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1.
Hiroshi Takaki Kenji Sunagawa Masaru Sugimachi Yasushi Hara Toru Kawada Takashi Kurita Yoichi Goto 《European journal of applied physiology and occupational physiology》1998,78(4):333-339
The transient response of oxygen uptake (V˙O2) to submaximal exercise, known to be abnormal in patients with cardiovascular disorders, can be useful in assessing the functional
status of the cardiocirculatory system, however, a method for evaluating it accurately has not yet been established. As an
alternative approach to the conventional test at constant exercise intensity, we applied a random stimulus technique that
has been shown to provide relatively noise immune responses of system being investigated. In 27 patients with heart failure
and 24 age-matched control subjects, we imposed cycle exercise at 50 W intermittently according to a pseudo-random binary
(exercise-rest) sequence, while measuring breath-by-breath V˙O2. After determining the transfer function relating exercise intensity (W˙) to V˙O2 and attenuating the high frequency ranges (>6 exercise-rest cycles · min−1), we computed the high resolution band-limited (0–6 cycles · min−1) V˙O2 response (0–120 s) to a hypothetical step exercise. The V˙O2 response showed a longer time constant in the patients than in the control subjects [47 (SD 37) and 31 (SD 8) s, respectively,
P < 0.05]. Furthermore, the amplitude of the V˙O2 response after the initial response was shown to be significantly smaller in the patients than in the control subjects [176
(SD 50) and 267 (SD 54) ml · min−1 at 120 s]. The average amplitude over 120 s correlated well with peak V˙O2 (r = 0.73) and ΔV˙O2/ΔW˙ (r = 0.70), both of which are well-established indexes of exercise tolerance. The data indicated that our band-limited V˙O2
step response using random exercise was more markedly attenuated and delayed in the patients with heart failure than in the normal controls
and that it could be useful in quantifying the overall functional status of the cardiocirculatory system.
Accepted: 6 January 1998 相似文献
2.
Greg J. Wilson Andrew D. Walshe Mark R. Fisher 《European journal of applied physiology and occupational physiology》1997,75(5):455-461
The aims of the present study were: (1) to assess aerobic metabolism in paraplegic (P) athletes (spinal lesion level, T4–L3)
by means of peak oxygen uptake (V˙O2peak) and ventilatory threshold (VT), and (2) to determine the nature of exercise limitation in these athletes by means of cardioventilatory
responses at peak exercise. Eight P athletes underwent conventional spirographic measurements and then performed an incremental
wheelchair exercise on an adapted treadmill. Ventilatory data were collected every minute using an automated metabolic system:
ventilation (l · min−1), oxygen uptake (V˙O2, l · min−1, ml · min−1 · kg−1), carbon dioxide production (V˙CO2, ml · min−1), respiratory exchange ratio, breathing frequency and tidal volume. Heart rate (HR, beats · min−1) was collected with the aid of a standard electrocardiogram. V˙O2peak was determined using conventional criteria. VT was determined by the breakpoint in the V˙CO2−V˙O2 relationship, and is expressed as the absolute VT (V˙O2, ml · min−1 · kg−1) and relative VT (percentage of V˙O2peak). Spirometric values and cardioventilatory responses at rest and at peak exercise allowed the measurement of ventilatory
reserve (VR), heart rate reserve (HRr), heart rate response (HRR), and O2 pulse (O2 P). Results showed a V˙O2peak value of 40.6 (2.5) ml · min−1 · kg−1, an absolute VT detected at 23.1 (1.5) ml · min−1 · kg−1
V˙O2 and a relative VT at 56.4 (2.2)% V˙O2peak. HRr [15.8 (3.2) beats · min−1], HRR [48.6 (4.3) beat · l−1], and O2 P [0.23 (0.02) ml · kg−1 · beat−1] were normal, whereas VR at peak exercise [42.7 (2.4)%] was increased. As wheelchair exercise excluded the use of an able-bodied
(AB) control group, we compared our V˙O2peak and VT results with those for other P subjects and AB controls reported in the literature, and we compared our cardioventilatory
responses with those for respiratory and cardiac patients. The low V˙O2peak values obtained compared with subject values obtained during an arm-crank exercise may be due to a reduced active muscle
mass. Absolute VT was somewhat comparable to that of AB subjects, mainly due to the similar muscle mass involved in wheelchair
and arm-crank exercise by P and AB subjects, respectively. The increased VR, as reported in patients with chronic heart failure,
suggested that P athletes exhibited cardiac limitation at peak exercise, and this contributed to the lower V˙O2peak measured in these subjects.
