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BRCA1 Haploinsufficiency Is Masked by RNF168-Mediated Chromatin Ubiquitylation
Authors:Dali Zong  Salomé Adam  Yifan Wang  Hiroyuki Sasanuma  Elsa Callén  Matilde Murga  Amanda Day  Michael J Kruhlak  Nancy Wong  Meagan Munro  Arnab Ray Chaudhuri  Baktiar Karim  Bing Xia  Shunichi Takeda  Neil Johnson  Daniel Durocher  André Nussenzweig
Institution:1. Laboratory of Genome Integrity, National Cancer Institute, NIH, Bethesda, MD, USA;2. The Lunenfeld-Tanenbaum Research Institute, Mount Sinai Hospital, Toronto, ON, Canada;3. Molecular Therapeutics Program, Fox Chase Cancer Center, Philadelphia, PA, USA;4. Department of Radiation Genetics, Graduate School of Medicine, Kyoto University, Kyoto, Japan;5. Genomic Instability Group, Spanish National Cancer Research Center, CNIO, Madrid, Spain;6. Laboratory of Cancer Biology and Genetics, National Cancer Institute, NIH, Bethesda, MD, USA;7. Department of Molecular Genetics, Erasmus University Medical Center, Rotterdam, the Netherlands;8. Pathology/Histotechnology Laboratory, Frederick National Laboratory for Cancer Research, Frederick, MD, USA;9. Department of Radiation Oncology, Rutgers Cancer Institute of New Jersey, New Brunswick, NJ, USA;10. Department of Molecular Genetics, University of Toronto, Toronto, ON, Canada
Abstract:
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  • Keywords:chromatin ubiquitylation  homologous recombination  cancer  RNF168  BRCA1  PALB2  RAD51  haploinsufficiency  genome stability  resection  replication fork protection
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