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AKT1 distinctively suppresses MyD88-depenedent and TRIF-dependent Toll-like receptor signaling in a kinase activity-independent manner
Institution:1. Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia;2. Department of Pharmacology and Toxicology, College of Pharmacy, Al-Azhar University, Cairo, Egypt;3. Department of Pharmaceutics, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia
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