Accepted: 22 April 1997 相似文献
3.
Jerzy A. Zoladz Zbigniew Szkutnik Joanna Majerczak Krzysztof Duda 《European journal of applied physiology and occupational physiology》1998,78(4):369-377
The purpose of this study was to develop a method to determine the power output at which oxygen uptake (V˙O2) during an incremental exercise test begins to rise non-linearly. A group of 26 healthy non-smoking men [mean age 22.1 (SD
1.4) years, body mass 73.6 (SD 7.4) kg, height 179.4 (SD 7.5) cm, maximal oxygen uptake (V˙O2max) 3.726 (SD 0.363) l · min−1], experienced in laboratory tests, were the subjects in this study. They performed an incremental exercise test on a cycle
ergometer at a pedalling rate of 70 rev · min−1. The test started at a power output of 30 W, followed by increases amounting to 30 W every 3 min. At 5 min prior to the first
exercise intensity, at the end of each stage of exercise protocol, blood samples (1 ml each) were taken from an antecubital
vein. The samples were analysed for plasma lactate concentration [La]pl, partial pressure of O2 and CO2 and hydrogen ion concentration [H+]b. The lactate threshold (LT) in this study was defined as the highest power output above which [La−]pl showed a sustained increase of more than 0.5 mmol · l−1 · step−1. The V˙O2 was measured breath-by-breath. In the analysis of the change point (CP) of V˙O2 during the incremental exercise test, a two-phase model was assumed for the 3rd-min-data of each step of the test: X
i
=at
i
+b+ɛ
i
for i=1,2,…,T, and E(X
i
)>at
i
+b for i =T+1,…,n, where X
1, … , X
n
are independent and ɛ
i
∼N(0,σ2). In the first phase, a linear relationship between V˙O2 and power output was assumed, whereas in the second phase an additional increase in V˙O2 above the values expected from the linear model was allowed. The power output at which the first phase ended was called the
change point in oxygen uptake (CP-V˙O2). The identification of the model consisted of two steps: testing for the existence of CP and estimating its location. Both
procedures were based on suitably normalised recursive residuals. We showed that in 25 out of 26 subjects it was possible
to determine the CP-O2 as described in our model. The power output at CP-V˙O2 amounted to 136.8 (SD 31.3) W. It was only 11 W – non significantly – higher than the power output corresponding to LT. The
V˙O2 at CP-V˙O2 amounted to 1.828 (SD 0.356) l · min−1 was [48.9 (SD 7.9)% V˙O2
max
]. The [La−]pl at CP-V˙O2, amounting to 2.57 (SD 0.69) mmol · l−1 was significantly elevated (P<0.01) above the resting level [1.85 (SD 0.46) mmol · l−1], however the [H+]b at CP-V˙O2 amounting to 45.1 (SD 3.0) nmol · l−1, was not significantly different from the values at rest which amounted to 44.14 (SD 2.79) nmol · l−1. An increase of power output of 30 W above CP-V˙O2 was accompanied by a significant increase in [H+]b above the resting level (P=0.03).
Accepted: 25 March 1998 相似文献
4.
Jerzy A. Zoladz Krzysztof Duda Joanna Majerczak 《European journal of applied physiology and occupational physiology》1998,77(5):445-451
A group of 12 healthy non-smoking men [mean age 22.3 (SD 1.1) years], performed an incremental exercise test. The test started
at 30 W, followed by increases in power output (P) of 30 W every 3 min, until exhaustion. Blood samples were taken from an antecubital vein for determination of plasma concentration
lactate [La−]pl and acid-base balance variables. Below the lactate threshold (LT) defined in this study as the highest P above which a sustained increase in [La−]pl was observed (at least 0.5 mmol · l−1 within 3 min), the pulmonary oxygen uptake (V˙O2) measured breath-by-breath, showed a linear relationship with P. However, at P above LT [in this study 135 (SD 30) W] there was an additional accumulating increase in V˙O2 above that expected from the increase in P alone. The magnitude of this effect was illustrated by the difference in the final P observed at maximal oxygen uptake (V˙O2max) during the incremental exercise test (P
max,obs at V˙O2max) and the expected power output at V˙O2max(P
max,exp at V˙O2max) predicted from the linear V˙O2-P relationship derived from the data collected below LT. The P
max,obs at V˙O2max amounting to 270 (SD 19) W was 65.1 (SD 35) W (19%) lower (P<0.01) than the P
max,exp at V˙O2max
. The mean value of V˙O2max reached at P
max,obs amounted to 3555 (SD 226) ml · min−1 which was 572 (SD 269) ml · min−1 higher (P<0.01) than the V˙O2 expected at this P, calculated from the linear relationship between V˙O2 and P derived from the data collected below LT. This fall in locomotory efficiency expressed by the additional increase in V˙O2, amounting to 572 (SD 269) ml O2 · min−1, was accompanied by a significant increase in [La−]pl amounting to 7.04 (SD 2.2) mmol · l−1, a significant increase in blood hydrogen ion concentration ([H+]b) to 7.4 (SD 3) nmol · l−1 and a significant fall in blood bicarbonate concentration to 5.78 (SD 1.7) mmol · l−1, in relation to the values measured at the P of the LT. We also correlated the individual values of the additional V˙O2 with the increases (Δ) in variables [La−]pl and Δ[H+]b. The Δ values for [La−]pl and Δ[H+]b were expressed as the differences between values reached at the P
max,obs at V˙O2max and the values at LT. No significant correlations between the additional V˙O2 and Δ[La−]pl on [H+]b were found. In conclusion, when performing an incremental exercise test, exceeding P corresponding to LT was accompanied by a significant additional increase in V˙O2 above that expected from the linear relationship between V˙O2 and P occurring at lower P. However, the magnitude of the additional increase in V˙O2 did not correlate with the magnitude of the increases in [La−]pl and [H+]b reached in the final stages of the incremental test.
