中国暇尖翅蝇属二新种记述（双翅目：尖翅蝇科）

记述暇尖翅蝇属Spilolonchoptera2新种,即短尾暇尖翅蝇S.brevicaudata sp.nov.和杨氏暇尖翅蝇S.yangi sp.nov.。     

马缨丹蛇潜蝇在中国大陆首报及中国蛇潜蝇属分布名录（双翅目：潜叶蝇科）

马缨丹蛇潜蝇Ophiomyia lantanae(Froggatt,1919)首次在云南报道,对其形态特征和外生殖器特征进行了重新拍照、描述,编制了中国蛇潜蝇属分种检索表,列出了24种蛇潜蝇的寄主植物和分布。     

卵甲蝇属一新种记述（双翅目：甲蝇科）

全世界已知卵甲蝇属共4种,本文记述1新种,即神农架卵甲蝇Oocelyphus shennongjianus sp.nov.,文中还编制了卵甲蝇属检索表。     

中国四川省有瓣蝇类三新种(双翅目:厕蝇科,蝇科)

报道采自中国四川省西部山区有瓣蝇类Calyptraae3新种：厕蝇科Fanniiade厕蝇属Fannia2新种：五枝厕蝇Fannia quinquiramula sp.nov.,虞氏厕蝇Fannia yui sp.nov.;蝇科Muscidae棘蝇属Phaonia 1新种：太子棘蝇Phaonia taizipingga sp.nov.。新种虞氏侧蝇的命名,系对虞以新教授在中国蠓科Ceratopogoinidae等医学昆虫研究所作杰出贡献的敬意。厕蝇科模式标本存中国沈阳师范学院昆虫研究所;蝇科模式标本存北京军事医学科学院微生物流行病研究所医学昆虫标本馆。     

改进蝇类昆虫触角电位技术的尝试

本文通过对美洲斑潜蝇触角的扫描电镜观察及触角电位的测定 ,提出对蝇类昆虫触角电位测定技术的改进方法 :将触角芒切断 ,露出血淋巴 ,记录电极与触角芒的断面相接 ,参照电极通过头孔插入脑血腔形成回路。     

中国有瓣蝇类三新种：（双翅目：丽蝇科,蝇科）

本文报道采自中国四川的瓣蝇类丽蝇科蜗蝇属Ｍｅｌｉｎｄａ Ｒｏｂｉｎｅａｕ－Ｄｅｓｖｏｉｄｙ １新种：端钩蜗蜗Ｍｅｌｉｎｄａ ａｐｉｃｉｈａｍａｔａ ｓｐ．ｎｏｖ;蝇科移属Ｃｏｅｎｏｓｉａ Ｍｅｉｇｅｎ２新种：黑杂移蝇Ｃｏｅｎｏｓｉａ ｎｉｇｒｉｍｉｘｔａ ｓｐ．ｎｏｖ,黄杂移蝇Ｃｏｅｎｏｓｉａ ｆｌａｒｉｍｉｘｔａ ｓｐ．ｎｏｖ。模式标本保存于沈阳师范学院昆虫研究所。     

中国四川有瓣蝇类四新种(双翅目:厕蝇科,蝇科,丽蝇科)

记述分布于中国四川西部的有瓣蝇类中的4新种：天府枵蝇Coelomyia tianfuensis sp．nov．(厕蝇科);蜀圆蝇Mydaea shuensis sp．nov(蝇科);中华重毫蝇Dichaetomyla sinica sp．nov(蝇科);西部变丽蝇Paradichosia xibuica sp．nov(丽蝇科)。模式标本存中国科学院上海生命科学研究院,植物生理生态研究所。     

中国角潜蝇属莎草潜蝇亚属分类研究(双翅目,潜蝇科)

对中国角潜蝇属莎草潜蝇亚属进行了分类研究,确认我国现知2种：即福建角潜蝇C.(B.)fujianisca sp.nov和苔角潜蝇Cerodontha(Butomomyza)cornigera(dc Meijcrc)。除新种描述并附特征图外,还提供该亚属中国种类分种检索表。模式标本保存于中国科学院动物研究所动物标本馆。     

Super-size flies

The increasing prevalence of obesity and other nutrition-related chronic diseases has prompted considerable efforts to understand their pathogenesis and treatment. One experimental approach is to overexpress, inactivate, or manipulate specific genes that regulate energy metabolism and fat storage. Many such techniques are fully established, routine tools in Drosophila and C. elegans, which provide elegant models for dissecting endocrine problems and metabolic pathways.     

Plastic flies

Individuals within species and populations vary. Such variation arises through environmental and genetic factors and ensures that no two individuals are identical. However, it is clear that not all traits show the same degree of intraspecific variation. Some traits, in particular secondary sexual characteristics used by males to compete for and attract females, are extremely variable among individuals in a population. Other traits, for example brain size in mammals, are not. Recent research has begun to explore the possibility that the extent of phenotypic variation (here referred to as “variability”) may be a character itself and subject to natural selection. While these studies support the concept of variability as an evolvable trait, controversy remains over what precisely the trait is. At the heart of this controversy is the fact that there are very few examples of developmental mechanisms that regulate trait variability in response to any source of variation, be it environmental or genetic. Here, we describe a recent study from our laboratory that identifies such a mechanism. We then place the study in the context of current research on the regulation of trait variability, and discuss the implications for our understanding of the developmental regulation and evolution of phenotypic variation.     

Asymmetric flies

What are the sources of phenotypic variation and which factors shape this variation are fundamental questions of developmental and evolutionary biology. Despite this simple formulation and intense research, controversy remains. Three points are particularly discussed: (1) whether adaptive developmental mechanisms buffering variation exist at all; (2) if yes, do they involve specific genes and processes, i.e., different from those involved in the development of the traits that are buffered?; and (3) whether different mechanisms specifically buffer the various sources of variation, i.e., genetic, environmental and stochastic, or whether a generalist process buffers them all at once. We advocate that experimental work integrating different levels of analysis will improve our understanding of the origin of phenotypic variation and thus help answering these contentious questions. In this paper, we first survey the current views on these issues, highlighting potential sources of controversy. We then focus on the stochastic part of phenotypic variation, as measured by fluctuating asymmetry, and on current knowledge about the genetic basis of developmental stability. We report our recent discovery that an individual gene, Cyclin G, plays a central role—adaptive or not—in developmental stability in Drosophila.¹ We discuss the implications of this discovery on the regulation of organ size and shape, and finally point out open questions.