文献   

9.
马立名 《蛛形学报》2005,14(2):70-73
记述胭螨科Rhodacaridae 2新种:新双色革鞍螨Gamasellodes novibicolor sp.nov.和长毛囊螨Asca dolichosetosa sp.nov.,并补充植囊螨Asca plantaria Ma,1996特征描述.  相似文献   

10.
记述新革螨属1新种:泰安新革螨Neogamasus taianensis Ma et Sun, sp. nov.,并对峨眉常革螨Vulgarogamasus emeishanensis Ma et Wang.1996和异形新革螨Neogamasus anomalus Ma et Yan,1998进行补充描述。  相似文献   

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Replacement of two to four guanines by adenines in the human telomere DNA repeat dG3(TTAG3)3 did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na+ and K+ solutions. Thermodynamic data showed that, in Na+ solution, the dG3(TTAG3)3 quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K+ solution, the highest destabilization was caused by the 4A tetrad. In K+, only the unmodified dG3(TTAG3)3 quadruplex rearranged into a K+‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.  相似文献   

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