Accepted: 30 October 1997 相似文献
5.
Y. Bhambhani R. Maikala S. Buckley 《European journal of applied physiology and occupational physiology》1998,78(5):422-431
The purpose of this study was to compare the rates of muscle deoxygenation in the exercising muscles during incremental arm
cranking and leg cycling exercise in healthy men and women. Fifteen men and 10 women completed arm cranking and leg cycling
tests to exhaustion in separate sessions in a counterbalanced order. Cardiorespiratory measurements were monitored using an
automated metabolic cart interfaced with an electrocardiogram. Tissue absorbency was recorded continuously at 760 nm and 850 nm
during incremental exercise and 6 min of recovery, with a near infrared spectrometer interfaced with a computer. Muscle oxygenation
was calculated from the tissue absorbency measurements at 30%, 45%, 60%, 75% and 90% of peak oxygen uptake (V˙O2) during each exercise mode and is expressed as a percentage of the maximal range observed during exercise and recovery (%Mox).
Exponential regression analysis indicated significant inverse relationships (P < 0.01) between %Mox and absolute V˙O2 during arm cranking and leg cycling in men (multiple R = −0.96 and −0.99, respectively) and women (R =−0.94 and −0.99, respectively). No significant interaction was observed for the %Mox between the two exercise modes and
between the two genders. The rate of muscle deoxygenation per litre of V˙O2 was 31.1% and 26.4% during arm cranking and leg cycling, respectively, in men, and 26.3% and 37.4% respectively, in women.
It was concluded that the rate of decline in %Mox for a given increase in V˙O2 between 30% and 90% of the peak V˙O2 was independent of exercise mode and gender.
Accepted: 31 March 1998 相似文献
6.
7.
Patrick Mucci Jacques Prioux Maurice Hayot Michèle Ramonatxo Christian Préfaut 《European journal of applied physiology and occupational physiology》1998,77(4):343-351
Exercise-induced hypoxaemia (EIH) in master athletes may be related to a diminished exercise hyper- pnoea. The aim of this
study was to determine whether EIH is associated with a change in the sensitivity of the ventilation response to activation
of the central chemoreceptors. The ventilation response to CO2 was measured in nine elderly untrained men (UT) [mean age 66.3 (SEM 3.1) years] and nine master athletes (MA) [mean age 62.7
(SEM 0.8) years] at rest, during moderate exercise (40% maximal oxygen uptake, V˙O2max), and during strenuous exercise (70% V˙O2max) using the rebreathing method. Our results showed that the ventilation response to CO2 did not differ with endurance training and/or exercise, that the threshold of the CO2 response (Th) increased with exercise (P < 0.001), that the increase in Th in MA was higher than in UT between rest and moderate exercise [ΔTh0–40: 8.55 (SEM 1.8) vs 3.06 (SEM 1.72) mmHg, P < 0.05], and that ΔTh0–40 and Th during moderate exercise were negatively correlated with arterial O2 saturation during maximal exercise (r = 0.50, P<0.05). We concluded therefore that exercise-induced hypoxaemia in master athletes may not be due to a lower ventilation response
to CO2, but may be partly related to a greater increase in Th during moderate exercise.
Accepted: 18 August 1997 相似文献
8.
