| 环境胁迫诱导的植物细胞程序性死亡 |
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| 引用本文： | 潘建伟,陈虹,顾青,朱睦元.环境胁迫诱导的植物细胞程序性死亡[J].遗传,2002,24(3):385-388. |
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| 作者姓名： | 潘建伟 陈虹 顾青 朱睦元 |
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| 作者单位： | 1.浙江大学生命科学学院，杭州 310012;
2.杭州商学院生物工程系，杭州，310035
1.College of Life Sciences,Zhejiang University,Hangzhou 310012,China；
2.Depantment of Bioengineering,Hangzhou University of Commerce,Hangzhou 310035,China |
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| 基金项目： | 国家自然科学基金 ( 39770 42 0 ,30 10 0 115 )，浙江省自然科学基金 ( 30 0 2 5 5 )，浙江大学百课工程建设项目 (B3-4 8) |
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| 摘 要： | 在最近的10年中，兴起了对植物细胞程序性死亡的研究。大量的证据表明，在各种环境胁迫因子诱导植物细胞PCD过程中，活性氧、乙烯、Ca2+、水杨酸、NO等成为重要的信号分子。像动物细胞凋亡一样，在植物PCD中也存在一条依赖于天冬氨酸特异性半胱氨酸蛋白酶(Caspases)活性的信号传导途径，其中，线粒体处于PCD调控的中心位置。
Abstract:Programmed cell death (PCD) research in higher plants has blossomed in the past ten years.Many evidences suggested that reactive oxygen species,ethylene,Ca2+,salicylic acid,nitric oxide etc.are important signal molecules during environmental stress-induced PCD in plants.Like apoptosis in animals,there also exists a Caspase-dependent PCD signal transduction pathway,in which mitochondrion plays a role of central depot.
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| 关 键 词： | 高等植物 环境胁迫因子 细胞程序性死亡 environmental stress factor 信号传导途径 Key words |
| 文章编号： | 0253-9772(2002)03-0385-04 |
| 修稿时间： | 2001年7月31日 |
Environmental Stress-Induced Programmed Cell Death in Higher Plants |
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| PAN Jian wei ,CHEN Hong ,GU Qing ,ZHU Mu yuan.Environmental Stress-Induced Programmed Cell Death in Higher Plants[J].Hereditas,2002,24(3):385-388. |
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| Authors: | PAN Jian wei CHEN Hong GU Qing ZHU Mu yuan |
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| Institution: | College of Life Sciences, Zhejiang University, Hangzhou 310012, China. |
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| Abstract: | Programmed cell death (PCD) research in higher plants has blossomed in the past ten years.Many evidences suggested that reactive oxygen species,ethylene,Ca 2+ ,salicylic acid,nitric oxide etc.are important signal molecules during environmental stress-induced PCD in plants.Like apoptosis in animals,there also exists a Caspase-dependent PCD signal transduction pathway,in which mitochondrion plays a role of central depot. |
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| Keywords: | environmental stress factor higher plant programmed cell death signal transduction pathway |
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