共查询到20条相似文献,搜索用时 0 毫秒
1.
2.
3.
4.
5.
6.
7.
8.
9.
10.
Herbert J. Paterson 《BMJ (Clinical research ed.)》1912,1(2681):1158-1159
11.
12.
Victor H. Kuenkel 《The Western journal of medicine》1952,77(6):374-376
Gross and microscopic anatomical evidence indicates that pain fibers involved in causalgia are those distributed to blood vessels—possibly to the arterioles—and that, for the greater part, these fibers constitute part of the general visceral afferent system.Several investigators have reported evidence that injury to a peripheral nerve of such a type as to cause damage to the vasomotor control of any area produces the initial pain in an extremity, and it is predicated that the arteriolar constriction causing the pain is then prolonged by the sensitization of arteriolar smooth muscle to the amount of epinephrine normally in the blood. If the condition is not treated, tissue anoxia occurs to such an extent that irreversible changes take place in the affected area.Treatment of causalgia in the lower extremities is directed toward interruption of either the vasomotor or afferent supply of blood vessels by blocking or excision of the second to fourth lumbar ganglia inclusive with the intervening chains. For the upper extremities, the blocking or disconnection of the second and third thoracic ganglia with interruption of the sympathetic chain between the third and fourth ganglia is considered a feasible method of treatment which does not produce the concomitant disability of Horner''s syndrome. 相似文献
13.
14.
Percy T. Phillips 《The Western journal of medicine》1927,26(5):629-633
15.
16.
17.
18.
19.
A. L. Urquhart 《BMJ (Clinical research ed.)》1930,2(3629):139-140
20.