David V. B. James Jonathan H. Doust 《European journal of applied physiology and occupational physiology》1998,77(6):551-555
Eight male endurance runners [mean ± (SD): age 25 (6) years; height 1.79 (0.06) m; body mass 70.5 (6.0) kg; % body fat 12.5
(3.2); maximal oxygen consumption (V˙O2max 62.9 (1.7) ml · kg−1 · min−1] performed an interval training session, preceded immediately by test 1, followed after 1 h by test 2, and after 72 h by
test 3. The training session was six 800-m intervals at 1 km · h−1 below the velocity achieved at V˙O2max with 3 min of recovery between each interval. Tests 1, 2 and 3 were identical, and included collection of expired gas, measurement
of ventilatory frequency (f
v
), heart rate (f
c), rate of perceived exertion (RPE), and blood lactate concentration ([La−]B) during the final 5 min of 15 min of running at 50% of the velocity achieved at V˙O2max (50% −V˙O2max).␣Oxygen uptake (V˙O2), ventilation (V˙
E
), and respiratory exchange ratio (R) were subsequently determined from duplicate expired gas collections. Body mass and plasma volume changes were measured preceding
and immediately following the training session, and before tests 1–3. Subjects ingested water immediately following the training
session, the volume of which was determined from the loss of body mass during the session. Repeated measures analysis of variance
with multiple comparison (Tukey) was used to test differences between results. No significant differences in body mass or
plasma volume existed between the three test stages, indicating that the differences recorded for the measured parameters
could not be attributed to changes in body mass or plasma volume between tests, and that rehydration after the interval training
session was successful. A significant (P < 0.05) increase was found from test 1 to test 2 [mean (SD)] for V˙O2 [2.128 (0.147) to 2.200 (0.140) 1 · min−1], f
c [125 (17) to 132 (16) beats · min−1], and RPE [9 (2) to 11 (2)]. A significant (P < 0.05) decrease was found for submaximal R [0.89 (0.03) to 0.85 (0.04)]. These results suggest that alterations in V˙O2 during moderate-intensity, constant-velocity running do occur following heavy-intensity endurance running training, and that
this is due to factors in addition to changed substrate metabolism towards greater fat utilisation, which could explain only
31% of the increase in V˙O2.
Accepted: 8 December 1997 相似文献
9.
Thierry Weissland P. Pelayo Jacques Vanvelcenaher Ga?lle Marais Jean-Marc Lavoie Hervé Robin 《European journal of applied physiology and occupational physiology》1997,76(5):428-433
The aims of the present study were: first, to assess the interindividual variations of a spontaneously chosen crank rate
(SCCR) in relation to the power developed during an incremental upper body exercise on an arm ergometer set at a constant
power regime, and second, to compare heart rate (HR) responses, expired minute ventilation (V˙
E) and oxygen consumption (V˙O2) when the pedal rates were chosen spontaneously (TSCCR) or set at ±10% of the freely chosen rates (T+10% and T−10%, respectively). The mean pedal rate values were linearly related (P < 0.01) with the power developed during arm cranking (r = 0.96), although large variations of pedalling rate strategies were observed between subjects. Maximal power (MP) and time
to exhaustion values were significantly higher (P < 0.05) during TSCCR than during T+10% and T−10%. Peak V˙O2 values were significantly higher (P < 0.05) in T+10% than in TSCCR and T−10%. The increase in HR, V˙
E, and V˙O2 mean values, in relation to the increase in the power developed, was significantly higher (P < 0.05) when the pedal rate was set at plus 10% of the SCCR (T±10%) than in the two other conditions. The findings of the present study suggest that the use of an electromagnetically braked
ergometer, which automatically adjusts the resistance component to maintain a constant work rate, should be used in order
to achieve the highest MP values during an incremental upper body exercise. A 10% increase of the SCCR should be used in order
to provide the highest peak V˙O2 value.
Accepted: 5 May 1997 相似文献
10.
