CYP2E1 Rsa Ι/Pst Ι polymorphism and lung cancer susceptibility: A meta‐analysis involving 10,947 subjects

The above article, published in the Journal of Cellular and Molecular Medicine on 14 September 2016 in Wiley Online Library (wileyonlinelibrary.com), and in Volume 19, pp. 2136‐2142, has been retracted by agreement between the authors, the journal Editor in Chief, Stefan Constantinescu, and John Wiley & Sons Ltd. The retraction has been agreed due to unattributed overlap of the language used in the “Materials and method” and “Discussion” sections of this study and the following article published in Lung Cancer: “CYP2E1 Rsa I/Pst I polymorphism is associated with lung cancer risk among Asians” by Ping Zhan, Jing Wang, Yu Zhang, Li‐Xin Qiu, Su‐feng Zhao, Qian Qian, Shu‐Zhen Wei, Li‐Ke Yu and Yong Song, Volume 69, 2010, pages 19‐25. REFERENCE Shen Z‐T, Wu X‐H, Li B, Shen J‐S, Wang Z, Li J, Zhu X‐X. CYP2E1 Rsa Ι/Pst Ι polymorphism and lung cancer susceptibility: a meta‐analysis involving 10,947 subjects. J Cell Mol Med. 2015;19:2136‐2142. https://doi.org/10.1111/jcmm.12579     

Retraction: “MiR-137 functions as a tumor suppressor in pancreatic cancer by targeting MRGBP”

Retraction: “MiR-137 functions as a tumor suppressor in pancreatic cancer by targeting MRGBP” by Feng Ding, Shuang Zhang, Shaoyang Gao, Jian Shang, Yanxia Li, Ning Cui, and Qiu Zhao, J Cell Biochem. 2018; 4799-4807: The above article, published online on 13 January 2018 in Wiley Online Library ( https://doi.org/10.1002/jcb.26676 ) has been retracted by agreement between the journal's Editor in Chief, Prof. Dr. Christian Behl, and Wiley Periodicals LLC. The retraction has been agreed after the authors stated that errors occurred during figure compilation, and that the experimental data in the article could not be verified. The investigation additionally revealed inconsistencies in several image elements. Thus, the editors consider the conclusions of this article to be invalid.     

Retraction: 7, 8, 3’-Trihydroxyflavone protectsH/R-induced apoptosis and induces in vivo growth of human embryonic stem cell-derived cardiomyocytes

The above article, from Journal of Cellular Biochemistry, “7, 8, 3’-Trihydroxyflavone protects H/R-induced apoptosis and induces in vivo growth of human embryonic stem cell-derived cardiomyocytes” by Jiaxiang Li, Cuiping Wang, Bao Zhang, and Baiyun Tang published online on 20 May 2017 in Wiley Online Library (https://wileyonlinelibrary.com), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Retraction: miR-200a mediates protection of thymosin β4 in cardiac microvascular endothelial cells as a novel mechanism under hypoxia-reoxygenation injury

The above article, from Journal of Cellular Biochemistry, “miR-200a mediates protection of thymosin β4 in cardiac microvascular endothelial cells as a novel mechanism under hypoxia-reoxygenation injury” by Yang Li, Xiaolong Zhu, Xiping Liu, Aolin Du, and Bo Yu published online on 31 August 2017 in Wiley Online Library ( https://wileyonlinelibrary.com ), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Retracted: Extracting the practices of paleogenomics: A study of ancient DNA labs and research in relation to Native Americans and Indigenous peoples

Retraction: Cortez, A. D., Lippert, D., Davis, J. L., Nicholas, G., Malhi, R. S., Weyrich, L. S., Claw, K. G., Bader, A. C., & Colwell, C. (2023). Extracting the practices of paleogenomics: A study of ancient DNA labs and research in relation to Native Americans and Indigenous peoples. American Journal of Biological Anthropology. https://doi.org/10.1002/ajpa.24714 . The above article, published online on 17 February 2023 in Wiley Online Library ( wileyonlinelibrary.com ), has been retracted by agreement between the authors, the journal Editor in Chief, Trudy Turner, and Wiley Periodicals LLC. The retraction has been agreed due to a former collaborator voiding permission for use of their early contribution to the team project.     

