A diabetic milieu increases ACE2 expression and cellular susceptibility to SARS-CoV-2 infections in human kidney organoids and patient cells

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1-deoxysphingolipids bind to COUP-TF to modulate lymphatic and cardiac cell development

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Soluble ACE2-mediated cell entry of SARS-CoV-2 via interaction with proteins related to the renin-angiotensin system

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Crosstalk between ILC2s and Th2 cells varies among mouse models

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Structural basis of human ACE2 higher binding affinity to currently circulating Omicron SARS-CoV-2 sub-variants BA.2 and BA.1.1

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Challenges and Reinterpretation of Antibody-Based Research on Phosphorylation of Tyr307 on PP2Ac

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Lyso-PAF,a biologically inactive phospholipid,contributes to RAF1 activation

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Vangl2 limits chaperone-mediated autophagy to balance osteogenic differentiation in mesenchymal stem cells

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Differential m6A RNA landscapes across hematopoiesis reveal a role for IGF2BP2 in preserving hematopoietic stem cell function

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Histone H2AK119 Mono-Ubiquitination Is Essential for Polycomb-Mediated Transcriptional Repression

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Nesprin-2 Recruitment of BicD2 to the Nuclear Envelope Controls Dynein/Kinesin-Mediated Neuronal Migration In Vivo

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Decoding m6A RNA methylome identifies PRMT6-regulated lipid transport promoting AML stem cell maintenance

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An epithelial-immune circuit amplifies inflammasome and IL-6 responses to SARS-CoV-2

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PP2AC Phospho-Tyr307 Antibodies Are Not Specific for this Modification but Are Sensitive to Other PP2AC Modifications Including Leu309 Methylation

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Tissue-resident FOLR2+ macrophages associate with CD8+ T cell infiltration in human breast cancer

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A BIN2-GLK1 Signaling Module Integrates Brassinosteroid and Light Signaling to Repress Chloroplast Development in the Dark

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CCL2 and CXCL12 Derived from Mesenchymal Stromal Cells Cooperatively Polarize IL-10+ Tissue Macrophages to Mitigate Gut Injury

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FOXA2 controls the anti-oxidant response in FH-deficient cells

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Enhanced SARS-CoV-2 entry via UPR-dependent AMPK-related kinase NUAK2

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SARS-CoV-2 infects human pancreatic β cells and elicits β cell impairment

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