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The invasive phenotype in HMT-3522 cells requires increased EGF receptor signaling through both PI 3-kinase and ERK 1,2 pathways
Authors:Price Daniel J  Avraham Shalom  Feuerstein Joseph  Fu Yigong  Avraham Hava Karsenty
Institution:  a Division of Experimental Medicine, Beth Israel-Deaconess Medical Center, Harvard Institutes of Medicine, Boston, MA.
Abstract:
Keywords:Egf  Breast Cancer  Pi 3-kinase  Erk 1  2  Cellular Invasion
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