捕食者有病的生态-流行病模型的分析

建立并分析了捕食者具有疾病且有功能反应的生态-流行病(SI)模型,讨论了解的有界性．应用特征根法得到了平衡点局部渐近稳定的充分条件,进一步分析了平衡点的全局稳定性,得到了边界平衡点和正平衡点全局稳定的充分条件。     

一类食饵染病且垂直传染的生态流行病扩散模型的整体性态

本文研究一类具有空间扩散和食饵染病且垂直传染的的生态一流行病模型的在整体性态.首先讨论解的存在唯一性和一致有界性;其次由线性化方法得到了该模型非负平衡点的局部渐近稳定的充分条件,并构造恰当的Lyapunov泛函证明非负平衡点的全局渐近稳定性,得到了捕食者灭绝、疾病消失和疾病成为地方病的充分条件.     

双密度制约的Holling Ⅱ型捕食动力系统的定性分析

研究食饵具有非线性密度制约捕食者具有线性密度制约的HollingⅡ型捕食动力系统．以食饵的环境容纳量为分支参数,由Hopf分支得到小振幅极限环的存在性,同时也得到了正平衡点的全局稳定性和非小振幅极限环的存在唯一性的充分条件．     

一类捕食者具有阶段结构的捕食系统的全局稳定性分析

本文建立了一类捕食者具有阶段结构的捕食系统,计算得到了不存在食饵种群时捕食者种群模型和食饵种群存在时捕食系统的平衡点,并证明了平衡点的存在性.分析和比较了两个模型平衡点的全局稳定性,最终确定了决定模型全局稳定性的捕食者种群基本再生数、食饵灭绝与否的捕食率阈值以及捕食存在时食饵种群的净增长率.     

具有疾病传播的捕食与被捕食模型的动力学行为（英文）

本文提出了一类模拟捕食与被捕食传染病流行动力学特征的微分方程,其中疾病是在捕食者中传播.捕食者种群感染病毒形成易感者类和染病者类.在没有食饵及染病者种群存在时,捕食者按Logistic函数增长.文章研究了种群的持久性、灭绝性及边界平衡点的全局稳定性。通过数值模拟验证了理论分析结果的正确性.     

一类具连续时滞的捕食-被捕食生态系统

本文讨论了一类具有强连续时滞的捕食-被捕食模型,分析了各非负平衡点的稳定性,利用区域连续收缩方法,得出非负平衡点全局稳定的充分条件,给出正平衡点全局稳定的充分条件,并给出系统出现Hopf的分支值．     

捕食者有病的捕食—食饵扩散模型的稳定性分析

研究了一个捕食者有病,食饵具有Logistic增长的修正的Leslie-Gower功能反应的捕食-食饵扩散模型.应用线性化和Lyapunov泛函方法,获传染病灭绝的平衡点E_2局部渐近稳定和全局渐近稳定的充分条件.并通过数值模拟验证主要结论.     

具阶段结构及比率依赖型捕食系统的Hopf分支与稳定性

本文研究了一类食饵具有阶段结构的比率依赖型捕食模型的稳定性和Hopf分支的存在性问题.通过分析相应的特征方程,得到了平衡点局部稳定的充分条件,并指出当时滞穿过某特定值时正平衡点出现Hopf分支.利用比较定理与迭代方法证明了正平衡点的全局渐近稳定性,得到正平衡点全局渐近稳定的充分条件.最后,举例说明所得结果的可行性.     

食饵有病的生态-流行病模型的稳定性研究

在考虑捕食者捕食染病的食饵对自身的不利作用的基础上建立了食饵有病的生态-流行病模型,得到了系统平衡点局部渐近稳定的充分条件;讨论了系统的非负不变性、解的有界性,并在此基础上研究了边界平衡点的全局稳定性,得到了平衡点全局稳定的充分条件。     

具有年龄结构的食蚜蝇-蚜虫捕食模型

现有的具有年龄结构的捕食-食饵模型总是假设只有成年捕食者捕食猎物,这与实际情况不符。建立了一个幼年捕食者捕食食饵的具有年龄结构的食蚜蝇-蚜虫模型,应用微分方程定性理论,讨论了系统平衡点及其稳定性:其中平衡点E1(0,0,0)为不稳定的;满足一定条件时,边界平衡点E2(K,0,0)及正平衡点E3(x*,y1*,y2*)为局部渐近稳定的;且应用一致持续生存理论得到了系统永久持续生存的条件,为有害生物综合治理提供了理论依据。     

Multiple stable equilibria in a predator-prey system

Necessary and sufficient conditions are given for three equilibria to occur in a predatorprey model and conditions are given for two of these to be stable. The existence of two stable equilibria requires predator intraspecific competition for either space or food, and the lower the prey growth rate the stronger this predator self-regulation must be. A prey growth rate that is skewed to the right, the ability of a few predators to survive at low prey densities, and predators with high searching effectiveness, long handling times, and large maximum per capita rate of increase all make two stable equilibria more likely.     

Global stability and persistence in LG–Holling type II diseased predator ecosystems

A Leslie–Gower–Holling type II model is modified to introduce a contagious disease in the predator population, assuming that disease cannot propagate to the prey. All the system’s equilibria are determined and the behaviour of the system near them is investigated. The main mathematical issues are global stability and bifurcations for some of the equilibria, together with sufficient conditions for persistence of the ecosystem. Counterintuitive results on the role played by intraspecific competition are highlighted.     