Retraction: The Escherichia coli ycbQRST operon encodes fimbriae with laminin-binding and epithelial cell adherence properties in Shiga-toxigenic E. coli O157:H7

Samadder, P., Xicohtencatl-Cortes, J., Saldaña, Z., Jordan, D., Tarr, P.I., Kaper, J.B., & Girón, J.A. The Escherichia coli ycbQRST operon encodes fimbriae with laminin-binding and epithelial cell adherence properties in Shiga-toxigenic E.coli O157:H7. Environmental Microbiology, 11, 1815–1826. https://doi.org/10.1111/j.1462-2920.2009.01906.x The above article, published online 1 July 2009 on Wiley Online Library ( wileyonlinelibrary.com ), has been retracted by agreement between the authors, the journal Editor-in-Chief, Applied Microbiology International and John Wiley & Sons Ltd. The retraction has been agreed due to concerns raised by a third party regarding the appearance of Figures 2 and 7. Figure 7D appears to be digitally manipulated while Figure 7F is a duplication of Figure 2, Lane 4. The raw data were not available upon request. As a result, the data and the conclusions are considered unreliable.     

Retraction: microRNA-588 is upregulated in human prostate cancer with prognostic and functional implications

The above article, from Journal of Cellular Biochemistry, “microRNA-588 is upregulated in human prostate cancer with prognostic and functional implications” by Nan Zhao, Tao Lin, Changbo Zhao, Shikai Zhao, Shiming Zhou, and Yuqiao Li published online on 5 October 2017 in Wiley Online Library (wileyonlinelibrary.com), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Retraction: lncRNA CCAT1 contributes to the growth and invasion of gastric cancer via targeting miR-219-1

The above article, from Journal of Cellular Biochemistry, “lncRNA CCAT1 contributes to the growth and invasion of gastric cancer via targeting miR-219-1” by Yanfeng Li, Guanyu Zhu, Yan Ma, and Hongyan Qu published online on 12 December 2017 in Wiley Online Library (wileyonlinelibrary.com), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Retraction: Plin5 deficiency promotes atherosclerosis progression through accelerating inflammation,apoptosis and oxidative stress

The above article, from Journal of Cellular Biochemistry, “Plin5 deficiency promotes atherosclerosis progression through accelerating inflammation, apoptosis and oxidative stress” by Peng-Li Zhou, Min Li, Xin-Wei Han, Yong-Hua Bi, Wen-Guang Zhang, Zheng-Yang Wu, and Gang Wu published online on 7 December 2017 in Wiley Online Library (wileyonlinelibrary.com), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Upregulation of oxidative stress markers in human microvascular endothelial cells by complexes of serum albumin and digestion products of glycated casein

The extent of absorption of dietary advanced glycation end products (AGEs) is not fully known. The possible physiological impact of these absorbed components on inflammatory processes has been studied little and was the aim of this investigation. Aqueous solutions of bovine casein and glucose were heated at 95°C for 5 h to give AGE‐casein (AGE‐Cas). Simulated stomach and small intestine digestion of AGE‐Cas and dialysis (molecular mass cutoff of membrane = 1 kDa) resulted in a low molecular mass (LMM) fraction of digestion products, which was used to prepare bovine serum albumin (BSA)‐LMM‐AGE‐Cas complexes. Stimulation of human microvascular endothelial cells with BSA‐LMM‐AGE‐Cas complexes significantly increased mRNA expression of the receptor of AGE (RAGE), galectin‐3 (AGE‐R3), tumor necrosis factor alpha, and a marker of the mitogen‐activated protein kinase pathway (MAPK‐1), as well as p65NF‐κB activation. Cells treated with LMM digestion products of AGE‐Cas significantly increased AGE‐R3 mRNA expression. Intracellular reactive oxygen species production increased significantly in cells challenged with BSA‐LMM‐AGE‐Cas and LMM‐AGE‐Cas. In conclusion, in an in vitro cell system, digested dietary AGEs complexed with serum albumin play a role in the regulation of RAGE and downstream inflammatory pathways. AGE‐R3 may protect against these effects. © 2009 Wiley Periodicals, Inc. J Biochem Mol Toxicol 23:364–372, 2009; Published online in Wiley InterScience ( www.interscience.wiley.com ). DOI 10.1002/jbt.20301     

Retracted: Cav1.3 is upregulated in osteoporosis rat model and promotes osteoclast differentiation from preosteoclast cell line RAW264.7

Retraction: ‘Cav1.3 is upregulated in osteoporosis rat model and promotes osteoclast differentiation from preosteoclast cell line RAW264.7' , by Ping Fan, Nan Hu, Xiuyuan Feng, Yining Sun, Dan Pu, Xiaohong Lv, Zhiming Hao, Yang Li, Wujun Xue, Lan He, J Cell Physiol. 2019; 12821–12827: The above article, published online on 11 February 2019 in Wiley Online Library ( https://onlinelibrary.wiley.com/doi/full/10.1002/jcp.27937 ), has been retracted by agreement between the authors, the journal's Editor in Chief, Prof. Dr. Gregg Fields, and Wiley Periodicals LLC. The retraction has been agreed after the authors asked to replace all the figures. The authors confirmed that the original figures are flawed but did not provide original raw files. An investigation revealed several duplications in figures 2, 3, and 4. Accordingly, the conclusions of this article are considered invalid.     

