共查询到19条相似文献,搜索用时 156 毫秒
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植物细胞受基因调控死亡,这种编程性定位的细胞死亡具有积极的生理功能,形成有利于自身发展的结构。基因调控细胞内的酶活动,细胞器产生变化,导致细胞死亡。外部因素可调节植物基因及基因调控产物的表达。 相似文献
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程序性细胞死亡是由基因调控的贯穿于真核细胞生理和发育过程的细胞自杀行为。动物细胞的程序性死亡分成3类凋亡、自噬和坏死;线粒体和溶酶体分别在前两个过程中起关键作用。关于植物细胞程序性死亡的分类还存在很多争议,焦点是植物是否有细胞凋亡这种形式,核心问题是植物细胞的线粒体外膜上没有Bcl-2家族的膜通透性调控蛋白。近年,程序性细胞死亡也在细菌中发现,LrgAB家族的膜通透性调控蛋白起着重要作用。最近的研究表明,植物叶绿体外被膜上也有LrgAB家族的同源蛋白,它们在控制叶绿体发育和程序性细胞死亡方面起重要作用。因此,叶绿体在植物细胞死亡调控中的作用应该更加受到关注。 相似文献
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细胞程序化死亡(PCD)是一种由基因控制的、主动的细胞死亡过程,它在植物正常生长发育过程中起着重要作用。本文就植物PCD的近期研究进展和其分子信号调控机制作一综合阐述。 相似文献
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植物中参与活性氧调控的基因网络 总被引:4,自引:0,他引:4
植物体内活性氧(reactive oxygen species,ROS)是氧化还原反应的必然副产物,具极高的活性和毒性,从而对细胞产生毒害。同时,活性氧作为信号分子对很多生理过程诸如植物生长发育、细胞程序化死亡及生物和非生物胁迫应答起调控作用。植物中ROS双重作用的协调机制目前尚不明确,确定的是细胞中ROS维持于稳定水平需要精细的调节。拟南芥中至少包括152个基因组成的网络参与ROS的调控,该网络具高度的灵活性和互补性。本文综述了ROS网络中鉴定的一些关键基因及细胞学定位和协同作用,ROS信号转导,尤其是叶绿体中ROS信号的调控。 相似文献
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维生素C过氧化物酶(ascorbate peroxidase,APX)是植物体内的重要酶系,是植物AsA-GSH氧化还原途径的重要组分,是清除H2O2(特别是叶绿体中的H2O2)的关键酶.本文综述了维生素C过氧化物酶表达调控方面的研究进展,包括逆境(干旱胁迫、空气污染、微量元素缺乏、离子胁迫、过度光强、照射以及盐胁迫等)与APX的表达调控、植物细胞程序性死亡(PCD)与APX的表达调控、植物生长发育与APX的表达调控、植物进化与APX表达调控等.植物体内的APX基因包括基质和类囊体两类,不同的APX基因序列存在一定差异,本文还综述了这两类APX基因在植物方面的分离和克隆进展情况,同时对APX基因的遗传转化进行了简要回顾,最后指出了APX今后的研究方向. 相似文献
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《中国细胞生物学学报》2018,(11)
植物在生长发育和抵抗逆境时都会发生细胞程序性死亡,植物受体激酶在细胞死亡调控中发挥着十分重要的作用。植物细胞可以通过质膜表面的受体激酶感受细胞间及环境信号,并将信号传递到下游,诱导一系列级联反应,导致细胞程序性死亡。植物受体激酶BAK1在植物程序性死亡中发挥着关键的作用, BAK1与BRI1、FLS2、BIR1、EFR、BIK1等受体激酶互作,识别和转导胞外信号,共同调控细胞死亡。该文以BAK1为中心,综述了近年发现的参与植物细胞死亡调控的BAK1受体激酶复合物介导的信号转导机制,并提出需要深入研究的科学问题。 相似文献
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Cytoplasmic male sterility: a window to the world of plant mitochondrial-nuclear interactions 总被引:2,自引:0,他引:2
Chase CD 《Trends in genetics : TIG》2007,23(2):81-90
Mitochondrial function depends on the coordinate action of nuclear and mitochondrial genomes. The genetic dissection of these interactions presents special challenges in obligate aerobes, because the viability of these organisms depends on mitochondrial respiration. The plant trait cytoplasmic male sterility (CMS) is determined by the mitochondrial genome and is associated with a pollen sterility phenotype that can be suppressed or counteracted by nuclear genes known as restorer-of-fertility genes. Here, I review the nature and the origin of the genes that determine CMS, together with recent investigations that have exploited CMS to provide new insights into plant mitochondrial-nuclear communication. These studies have implicated mitochondrial signaling pathways, including those involved in regulating cell death and nuclear gene expression, in the elaboration of CMS. The molecular cloning of nuclear genes that restore fertility (i.e. restorer-of-fertility genes) has identified genes encoding pentatricopeptide-repeat proteins as key regulators of plant mitochondrial gene expression. 相似文献
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Khanna HK Paul JY Harding RM Dickman MB Dale JL 《Molecular plant-microbe interactions : MPMI》2007,20(9):1048-1054
The death of plant cells in culture following exposure to Agrobacterium tumefaciens remains a major obstacle in developing Agrobacterium-mediated transformation into a highly efficient genotype-independent technology. Here, we present evidence that A. tumefaciens exposure induces cell death in banana cell suspensions. More than 90% of embryogenic banana cells died after exposure to A. tumefaciens and cell death was accompanied by a subset of features associated with apoptosis in mammalian cells, including DNA laddering, fragmentation, and formation of apoptotic-like bodies. Importantly, these cellular responses were inhibited in cells expressing the animal antiapoptosis