中国寄蝇科狭颊寄蝇属研究

综合研究中国狭颊寄蝇属73种的鉴别方法、分布、寄主种类;研究并发现一些种类的变异特点,♀、♂异型等现象;文中附有73种的检索表及特征图,15个新种的描述;订正了5种为新异名。重建了狭颊寄蝇属的分类系统。     

中国寄蝇科狭颊寄蝇属研究(双翅目:寄蝇科)

综合研究中国狭颊寄蝇属73种的鉴别方法、分布、寄主种类;研究并发现一些种类的变异特点,♀、♂异型等现象;文中附有73种的检索表及特征图,15个新种的描述;订正了5种为新异名.重建了狭颊寄蝇属的分类系统.     

中国寄蝇二新种(双翅目:寄蝇科)

本文记述采自北京和云南的寄蝇二新种。新种模式标本保存于中国科学院动物研究所。 黑棒鞘寄蝇 Thecocarcalia melanohalterata, 新种(图1—5) 头、胸、腹全部黑色。侧额及胸部背面覆黄褐色粉被,其余部分覆灰色粉被;沿腹部背中线具一狭窄的黑纵条,粉被在腹部占第3背板基部的1/5,第4,5背板的1/2—3/5;上下腋瓣,平衡棒基半部、爪垫、唇瓣及雌蝇下颚须端半部黄褐色,平衡棒端半部黑褐色,翅灰色透明。     

中国寄蝇科的记述黄角寄蝇属新属

在整理研究埃内寄蝇族Ernestiini标本的过程中,发现一个新属,即黄角寄蝇属Flavicorniculum新属,分布于我国浙江、四川、广西、云南诸省。共包括四个新种,现记述于后。 所有模式标本均保存于中国科学院动物研究所。     

中国寄蝇科的记述Ⅵ.蜉寄蝇属

蜉寄蝇属Phorocera R.-D.,1830年,目前在世界范围内共包括4种,是寄蝇亚科中一个较小的属;根据我国现有标本已发现7种,其中有3个新种。本属的种类均为大卵型寄蝇,多寄生于鳞翅目害虫,特别是Ph.agilis R.-D.(=silvestris R.-D.)为舞毒蛾的重     

中国金绿寄蝇属记述：双翅目：寄蝇科

金绿寄蝇属Chrysocosmius在全世界已记载5种(古北界3种,东洋界2种),本文报道我国7种,其中包括4新种和2中国新纪录。新种为:巨眼鬃金绿寄蝇Chrysocosrmius ocellosetus,单鬃金绿寄蝇Chr.monostus,双齿金绿寄蝇Chr.bidentatus,亚合眼金绿寄蝇Chr.euholopticus。本文还编有种检索表。     

中国寄蝇科的记述Ⅷ.刺蛾寄蝇属

刺蛾寄蝇属Chaetexorista B.B.1894目前在世界范围内已记载有4种,即Ch.javanaB.B.1894,Ch.eutachinoides Bar.1932,Ch.solomonensis Bar.1936和Ch.klapperichiMesnil 1960;在我国,现共发现6种,其中有3个是新种,另有1种为我国初次记录。 本属寄蝇的寄主,文献中尚无记载,通过野外采集和室内饲养,我们得知,这一属的寄蝇是刺蛾科幼虫的重要天敌。 本文以记述新种为主,其余3种的主要特征仅在检索表内加以说明;对于它们的生物学特性和寄主种类,将在经济昆虫志内做详细介绍。新种的模式标本均保存于中国科学院动物研究所。     

中国寄蝇科的记述Ⅸ.豪寄蝇属Hystriomyia

豪寄蝇属Hystriomyia Portschinsky建立于1882年,至今记载的仅有3种;在我国又发现两个新种。本文以记述新种为主,为了便于识别,将其余3种的主要特征列入检索表中,并附图加以说明。新种的模式标本均保存于中国科学院北京动物研究所。 种检索表 1(2)体表的鬃与毛全部黑色:头部具外侧额鬃,肛尾叶长三角形,末端具1小齿(图Ⅰ):缝后翅内鬃3;第5腹板     

中国瑟寄蝇属种类纪要（双翅目：寄蝇科）

研究了我国蜗寄蝇族瑟寄蝇属Cyrtophleba(双翅目:寄蝇科)2种,其中茹瑟寄蝇Cyrtophleba ruricola 分布于辽宁,山西,宁夏,新疆;变瑟寄蝇Cyrtophleba vernalis为中国新记录种,分布于辽宁。给出了上述2种的鉴别特征及变瑟寄蝇头和雄性尾器的特征图。研究标本分别保存于沈阳师范大学昆虫标本室(SYNU),中国科学院动物研究所(IZCAS)和国家林业局森林病虫害防治总站(GSFPM)。     

中国利索寄蝇属的研究(双翅目:寄蝇科)