Processing of precursor interleukin 1 beta and inflammatory disease

The processing of precursor interleukin 1 beta (IL1 beta) by elastase, cathepsin G, and collagenase, the major proteases released at sites of inflammation, was investigated using recombinant pro-IL1 beta. Each of these proteases cleaved the 31-kDa inactive precursor to a form similar in size and specific activity (greater than 10(8) units/mg) to the 17-kDa mature protein isolated from activated monocytes. Elastase, collagenase, and cathepsin G cleaved the IL1 beta precursor at distinct sites which are amino-terminal to the monocyte-processing site, Ala-117 (Cameron, P., Lumjuco, G., Rodkey, J., Bennett, C., and Schmidt, J. A. (1985) J. Exp. Med. 162, 790-801). Amino-terminal sequencing of the products of digestion by elastase and cathepsin G determined that resultant active IL1 beta proteins contained an additional 13 or 3 amino acids relative to mature IL1 beta. Synovial fluid collected from patients with inflammatory polyarthritis and bronchoalveolar lavage fluid from patients with sarcoidosis supplied similar processing activity(s). Control fluids from patients who had no symptoms of inflammatory disease did not exhibit processing activity. Lavage fluids that processed precursor IL1 beta were demonstrated to contain cathepsin G and/or elastase activity, whereas controls were negative. Because a significant fraction of IL1 beta may be secreted from monocytes as the inactive 31-kDa precursor (Hazuda, D. J., Lee, J. C., and Young, P. R. (1988) J. Biol. Chem. 263, 8473-8479, Bomford, R., Absull, E., Hughes-Jenkins, C., Simpkin, D., and Schmidt, J. (1987) Immunology 62, 543-549, and Mizel, S. B. (1988) in Cellular and Molecular Aspects of Inflammation Poste, G., and Crooke, S., eds) pp. 75-93, Plenum Publishing Corp., New York), these results suggest that in vivo the IL1 beta precursor can be processed after secretion by any of several proteases released at inflammatory sites.     

Mitochondria in biology and medicine

Ever since the first diagnosis of a mitochondrial disease in 1959 (Ernster et al., 1959), the interest for mitochondrial cytopathies has continued to increase. Originally it was believed that the condition was very rare and primarily effected high-energy requiring tissues resulting in a select few pathologies (Luft, 1994). Since 1959, the understanding of mitochondrial cytopathies has evolved immensely and mitochondrial cytopathies are now known to be the largest group of metabolic diseases and to be resulting in a wide variety of pathologies. "Mitochondria in Biology and Medicine" was the title of the first annual conference of Society of Mitochondrial Research and Medicine - India. The conference was organized by A. S. Sreedhar, Keshav Singh and Kumarasamy Thangaraj, and was held at The Centre for Cellular and Molecular Biology (CCMB) Hyderabad, India, during 9-10 December 2011. The conference featured talks from internationally renowned scientists within the field of mitochondrial research and offered both students and fellow researchers a comprehensive update to the newest research within the field. This paper summarizes key outcomes of the presentations.     

Retraction

Retraction: “HCFU inhibits cervical cancer cells growth and metastasis by inactivating Wnt/β-catenin pathway” from Journal of Cellular Biochemistry by Ping Liu, Shuying Ma, Hua Liu, Huazhen Han, and Shanshan Wang published online on 12 December 2017 in Wiley Online Library (wileyonlinelibrary.com), has been withdrawn by agreement between the journal Editor in Chief Gary S. Stein and John Wiley and Sons, Inc. The retraction has been agreed because the authors are not responding to requests to finalize their article for publication in the journal as the Version of Record.     

Retracted: Nrf2: a novel therapeutic target in fragile X syndrome is modulated by NNZ2566

Retraction : “Nrf2: a novel therapeutic target in fragile X syndrome is modulated by NNZ2566” by R. M. J. Deacon, M. J. Hurley, C. M. Rebolledo, M. Snape, F. J. Altimiras, L. Farías, M. Pino, R. Biekofsky, L. Glass and P. Cogram. The above article, from Genes, Brain and Behavior, published online on 12th May 2017 in Wiley Online Library ( wileyonlinelibrary.com ), has been retracted by agreement between the journal Editor in Chief, Andrew Holmes and John Wiley & Sons Ltd. The retraction has been agreed as all authors cannot agree on a revised author order, and at least one author continues to dispute the original order. In this case, the original article is being retracted on the grounds that the journal does not have permission to publish. Reference: Deacon, R. M. J., Hurley, M. J., Rebolledo, C. M., Snape, M., Altimiras, F. J., Farías, L., Pino, M., Biekofsky, R., Glass, L. and Cogram, P. (2017), Nrf2: a novel therapeutic target in fragile X syndrome is modulated by NNZ2566. Genes, Brain and Behavior. doi:10.1111/gbb.12373.     