M. Faina V. Billat R. Squadrone M. De Angelis J. P. Koralsztein A. Dal Monte 《European journal of applied physiology and occupational physiology》1997,76(1):13-20
Using 23 elite male athletes (8 cyclists, 7 kayakists, and 8 swimmers), the contribution of the anaerobic energy system to
the time to exhaustion (t
lim) at the minimal exercise intensity (speed or power) at which maximal oxygen uptake (V˙O2
max) occurs (I
V˙O2
max) was assessed by analysing the relationship between the t
lim and the accumulated oxygen deficit (AOD). After 10-min warming up at 60% of V˙O2
max, the exercise intensity was increased so that each subject reached his I
V˙O2max
in 30 s and then continued at that level until he was exhausted. Pre-tests included a continuous incremental test with 2 min
steps for determining the I
V˙O2max
and a series of 5-min submaximal intensities to collect the data that would allow the estimation of the energy expenditure
at I
V˙O2max
. The AOD for the t
lim exercise was calculated as the difference between the above estimation and the accumulated oxygen uptake. The mean percentage
value of energy expenditure covered by anaerobic metabolism was 15.2 [(SD 6)%, range 8.9–24.1] with significant differences
between swimmers and kayakists (16.8% vs 11.5%, P≤0.05) and cyclists and kayakists (16.4% vs 11.5%, P≤0.05). Absolute AOD values ranged from 26.4 ml · kg−1 to 83.6 ml · kg−1 with a mean value of 45.9 (SD 18) ml · kg−1. Considering all the subjects, the t
lim was found to have a positive and significant correlation with AOD (r = 0.62, P≤0.05), and a negative and significant correlation with V˙O2
max (r = −0.46, P≤0.05). The data would suggest that the contribution of anaerobic processes during exercise performed at I
V˙O2max
should not be ignored when t
lim is used as a supplementary parameter to evaluate specific adaptation of athletes.
Accepted: 17 December 1996 相似文献
11.
Douglas G. Bell Ira Jacobs Jiri Zamecnik 《European journal of applied physiology and occupational physiology》1998,77(5):427-433
This study investigated the effects of acute ingestion of caffeine (C), ephedrine (E) and their combination (C+E) on time
to exhaustion during high-intensity exercise. Using a repeated-measures, double-blind design, eight male subjects exercised
on a cycle ergometer at a power output that led to exhaustion after about 12.6 min during a placebo (P) control trial. They
did this 1.5 h after ingesting either C (5 mg · kg−1), E (1 mg · kg−1), C+E, or P. Trials were separated by 1 week. Venous blood was sampled before and during exercise. The mean (SD) times to
exhaustion were 12.6 (3.1) (P), 14.4 (4.1) (C), 15.0 (5.7) (E) and 17.5 (5.8) (C+E) min. Only the C+E treatment significantly
increased time to exhaustion compared to P. Oxygen consumption (V˙O2), carbon dioxide production (V˙CO2), minute ventilation (V˙
E) and the respiratory exchange ratio (RER) were similar during exercise for all trials. Heart rate during exercise was significantly
increased for the C+E and C trials compared to P. Subjective ratings of perceived exertion during exercise were significantly
lower after C+E compared to P. All treatments significantly increased lactate levels. Free fatty acid (FFA) levels were significantly
increased by C ingestion. Glycerol levels were increased by C+E and C ingestion. Glucose levels were also higher with the
drug treatments compared to P. Increased monamine availability after C+E treatment was suggested by measurements of catecholamines
and dopamine. In conclusion, the combination of C+E significantly prolonged exercise time to exhaustion compared to P, while
neither C nor E treatments alone significantly changed time to exhaustion. The improved performance was attributed to increased
central nervous system stimulation.
Accepted: 23 September 1997 相似文献
12.
Fifteen young adult Singaporean male physical education students maximum oxygen consumption [(V˙O2max) = 56 (4.7) ml · kg−1 · min−1] performed three prolonged runs in a counterbalanced design. The running bouts varied in time (40 vs 60 min) and intensity
(70% vs 80% V˙O2
max
). Each prolonged run was separated by 7 days. The running economy (RE) at 10.8 km · h−1 during 10-min running bouts was measured before (RE1) and after (RE2) each prolonged run. A control study involved monitoring
RE at 10.8 km · h−1 before and after 60 min rest. There were no differences between RE1 and RE2 values during the control run. However, there
were differences between RE1 and RE2 values when separated by a prolonged run. For example, the mean (SD) changes in oxygen
consumption (ml · kg−1 · min−1) values were 38.2 (2.5) versus 40.1 (2.6) (40 min at 80% V˙O2
max
), 38.9 (2.8) versus 41.5 (2.6) (60 min at 70% V˙O2
max
), and 39.0 (3.1) versus 42.7 (2.9) (60 min at 80% V˙O2
max
; P < 0.01). The results of this investigation support the hypothesis that RE deteriorates during prolonged running, and that
the magnitude of the deterioration in RE increases with both increasing exercise intensity and duration.
Accepted: 14 July 1997 相似文献
13.
