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A gene capable of blocking apoptosis can substitute for the herpes simplex virus type 1 latency-associated transcript gene and restore wild-type reactivation levels. 总被引:9,自引:0,他引:9
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Guey-Chuen Perng Barak Maguen Ling Jin Kevin R. Mott Nelson Osorio Susan M. Slanina Ada Yukht Homayon Ghiasi Anthony B. Nesburn Melissa Inman Gail Henderson Clinton Jones Steven L. Wechsler 《Journal of virology》2002,76(3):1224-1235
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Workman A Eudy J Smith L da Silva LF Sinani D Bricker H Cook E Doster A Jones C 《Journal of virology》2012,86(5):2459-2473
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Leticia Frizzo da Silva Insun Kook Alan Doster Clinton Jones 《Journal of virology》2013,87(20):11214-11222
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Herpes simplex virus type 1 serum neutralizing antibody titers increase during latency in rabbits latently infected with latency-associated transcript (LAT)-positive but not LAT-negative viruses.
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Guey-Chuen Perng Susan M. Slanina Ada Yukht Homayon Ghiasi Anthony B. Nesburn Steven L. Wechsler 《Journal of virology》1999,73(11):9669-9672
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A mutation in the latency-related gene of bovine herpesvirus 1 inhibits protein expression from open reading frame 2 and an adjacent reading frame during productive infection
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The latency-related (LR) gene of bovine herpesvirus 1 (BHV-1) is abundantly expressed during latency. A mutant BHV-1 strain that contains three stop codons at the 5′ terminus of the LR gene (LR mutant) does not reactivate from latency. This study demonstrates that the LR mutant does not express open reading frame 2 or an adjacent reading frame that lacks an initiating ATG (reading frame C). Since the LR mutant and wild-type BHV-1 express similar levels of LR RNA, we conclude that LR protein expression plays an important role in regulating the latency reactivation cycle in cattle. 相似文献