利索寄蝇属Lixophaga Townsend,1908隶于寄蝇科Tackinldae,卷蛾寄蝇族Blondeliinio该属大部分种类分布于新北界,古北界已描述的种类有3种,而且都采自日本,东洋界尚无记载。1964年,中国从古巴引进螟利索寄蝇L. diatmeae(Townsend),多年饲养未获成功。本文记述中国利索寄蝇属3种,其中一新种和二新纪录;为了便于比较和鉴定,把掌握有标本的另外两个种,即L.diatraeae(Townsend)和L. fallax Mesnil均     

Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis

It has now been over twenty years since a novel herpesviral genome was identified in Kaposi's sarcoma biopsies. Since then, the cumulative research effort by molecular biologists, virologists, clinicians, and epidemiologists alike has led to the extensive characterization of this tumor virus, Kaposi's sarcoma-associated herpesvirus(KSHV; also known as human herpesvirus 8(HHV-8)), and its associated diseases. Here we review the current knowledge of KSHV biology and pathogenesis, with a particular emphasis on new and exciting advances in the field of epigenetics. We also discuss the development and practicality of various cell culture and animal model systems to study KSHV replication and pathogenesis.     

The structure, reproduction and taxonomy of Vidalia and Osmundaria (Rhodophyta, Rhodomelaceae)

Comprises species occurring mostly in subtidal habitats in tropical, subtropical and warm-temperate areas of the world. An analysis of the type species, V. spiralis (Sonder) Lamouroux ex J. Agardh, a species from Australia, establishes basic characters for distinguishing species in the genus. These characters are (1) branching patterns of thalli, (2) flat blades that may be spiralled on their axis, (3) width of the blade, (4) primary or secondary derivation of sterile and fertile branchlets and (5) position of sterile and fertile branchlets on the thalli. Application of the latter two characters provides an important basic method for separation of species into three major groups. Osmundaria , a genus known only in southern Australia, was studied in relation to Vidalia , and its separation from the Vidalia assemblage is not accepted. Species of Vidalia therefore are transferred to the older genus name, Osmundaria. Two new species, Osmundaria papenfussii and Osmundaria oliveae are described from Natal. Confusion in the usage of the epithet, Vidalia fimbriala Brown ex Turner has been clarified, and Vidalia gregaria Falkenberg, described as an epiphyte on Osmundaria pro/ifera Lamouroux, is revealed to be young branches of the host, Osmundaria prolifera.     

New or rare chromosome counts in Artemisia L. (Asteraceae, Anthemideae) and related genera from Kazakhstan

Fifteen chromosome counts of six Artemisia taxa and one species of each of the genera Brachanthemum, Hippolytia, Kaschgaria, Lepidolopsis and Turaniphytum are reported from Kazakhstan. Three of them are new reports, two are not consistent with previous counts and the remainder are confirmations of very scarce (one to four) earlier records. All the populations studied have the same basic chromosome number, x = 9, with ploidy levels ranging from 2x to 6x. Some correlations between ploidy level, morphological characters and distribution are noted.     

肝癌中HBV和HCV基因和抗原的分布及意义

采用原位分子杂交方法检测HCV RNA及HBV X基因;采用免疫组织化学方法研究HCV核心抗原,非结构区C33c抗原及HBxAg在肝细胞肝癌中的定位及分布.结果表明(1)HCV RNA、HBV X基因在肝细胞肝癌组织检出率分别为40%(55/136)和82%(112/136).HCV RNA定位于癌细胞的胞浆内,阳性细胞呈散在、灶状及弥漫分布三种形式;HBV X基因在肝癌细胞中的分布呈胞浆型、核型及核浆型,阳性细胞也呈上述三种分布形式;(2)HCV C33c抗原、核心抗原在肝细胞肝癌中的阳性率为81%(133/164)及86%(141/164).C33c抗原定位于癌细胞及肝细胞的胞浆内;核心抗原既定位于癌细胞核中,又可定位于胞浆中.C33c抗原阳性细胞以灶状分布为主;而核心抗原阳性细     

Selection with two alleles and complete dominance

For a plant selection model with frequency-independent viabilities, fertilities and selfing rates, it is shown that apart from global fixation, for certain parameter combinations a protected polymorphism and facultative fixation (either allele may become fixed according to initial frequencies) may both occur. Facultative fixation requires different selling rates for the dominant and recessive type. Protection of the polymorphism requires resource allocation for male and female function. In this connection the problem of purely genetically caused population extinction is discussed.
For general frequency dependence and regular segregation, the chances for establishment of a completely recessive gene are compared to those of a completely dominant gene. It is proven that the process of establishment of the recessive gene, despite a fitness advantage, may be considerably endangered by drift effects if random mating prevails. The recessive gene may reach the same effectivity in establishment as a dominant gene, only if the recessive homozygote mates exclusively with its own type during the period of establishment.