This study investigated the effects on running economy (RE) of ingesting either no fluid or an electrolyte solution with
or without 6% carbohydrate (counterbalanced design) during 60-min running bouts at 80% maximal oxygen consumption (V˙O2max). Tests were undertaken in either a thermoneutral (22–23°C; 56–62% relative humidity, RH) or a hot and humid natural environment
(Singapore: 25–35°C; 66–77% RH). The subjects were 15 young adult male Singaporeans [V˙O2max = 55.5 (4.4 SD) ml kg−1 min−1]. The RE was measured at 3 m s−1 [65 (6)% V˙O2max] before (RE1) and after each prolonged run (RE2). Fluids were administered every 2 min, at an individual rate determined
from prior tests, to maintain body mass (group mean = 17.4 ml min−1). The V˙O2 during RE2 was higher (P < 0.05) than that during the RE1 test for all treatments, with no differences between treatments (ANOVA). The mean increase
in V˙O2 from RE1 to RE2 ranged from 3.4 to 4.7 ml kg−1 min−1 across treatments. In conclusion, the deterioration in RE at 3 m s−1 (65% V˙O2max) after 60 min of running at 80% V˙O2max appears to occur independently of whether fluid is ingested and regardless of whether the fluid contains carbohydrates or
electrolytes, in both a thermoneutral and in a hot, humid environment.
Accepted: 30 October 1997 相似文献
14.
Carlo Capelli Dave R. Pendergast B. Termin 《European journal of applied physiology and occupational physiology》1998,78(5):385-393
The energy cost per unit of distance (C
s, kilojoules per metre) of the front-crawl, back, breast and butterfly strokes was assessed in 20 elite swimmers. At sub-maximal
speeds (v), C
s was measured dividing steady-state oxygen consumption (V˙O2) by the speed (v, metres per second). At supra-maximal v, C
s was calculated by dividing the total metabolic energy (E, kilojoules) spent in covering 45.7, 91.4 and 182.9 m by the distance. E was obtained as: E = E
an+V˙O2max
t
p−V˙O2max(1−e−(
t
p/)), where E
an was the amount of energy (kilojoules) derived from anaerobic sources, V˙O2max litres per second was the maximal oxygen uptake, α (=20.9 kJ · l O2
−1) was the energy equivalent of O2, τ (24 s) was the time constant assumed for the attainment of V˙O2max at muscle level at the onset of exercise, and t
p (seconds) was the performance time. The lactic acid component was assumed to increase exponentially with t
p to an asymptotic value of 0.418 kJ · kg−1 of body mass for t
p ≥ 120 s. The lactic acid component of E
an was obtained from the net increase of lactate concentration after exercise (Δ[La]b) assuming that, when Δ[La]b = 1 mmol · l−1 the net amount of metabolic energy released by lactate formation was 0.069 kJ · kg−1. Over the entire range of v, front crawl was the least costly stroke. For example at 1 m · s−1, C
s amounted, on average, to 0.70, 0.84, 0.82 and 0.124 kJ · m−1 in front crawl, backstroke, butterfly and breaststroke, respectively; at 1.5 m · s−1, C
s was 1.23, 1.47, 1.55 and 1.87 kJ · m−1 in the four strokes, respectively. The C
s was a continuous function of the speed in all of the four strokes. It increased exponentially in crawl and backstroke, whereas
in butterfly C
s attained a minimum at the two lowest v to increase exponentially at higher v. The C
s in breaststroke was a linear function of the v, probably because of the considerable amount of energy spent in this stroke for accelerating the body during the pushing
phase so as to compensate for the loss of v occurring in the non-propulsive phase.
Accepted: 14 April 1998 相似文献
15.
A. S. Weller C. E. Millard P. L. Greenhaff I. A. Macdonald 《European journal of applied physiology and occupational physiology》1998,77(3):217-223
In a previous study, rectal temperature (T
re) was found to be lower, and oxygen consumption (V˙O2) and the respiratory exchange ratio (R) were higher in a cold (+5°C), wet and windy environment (COLD), compared with a thermoneutral environment during intermittent
walking at ≈30% of peak V˙O2 (Weller AS, Millard CE, Stroud MA et al. Am J Physiol 272:R226–R233, 1997). The aim of the present study was to establish
whether these cold-induced responses are influenced by prior fasting, as impaired thermoregulation has been demonstrated in
cold-exposed, resting men following a 48-h fast. To address this question, eight men attempted a 360-min intermittent (15 min
rest, 45 min exercise) walking protocol under COLD conditions on two occasions. In one condition, the subjects started the
exercise protocol ≈120 min after a standard meal (FED/COLD), whereas in the other the subjects had fasted for 36 h (FASTED/COLD).
