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Overexpression of DNA ligase III in mitochondria protects cells against oxidative stress and improves mitochondrial DNA base excision repair
Institution:1. Center for Healthy Aging, SUND, University of Copenhagen, Denmark;2. Laboratory of Molecular Gerontology, National Institute on Aging, 251 Bayview Boulevard, Baltimore, USA;1. China CDC Key Laboratory of Radiological Protection and Nuclear Emergency, National Institute for Radiological Protection, Chinese Center for Disease Control and Prevention, Beijing 100088, PR China;2. Department of Cardiology, Beijing Chao-Yang Hospital, Capital Medical University, Beijing 100020, PR China;1. Graduate Program in University of Brasilia, Health Sciences and Technologies, Brasilia, Brazil;2. JAXA/Institute of Space and Astronautical Science, Sagamihara, Japan;3. Department of Hard Tissue Engineering, Tokyo Medical and Dental University, Tokyo, Japan;1. Division of Medical Oncology, University of Ottawa Faculty of Medicine and The Ottawa Hospital Cancer Centre, Ottawa, Ontario, Canada;2. Centre for Cancer Therapeutics, Ottawa Hospital Research Institute, Ottawa, Ontario, Canada;3. Regenerative Medicine Program, Ottawa Hospital Research Institute, Ottawa, Ontario, Canada;4. Department of Biochemistry, University of Ottawa Faculty of Medicine, Ottawa, Ontario, Canada;1. National Key Laboratory of Crop Genetics and Germplasm Enhancement, College of Horticulture, Nanjing Agricultural University, Nanjing 210095, PR China;2. Department of Plant Sciences, North Dakota State University, Fargo, ND 58108, USA
Abstract:
Keywords:Mitochondrial DNA repair intermediates  Oxidative stress  Autophagy  Cell survival
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