genes Bcl-xL, Bcl-2 3' untranslated region, and CED-9. Inhibition of cell death resulted in up to 90% of cell clumps transformed with Bcl-xL, a 100-fold enhancement over vector controls, approaching the transformation and regeneration of every "transformable" cell. Similar results using sugarcane, a crop plant known for recalcitrance to Agrobacterium transformation, suggest that antiapoptosis genes may inhibit these phenomena and increase the transformation frequency of many recalcitrant plant species, including the major monocot cereal crop plants. Evidence of inhibition of plant cell death by cross-kingdom antiapoptotic genes also contributes to the growing evidence that genes for control of programmed cell death are conserved across wide evolutionary distances, even though these mechanisms are not well understood in plants. 相似文献
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Petr Babula Vojtěch Adam René Kizek Zdeněk Sladký Ladislav Havel 《Environmental and Experimental Botany》2009,65(2-3):330-337
Juglone and plumbagin are plant bioactive derivatives of 1,4-naphthoquinone occurring in plants, whereas lots of these plants belong to invasive species. Clarifying of action of juglone and plumbagin applied on plant cell model represented by tobacco BY-2 cells was the basic aim of this work. It was shown that naphthoquinones are able to induce various structural, functional and enzymatic changes leading to processes of apoptic-like cell death. Using dihydroethidium as fluorescent probe the mechanism of naphthoquinones action was explained. They are able to generate reactive oxygen species, which play important role in processes of programmed cell death. Disruption of mitochondrial respiratory chain was detected too. This study shown that mechanism of naphthoquinones action to plant cells is very complex and predestine them to be very effective compounds in plant competition fight. 相似文献
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The disease resistance signaling components EDS1 and PAD4 are essential regulators of the cell death pathway controlled by LSD1 in Arabidopsis 总被引:1,自引:0,他引:1
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Specific recognition of pathogens is mediated by plant disease resistance (R) genes and translated into a successful defense response. The extent of associated hypersensitive cell death varies from none to an area encompassing cells surrounding an infection site, depending on the R gene activated. We constructed double mutants in Arabidopsis between positive regulators of R function and a negative regulator of cell death, LSD1, to address whether genes required for normal R function also regulate the runaway cell death observed in lsd1 mutants. We report here that EDS1 and PAD4, two signaling genes that mediate some but not all R responses, also are required for runaway cell death in the lsd1 mutant. Importantly, this novel function of EDS1 and PAD4 is operative when runaway cell death in lsd1 is initiated through an R gene that does not require EDS1 or PAD4 for disease resistance. NDR1, another component of R signaling, also contributes to the control of plant cell death. The roles of EDS1 and PAD4 in regulating lsd1 runaway cell death are related to the interpretation of reactive oxygen intermediate-derived signals at infection sites. We further demonstrate that the fate of superoxide at infection sites is different from that observed at the leading margins of runaway cell death lesions in lsd1 mutants. 相似文献
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植物类病变突变体的诱发与突变机制 总被引:6,自引:0,他引:6
植物类病变突变体(lesion mimic mutant,LMM)是在无明显逆境或病原物侵染时,植物自发地形成类似病斑的一类突变体。它涉及到细胞程序性死亡(programmed cell death,PCD),往往能提高植物的抗病能力。因此,它对于揭示植物抗病反应机制,增加植物的广谱抗性具有重要意义。现就植物类病变突变体的诱发与表型特点、突变基因的分子定位与克隆及类病变表型的形成机制研究进展作一简要综述,以期为植物细胞程序性死亡机制和抗病分子作用机制研究提供有益的信息。 相似文献
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