The first two exercise periods were conducted at a higher intensity (HIGHER, 6 km · h−1 and 10% incline), than the four subsequent exercise periods (LOW, 5 km · h−1 and 0% incline). There was no difference in the time endured in FED/COLD and FASTED/COLD. In FASTED/COLD com pared with FED/COLD,
R was lower during HIGHER and LOW, and T
re was lower during LOW, whereas there was no difference in V˙O2, mean skin temperature and heart rate. Therefore, although the 36-h fast impaired temperature regulation during intermittent
low-intensity exercise in the cold, wet and windy environment, it was unlikely to have been the principal factor limiting
exercise performance under these experimental conditions.
Accepted: 26 August 1997 相似文献
16.
S. Grant W. Davidson T. Aitchison J. Wilson 《European journal of applied physiology and occupational physiology》1998,78(4):324-332
The aim of this study was to compare the exercise intensity and rating of perceived exertion (RPE) of a high-impact (HIP)
and a low-impact (LIP) university aerobic dance session. Ten women [mean (SD) age 22.9 (2.6) years] took part in the study.
An incremental treadmill test was performed by each subject to determine maximum oxygen consumption (V˙O2
max
) and maximum heart rate (HR
max
). The measured V˙O2
max
[mean (SD)] was 49.0 (7.5) ml · kg−1 · min−1. The subjects were randomly assigned to LIP and HIP sessions (i.e. five of the subjects participated in the HIP session first,
and the other five participated in the LIP session first). In a laboratory, heart rate, oxygen uptake and RPE were measured
throughout each session for each subject. Expired air was collected continuously throughout the sessions using Douglas bags
(ten bags over a 30-min period). The sessions consisted of 20 min of aerobic exercise (bags 1–7) followed by 5 min of local
muscular endurance exercise (bags 8 and 9) and 5 min of flexibility exercises (bag 10). The mean intensity of the aerobic
section of the LIP and HIP sessions was 51.6% and 64.7% V˙O2
max
, respectively. Ninety-five percent confidence intervals for the average difference between the HIP and LIP sessions demonstrate
that the %V˙O2
max
was between 12% and 14% higher for the HIP session. The mean %HR
max
for the LIP and HIP sessions was 71.4% and 76.7%, respectively, with the %HR
max
in the HIP session being between 5.4% and 7.2% higher on average than that of the LIP session. On average, the RPE for the
aerobic section of the HIP session (12.1) was consistently higher than that of the LIP session (11.1). HIP activity has the
potential to maintain/improve the aerobic fitness of its participants. According to the literature, the exercise intensity
elicited by LIP activity may have a limited training effect for the population utilised in this study, and for some individuals
may result in detraining. Conversely, LIP activities may be an appropriate mode of exercise for overweight and unfit individuals.
Accepted: 5 January 1998 相似文献
17.
M. Sandsund M. Sue-Chu J. Helgerud R. E. Reinertsen L. Bjermer 《European journal of applied physiology and occupational physiology》1998,77(4):297-304
The effects of whole-body exposure to ambient temperatures of −15°C and 23°C on selected performance-related physiological
variables were investigated in elite nonasthmatic cross-country skiers. At an ambient temperature of −15°C we also studied
the effects of the selective β2-adrenergic agonist Salbutamol (0.4 mg × 3) which was administered 10 min before the exercise test. Eight male cross-country
skiers with known maximal oxygen uptakes (V˙O2
max
) of more than 70 ml · kg−1 · min−1 participated in the study. Oxygen uptake (V˙O2), heart rate (f
c), blood lactate concentration ([La−]b) and time to exhaustion were measured during controlled submaximal and maximal running on a treadmill in a climatic chamber.
Lung function measured as forced expiratory volume in 1 s (FEV1) was recorded immediately before the warm-up period and at the conclusion of the exercise protocol. Submaximal V˙O2 and [La−]b at the two highest submaximal exercise intensities were significantly higher at −15°C than at 23°C. Time to exhaustion was
significantly shorter in the cold environment. However, no differences in V˙O2
max
or f
c were observed. Our results would suggest that exercise stress is higher at submaximal exercise intensities in a cold environment
and support the contention that aerobic capacity is not altered by cold exposure. Furthermore, we found that after Salbutamol
inhalation FEV1 was significantly higher than after placebo administration. However, the inhaled β2-agonist Salbutamol did not influence submaximal and maximal V˙O2, f
c, [La−]b or time to exhaustion in the elite, nonasthmatic cross-country skiers we studied. Thus, these results did not demonstrate
any ergogenic effect of the β2-agonist used.
Accepted: 18 August 1997 相似文献
18.