Effect of disease-selective predation on prey infected by contact and external sources

We propose and analyze a simple mathematical model for susceptible prey (S)–infected prey (I)–predator (P) interaction, where the susceptible prey population (S) is infected directly from external sources as well as through contact with infected class (I) and the predator completely avoids consuming the infected prey. The model is analyzed to obtain different thresholds of the key parameters under which the system exhibits stability around the biologically feasible equilibria. Through numerical simulations we display the effects of external infection and the infection through contact on the system dynamics in the absence as well as in the presence of the predator. We compare the system dynamics when infection occurs only through contact, with that when it occurs through contact and external sources. Our analysis demonstrates that under a disease-selective predation, stability and oscillations of the system is determined by two key parameters: the external infection rate and the force of infection through contact. Due to the introduction of external infection, the predator and the prey population show limit-cycle oscillations over a range parametric values. We suggest that while predicting the dynamics of such an eco-epidemiological system, the modes of infection and the infection rates might be carefully investigated.     

A stage-structured predator–prey model with distributed maturation delay and harvesting

ABSTRACT

A stage-structured predator–prey system with distributed maturation delay and harvesting is investigated. General birth and death functions are used. The local stability of each feasible equilibria is discussed. By using the persistence theory, it is proven that the system is permanent if the coexistence equilibrium exists. By using Lyapunov functional and LaSalle invariant principle, it is shown that the trivial equilibrium is globally stable when the other equilibria are not feasible, and that the boundary equilibrium is globally stable if the coexistence equilibrium does not exist. Finally, sufficient conditions are derived for the global stability of the coexistence equilibrium.     

General Allee effect in two-species population biology

The main objective of this work is to present a general framework for the notion of the strong Allee effect in population models, including competition, mutualistic, and predator–prey models. The study is restricted to the strong Allee effect caused by an inter-specific interaction. The main feature of the strong Allee effect is that the extinction equilibrium is an attractor. We show how a ‘phase space core’ of three or four equilibria is sufficient to describe the essential dynamics of the interaction between two species that are prone to the Allee effect. We will introduce the notion of semistability in planar systems. Finally, we show how the presence of semistable equilibria increases the number of possible Allee effect cores.     

Dynamics of a intraguild predation model with generalist or specialist predator

Intraguild predation (IGP) is a combination of competition and predation which is the most basic system in food webs that contains three species where two species that are involved in a predator/prey relationship are also competing for a shared resource or prey. We formulate two intraguild predation (IGP: resource, IG prey and IG predator) models: one has generalist predator while the other one has specialist predator. Both models have Holling-Type I functional response between resource-IG prey and resource-IG predator; Holling-Type III functional response between IG prey and IG predator. We provide sufficient conditions of the persistence and extinction of all possible scenarios for these two models, which give us a complete picture on their global dynamics. In addition, we show that both IGP models can have multiple interior equilibria under certain parameters range. These analytical results indicate that IGP model with generalist predator has “top down” regulation by comparing to IGP model with specialist predator. Our analysis and numerical simulations suggest that: (1) Both IGP models can have multiple attractors with complicated dynamical patterns; (2) Only IGP model with specialist predator can have both boundary attractor and interior attractor, i.e., whether the system has the extinction of one species or the coexistence of three species depending on initial conditions; (3) IGP model with generalist predator is prone to have coexistence of three species.     

Competitive divergence in non-random mating populations

A haploid model of frequency-dependent selection and assortative mating is introduced and analyzed for the case of a single multiallelic autosomal locus. Frequency-dependent selection is due to intraspecific competition mediated by a quantitative character under stabilizing or directional selection. Assortment is induced by the same trait. We analyze the equilibrium structure and the local stability properties of all possible equilibria. In the limit of weak selection we obtain global stability properties by finding a Lyapunov function. We provide necessary and sufficient conditions for the maintenance of polymorphism in terms of the strength of stabilizing selection, intraspecific competition and assortment. Our results also include criteria for the ability of extreme types to invade the population. Furthermore, we study the occurrence of disruptive selection and provide necessary and sufficient conditions for intraspecific divergence to occur.     

Oscillations in a size-structured prey-predator model

This article introduces a predator–prey model with the prey structured by body size, based on reports in the literature that predation rates are prey-size specific. The model is built on the foundation of the one-species physiologically structured models studied earlier. Three types of equilibria are found: extinction, multiple prey-only equilibria and possibly multiple predator–prey coexistence equilibria. The stabilities of the equilibria are investigated. Comparison is made with the underlying ODE Lotka–Volterra model. It turns out that the ODE model can exhibit sustain oscillations if there is an Allee effect in the net reproduction rate, that is the net reproduction rate grows for some range of the prey’s population size. In contrast, it is shown that the structured PDE model can exhibit sustain oscillations even if the net reproductive rate is strictly declining with prey population size. We find that predation, even size-non-specific linear predation can destabilize a stable prey-only equilibrium, if reproduction is size specific and limited to individuals of large enough size. Furthermore, we show that size-specific predation can also destabilize the predator–prey equilibrium in the PDE model. We surmise that size-specific predation allows for temporary prey escape which is responsible for destabilization in the predator–prey dynamics.