厚厉螨属四新种和一新纪录（蜱螨亚纲：厚厉螨科）

厚厉螨属Pachylaelaps Berles,1888全世界已有40余种,我国迄今报告过4种。本文记述5种厚厉螨,其中西西里厚厉螨Paehylaelaps siculus Berlese,1892为我国首次纪录,其他4种为新种:四联厚厉赌P.quadricombinatus sP.nov.、短毛厚厉螨P.brevisetosus sp.nov、疣足厚厉螭P.turocoxus sp.nov.和方形厚厉螨 P.quadratus sp.nov.。     

中国厚厉螨属四新记录种

记述中国厚厉螨属Pachylaelaps 4新记录种,即巨大厚厉螨Pachylaelaps grandis Koroleva、东方厚厉螨 Pachylaelaps orientalis Koroleva、天山厚厉螨Pachylaelaps tianschanicus Koroleva和布氏厚厉螨Pachylaelaps buyakovae Goncharova et Koroleva。标本保存于沈阳农业大学植物保护学院。     

克瓦厚厉螨雄螨及后若螨描述(蜱螨亚纲:革螨股:厚厉螨科)

克瓦厚厉螨（Pachylaelaps kievati Davydova,1971）,在所查到的献中仅描述了雌螨（KoponeBa,1977）,本描述其雄螨及后若螨。     

西藏厚厉螨属一新种和毛绥螨属一新种(蜱螨亚纲:厚厉螨科,裂胸螨科)

本文报道革螨二新种,西藏厚厉螨Pachylaelapsxizangensissp．nov．和亚东毛绥螨Lasioseiusyadongensissp．nov,1985年采自西藏亚东。     

厚厉螨科二新种(蜱螨亚纲：革螨股)

记述厚厉螨科二新种：光滑厚厉螨Pachylaelaps nuditectus sp. nov.和陈氏厚绥螨Pachyseius chenpengi sp. nov.。     

厚厉螨属一新种(蜱螨亚纲:革螨股)

记述厚厉螨属一新种,即枝沟厚厉螨Ｐａｃｈｙｌａｅｌａｐｓｒａｍｏｐｅｒｉｔｒｅｍａｔｕｓｓｐ．ｎｏｖ．。     

我国厚厉螨属二新纪录（蜱螨亚纲：厚厉螨科）

厚厉螨属Ｐａｃｈｙｌａｅｌａｐｓ Ｂｅｒｌｅｓｅ，１８８８我国前已记录有８种。最近我们在整理采自宁夏自治区的革螨中又发现２个国内新纪录种。     

土壤革螨3新种记述（蜱螨亚纲：厉螨科,寄螨科,厚厉螨科）

记述革螨3新种:(1)周氏下盾螨llypoaspis zhoumanshuae sp.nov.,(2)狭腹新革螨Neogamasus stenoventralis sp nov,(3)新梳厚厉螨Pachylaelaps neoxenillitus sp.nov.。     

中国厚厉螨科二新种(蜱螨亚纲,中气门亚目)(英文)

记述厚厉螨科2新种:长白厚厉螨Pachylaelaps changbaiensis sp.nov.和桓仁厚绥螨Pachyseius huanrenensis sp.nov.,模式标本保存于辽宁省沈阳农业大学植物保护学院.     

西藏厚厉螨属一新种和毛绥属一新种

本文报道革螨二新种,西藏厚厉螨Ｐａｃｈｙｌａｅｌａｐｓ ｘｉｚｎｇｅｎｓｉｓ ｓｐ,ｏｖ,和亚东毛绥螨Ｌａｓｉｏｓｅｉｕｓ ｙａｄｏｎｇｅｎｓｉｓ ｓｐ,ｎｏｖ。,１９８５采自西藏亚东。     

Quadruplexes of human telomere DNA analogs designed to contain G:A:G:A,G:G:A:A,and A:A:A:A tetrads

Replacement of two to four guanines by adenines in the human telomere DNA repeat dG₃(TTAG₃)₃ did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na⁺ and K⁺ solutions. Thermodynamic data showed that, in Na⁺ solution, the dG₃(TTAG₃)₃ quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K⁺ solution, the highest destabilization was caused by the 4A tetrad. In K⁺, only the unmodified dG₃(TTAG₃)₃ quadruplex rearranged into a K⁺‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.