Keisho Katayama Yasitake Sato Koji Ishida Shigeo Mori Miharu Miyamura 《European journal of applied physiology and occupational physiology》1998,78(3):189-194
The present study was performed to investigate the effects of a combination of intermittent exposure to hypoxia during exercise
training for short periods on ventilatory responses to hypoxia and hypercapnia (HVR and HCVR respectively) in humans. In a
hypobaric chamber at a simulated altitude of 4,500 m (barometric pressure 432 mmHg), seven subjects (training group) performed
exercise training for 6 consecutive days (30 min · day−1), while six subjects (control group) were inactive during the same period. The HVR, HCVR and maximal oxygen uptake (V˙O2 max) for each subject were measured at sea level before (pre) and after exposure to intermittent hypoxia. The post exposure test
was carried out twice, i.e. on the 1st day and 1 week post exposure. It was found that HVR, as an index of peripheral chemosensitivity
to hypoxia, was increased significantly (P < 0.05) in the control group after intermittent exposure to hypoxia. In contrast, there was no significant increase in HVR
in the training group after exposure. The HCVR in both groups was not changed by intermittent exposure to hypoxia, while V˙O2 max increased significantly in the training group. These results would suggest that endurance training during intermittent exposure
to hypoxia depresses the increment of chemosensitivity to hypoxia, and that intermittent exposure to hypoxia in the presence
or absence of exercise training does not induce an increase in the chemosensitivity to hypercapnia in humans.
Accepted: 18 March 1998 相似文献
19.
Renza Perini Stefania Milesi Luca Biancardi David R. Pendergast Arsenio Veicsteinas 《European journal of applied physiology and occupational physiology》1998,77(4):326-332
Power spectrum analysis of heart-rate variability was made in seven men [mean age 22 (SEM 1) years] in head-out water immersion
(W) and in air (A, control) at rest and during steady-state cycling to maximal intensity (maximum oxygen uptake, V˙O2max). At rest W resulted in a trebled increase in the total power (P < 0.05), coupled with minimal changes in the power (as a percentage of the total) of the high frequency peak (HF, centred
at 0.26 Hz; 18% vs 28%) and of the low frequency peak (LF, 0.1 Hz; 24% vs 32%). A third peak at about 0.03 Hz (very low frequency,
VLF) represented the remaining power both in W and A. These changes as a whole indicated that immersion caused a vagal dominance
in cardiac autonomic interaction, due to the central pooling of blood and/or the pressure of water on the trunk. Exercise
caused a decrease in the total power in W and A. The LF% did not change up to about 50% V˙O2max, thereafter decreasing towards nil in both conditions. The HF% decreased in similar ways in W and A to about half at 55%–60%
V˙O2max and then increased to reach 1.5 times the resting values at V˙O2max. The central frequency of HF increased linearly with oxygen uptake, showing a tendency to be higher in W than in A at medium
to high intensities. The VLF% remained unchanged. The lack of differences in the LF peak between W and A during exercise would
suggest that blood distribution had no effect on the readjustments in control mechanisms of arterial pressure. On the other
hand, the findings of similar HF powers and the very similar values for ventilation in W and A confirmed the direct effect
of the respiratory activity in heart rate modulation during exercise.
Accepted: 25 August 1997 相似文献
20.
Stephen S. Cheung Tom M. McLellan 《European journal of applied physiology and occupational physiology》1998,78(1):50-58
The purpose of the present study was to determine the separate and combined effects of a short-term aerobic training program
and hypohydration on tolerance during light exercise while wearing nuclear, biological, and chemical protective clothing in
the heat (40°C, 30% relative humidity). Males of moderate fitness [<50 ml · kg−1 · min−1 maximal O2 consumption (V˙O2
max
)] were tested while euhydrated or hypohydrated by ≈2% of body weight through exercise and fluid restriction the day preceding
the trials. Tests were conducted before and after either a 2-week program of daily aerobic training (1 h treadmill exercise
at 65% V˙O2
max
for 12 days; n = 8) or a control period (n = 7), which had no effect on any measured variable. The training increased V˙O2
max
by 6.5%, while heart rate (f
c) and the rectal temperature (T
re) rise decreased during exercise in a thermoneutral environment. In the heat, training resulted in a decreased skin temperature
and increased sweat rate, but did not affect f
c, T
re or tolerance time (TT). In both training and control groups, hypohydration significantly increased T
re and f
c and decreased the TT. It was concluded that the short-term aerobic training program had no benefit on exercise-heat tolerance
in this uncompensable heat stress environment.
Accepted: 12 November 1997 相似文献