共查询到20条相似文献,搜索用时 46 毫秒
1.
2.
3.
4.
5.
6.
EBV-induced gene 3 transcription is induced by TLR signaling in primary dendritic cells via NF-kappa B activation 总被引:7,自引:0,他引:7
Wirtz S Becker C Fantini MC Nieuwenhuis EE Tubbe I Galle PR Schild HJ Birkenbach M Blumberg RS Neurath MF 《Journal of immunology (Baltimore, Md. : 1950)》2005,174(5):2814-2824
7.
8.
9.
10.
11.
12.
13.
14.
15.
Multiple cis Regulatory Elements Control RANTES Promoter Activity in Alveolar Epithelial Cells Infected with Respiratory Syncytial Virus
下载免费PDF全文
![点击此处可从《Journal of virology》网站下载免费的PDF全文](/ch/ext_images/free.gif)
Antonella Casola Roberto P. Garofalo Helene Haeberle Todd F. Elliott Rongtuan Lin Mohammad Jamaluddin Allan R. Brasier 《Journal of virology》2001,75(14):6428-6439
16.
17.
18.
19.
Noulin N Quesniaux VF Schnyder-Candrian S Schnyder B Maillet I Robert T Vargaftig BB Ryffel B Couillin I 《Journal of immunology (Baltimore, Md. : 1950)》2005,175(10):6861-6869
Inhaled endotoxin induces an inflammatory response that contributes to the development and severity of asthma and other forms of airway disease. Here, we show that inhaled endotoxin-induced acute bronchoconstriction, TNF, IL-12p40, and KC production, protein leak, and neutrophil recruitment in the lung are abrogated in mice deficient for the adaptor molecule MyD88. Bronchoconstriction, inflammation, and protein leak are normal in Toll/IL-1R domain-containing adaptor inducing IFN-beta-deficient mice. MyD88 is involved in TLR, but also in IL-1R-associated kinase 1-mediated IL-1R and -18R signaling. We exclude a role for IL-1 and IL-18 pathways in this response, as IL-1R1 and caspase-1 (ICE)-deficient mice develop lung inflammation while TLR4-deficient mice are unresponsive to inhaled LPS. Significantly, using bone marrow chimera, we demonstrate that both hemopoietic and resident cells are necessary for a full MyD88-dependent response to inhaled endotoxin; bronchoconstriction depends on resident cells while cytokine secretion is mediated by hemopoietic cells. 相似文献
20.
Gu JQ Ikuyama S Wei P Fan B Oyama J Inoguchi T Nishimura J 《American journal of physiology. Endocrinology and metabolism》2008,295(6):E1390-E1400