四川省小蹦蝗属一新种（直翅目：蝗总科：斑腿蝗科）

本文记述采自中国四川省的小蹦蝗属１新种－－万县小蹦蝗Ｐｅｄｏｐｏｄｉｓｍａ ｗａｎｘｉａｎｅｎｓｉｓ,ｓｐ．ｎｏｖ．,模式标本存陕西师范大学动物研究所。     

湖南武陵山地区小蹦蝗属一新种（直翅目：斑腿蝗科）

本文记述了采自湖南武陵山区的小蹦蝗属ＰｅｄｏｐｏｄｉｓｍａＺｈｅｎｇ一新种,小尾小蹦蝗Ｐ．ｆｕｒｃｕｌａｓｐ．ｎｏｖ该新种近似于秦岭小蹦蝗Ｐ．ｔｓｉｎｌｉｎｇｅｎｓｉｓ（Ｃｈｅｎｇ）。模式标本保存在陕西师范大学动物研究所蝗虫研究室。     

浙江省小蹦蝗属一新种

本文记述采自中国浙江省的小蹦蝗属１新种，即乌岩岭小蹦蝗Ｐｅｄｏｐｏｄｉｓｍａ ｗｕｙａｎｌｉｎｇｅｎｓｉｓ，ｓｐ．ｎｏｖ．模式标本保存于山东大学生物系。     

湖南衡山蹦蝗属一新种(直翅目:斑腿蝗科)

本文记述采自湖南省衡山地区的蹦蝗属Ｓｉｎｏｐｏｄｉｓｍａ Ｃｈａｎｇ一新种,衡山蹦蝗Ｓ．ｈｅｎｇｓｈａｎｉｃａ ｓｐ．ｎｏｖ．。     

东北地区蝗虫－新属及三新种（直翅目：蝗总科）

本文记述了采自东北地区蝗虫的一新属,拟蹦蝗属Ｓｉｎｏｐｏｄｉｓｍｏｉｄｅｓｇｅｎ．ｎｏｖ．及三新种,千山拟蹦蝗Ｓｉｎｏｐｏｄｉｓｍｏｉｄｅｓｑｉａｎｓｈａｎｅｎｓｉｓｓｐ．ｎｏｖ．、草绿拟蹦蝗Ｓｉｎｏｐｏｄｉｓｍｏｉｄｅｓｐｒａｓｉｎａｓｐ．ｎｏｖ．和长白山玛蝗Ｍｉｒａｍｅｌｌａｃｈａｎｇｂａｉｓｈａｎｅｎｓｉｓｓｐ．ｎｏｖ．。模式标本保存于陕西师范大学动物研究所。     

斑腿蝗科三个种类精子的比较研究：直翅目：蝗总科

本文通过对日本黄脊蝗Ｐａｔａｎｇａｊａｐｏｉｎｃａ（Ｉ．Ｂｄ）,短角直斑腿蝗ＳｔｅｎｏｃａｔａｎｔｏｐｓｍｉｓｔｓｈｅｎｋｏｉＷｉｌｌ和霍山蹦蝗ＳｉｎｏｐｏｄｉｓｍａｈｏｕｓｈａｎａＨｕａｎｇ精子的比较,发现了它们的精子的线粒体衍生体和中心粒侧体的超微形态结构十分相似,在精子发生过程中７、８两期精细胞线粒体衍生体周围微管的排数相一致,这３个种精子间的差异主要反映在外形头和尾的长度,线粒体衍生体的大     

四种斑腿蝗科昆虫染色体带型的比较

应用染色体C带及银染核仁组成区定位分析方法对斑腿蝗科4种昆虫,即小蹦蝗属2种;峨眉小蹦蝗Pedopodisma emeiensis和秦岭小蹦蝗P.tsinlingensis;无翅蝗属2种;小无翅蝗Zubovskiaparvula和柯氏无翅蝗Z.koeppeni进行了研究,结果表明：4种蝗虫染色体数目（2n♂=21,XO)一致,小蹦蝗属M8染色体上具有端带;无翅蝗属X染色体具有特殊带纹,银染核仁组成区定位在两属各有特点,小蹦蝗属位于第8号染色体端部区域;无翅蝗属位于X染色体端部区域,除上述属内共性之外,各物种C带带纹又存在明显差异,其中峨眉小蹦蝗带型极为特殊。文中还对上述4种蝗虫带型结构特征及其进化意义作了讨论。     

中国四川省小蹦蝗属一新种(直翅目,蝗总科,斑腿蝗科)

记述采自中国四川省斑腿蝗科秃蝗亚科小蹦蝗属Pedopodisma Zheng,1980的1新种,阿坝小蹦蝗Pedopodisma abaensis sp.nov..新种同神农架小蹦蝗Pedopodisma shennongjiaensis Wang et Li,1996近似,其区别特征为:体较小,体长♂12.1～12.5mm,♀ 16.5～19.2 mm;触角中段一节长为宽的2.0倍;雄性肛上板基部纵沟宽度一致,端部两侧缺纵脊;雄性下生殖板顶端较粗;雌性产卵瓣较狭,上产卵瓣长为宽的3.0倍.模式标本保存于山东农业大学植物保护学院,泰安.阿坝小蹦蝗,新种Pedopodisma abaensis sp.nov.(图1～7)正模♂.副模:1 ♂,2♀♀,四川阿坝(32°54'N,101°44′E;海拔3 300 m),2011-08-24,郑方强、叶保华采.词源:新种种名源自模式标本产地地名.     

南疆束颈蝗属二新种：蝗总科：斑翅蝗科

本文记述采自新疆南部地区束颈蝗属二新种,叶城束颈蝗Ｓｐｈｉｎｇｏｎｏｔｕｓｙｅｃｈｅｎｇｅｎｓｉｓ ｓｐ．ｎｏｖ．及塔克拉玛束颈蝗Ｓｐｈｉｎｇｏｎｏｔｕｓ ｔａｋｒａｍａｅｎｓｉｓ ｓｐ．ｎｏｖ．模式标本保存于陕西范大学动物研究所标本室。     

青海省雏蝗属三新种（直翅目：网翅蝗科）

记述采自青海少网翅蝗科雏蝗属３新种,即祁连山雏蝗Ｃｈｏｒｔｈｉｐｐｕｓ ｑｉｌｉａｎｓｈａｎｅｎｓｉｓ,ｓｐ．ｎ．、积石山雏蝗Ｃｈｏｕｔｈｉｐｐｕｓ ｕｉｓｈｉｓｈａｎｅｎｓｉｓ,ｓｐ．ｎ．及循化雏蝗Ｃｈｏｒｔｈｉｐｐｕｓ ｘｕｎｈｕａｅｎｓｉｓ,ｓｐ．ｎ．。模式标本保存于陕西师范大学动物研究所。     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.