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CaC3H14 encoding a tandem CCCH zinc finger protein is directly targeted by CaWRKY40 and positively regulates the response of pepper to inoculation by Ralstonia solanacearum 总被引:1,自引:0,他引:1 下载免费PDF全文
Ailian Qiu Yufen Lei Sheng Yang Ji Wu Jiazhi Li Bingjin Bao Yiting Cai Song Wang Jinhui Lin Yuzhu Wang Lei Shen Jinsen Cai Deyi Guan Shuilin He 《Molecular Plant Pathology》2018,19(10):2221-2235
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Proteomics analysis of Medicago truncatula response to infection by the phytopathogenic bacterium Ralstonia solanacearum points to jasmonate and salicylate defence pathways 下载免费PDF全文
Ahad Yamchi Cécile Ben Michel Rossignol Sayed Reza Zareie Aghafakhr Mirlohi Badraldin Ebrahim Sayed‐Tabatabaei Carole Pichereaux Ahmad Sarrafi Martina Rickauer Laurent Gentzbittel 《Cellular microbiology》2018,20(4)
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A novel leucine‐rich repeat protein,CaLRR51, acts as a positive regulator in the response of pepper to Ralstonia solanacearum infection 下载免费PDF全文
Wei Cheng Zhuoli Xiao Hanyang Cai Chuanqing Wang Yang Hu Yueping Xiao Yuxing Zheng Lei Shen Sheng Yang Zhiqin Liu Shaoliang Mou Ailian Qiu Deyi Guan Shuilin He 《Molecular Plant Pathology》2017,18(8):1089-1100
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Involvement of salicylic acid,ethylene and jasmonic acid signalling pathways in the susceptibility of tomato to Fusarium oxysporum 下载免费PDF全文
Phytohormones, such as salicylic acid (SA), ethylene (ET) and jasmonic acid (JA), play key roles in plant defence following pathogen attack. The involvement of these hormones in susceptibility following Fusarium oxysporum (Fo) infection has mostly been studied in Arabidopsis thaliana. However, Fo causes vascular wilt disease in a broad range of crops, including tomato (Solanum lycopersicum). Surprisingly little is known about the involvement of these phytohormones in the susceptibility of tomato towards Fo f. sp. lycopersici (Fol). Here, we investigate their involvement by the analysis of the expression of ET, JA and SA marker genes following Fol infection, and by bioassays of tomato mutants affected in either hormone production or perception. Fol inoculation triggered the expression of SA and ET marker genes, showing the activation of these pathways. NahG tomato, in which SA is degraded, became hypersusceptible to Fol infection and showed stronger disease symptoms than wild‐type. In contrast, ACD and Never ripe (Nr) mutants, in which ET biosynthesis and perception, respectively, are impaired, showed decreased disease symptoms and reduced fungal colonization on infection. The susceptibility of the def1 tomato mutant, and a prosystemin over‐expressing line, in which JA signalling is compromised or constitutively activated, respectively, was unaltered. Our results show that SA is a negative and ET a positive regulator of Fol susceptibility. The SA and ET signalling pathways appear to act synergistically, as an intact ET pathway is required for the induction of an SA marker gene, and vice versa. 相似文献
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Xiufang Liu Yunzhi Song Fangyu Xing Ning Wang Fujiang Wen Changxiang Zhu 《Protoplasma》2016,253(5):1265-1281