Immunochemical detection of advanced glycosylation end products in vivo.

Reducing sugars react with protein amino groups to form a diverse group of protein-bound moieties with fluorescent and cross-linking properties. These compounds, called advanced glycosylation end products (AGEs), have been implicated in the structural and functional alterations of proteins that occur during aging and long-term diabetes. Although several AGEs have been identified on the basis of de novo synthesis and tissue isolation procedures, the measurement of AGE compounds in vivo has remained difficult. As an approach to the study of AGE formation in vivo, we prepared polyclonal antiserum to an AGE epitope(s) which forms in vitro after incubation of glucose with ribonuclease (RNase). This antiserum proved suitable for the detection of AGEs which form in vivo. Both diabetic tissue and serum known to contain elevated levels of AGEs readily competed for antibody binding. Cross-reactivity studies revealed the presence of a common AGE epitope(s) which forms after the incubation of diverse proteins with glucose. Cross-reactive epitopes also formed with glucose 6-phosphate or fructose. These data suggest that tissue AGEs which form in vivo appear to contain a common immunological epitope which cross-reacts with AGEs prepared in vitro, supporting the concept that immunologically similar AGE structures form from the incubation of sugars with different proteins (Horiuchi, S., Araki, N., and Morino, Y. (1991) J. Biol. Chem. 266, 7329-7332). None of the known AGEs, such as 4-furanyl-2-furoyl-1H-imidazole, 1-alkyl-2-formyl-3,4-diglycosylpyrrole, pyrraline, carboxymethyllysine, or pentosidine, were found to compete for binding to anti-AGE antibody. These data further suggest that the dominant AGE epitope which forms from the reaction of glucose with proteins under native conditions is immunologically distinct from the structurally defined AGEs described to date.     

Methode und Gerät zur Untersuchung des Wanderungsvermögens von Fritfliegenlarven

Schwantes, H.O.: Biologie der Pilze. Eine Einführung in die angewandte Mykologie. 478 S., 60 Abb., 29 Tab. Verlag Eugen Ulmer, Stuttgart, 1996, UNI‐Taschenbücher 1871, 42,80 DM. ISBN 3–8252–1871–6

Westhoff, P.; Jeske, H.; Jürgens, G.; Kloppstock, K.; Link, G.: Molekulare Entwicklungsbiologie. Vom Gen zur Pflanze. Georg Thieme Verlag Stuttgart, New York; 1996; 112 Abb., 19 Tab. ISBN 3–13–102021–0

McNamara, K.J. (Herausgeber): Evolutionary Change and Heterochrony. John Wiley &; Sons; Chichester, New York, Brisbane, Toronto, Singapur (1995), ISBN 0–471–95837–9

Sobral, B.W.S. (Herausgeber): The Impact of Plant Molecular Genetics. Birkhäuser Verlag AG Basel (1996), ISBN 3–7643–3802–4

McIntosh, R.A.; Wellings, C.R.; Park, R.F.: Wheat rusts ‐ An atlas of resistance genes. 200 S., 119 Bildseiten, Kluwer Academic Publishers, Dordrecht, Boston, London, 1995, 125,00 Dfl., ISBN 0–7923–3430–2

Friedrich, G.; Rode, H. (Hsg.): Pflanzenschutz im integrierten Obstbau. Verlag Eugen Ulmer Stuttgart. 1996. 494 S., 128 Farbfotos, 94 Zeichnungen, 41 Tab., 3. völlig neubearbeitete Auflage. Preis 88,‐DM; ISBN 3–8001–5541–9

Nultsch, W.: Allgemeine Botanik. 10. neubearbeitete erweiterte Auflage. Georg Thieme Verlag Stuttgart New York. 1996, 602 Seiten, 234 Abb. in 525 Einzeldarstellungen, 19 Boxen. Glossarium mit 752 Stichworten. Preis 44,‐DM; ISBN 3–13–383310–3

Berndt, J.: Umweltbiochemie. Uni‐Taschenbücher 1838. Gustav Fischer Verlag Stuttgart Jena. 1995; 278 S., 101 Abb., 33 Tab., Preis 34.80 DM. UTB‐ISBN 3–8252–1838–4

Kalusche, D.: Ökologie in Zahlen. Eine Datensammlung in Tabellen mit über 10.000 Einzelwerten. Gustav Fischer Verlag. Stuttgart, Jena, New York. 1996. 415 S. Preis 54, ‐DM; ISBN 3–437–20521–8