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Thyroid hormone receptors and cancer

Authors:	Won Gu Kim Sheue-yann Cheng

Affiliation:	Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20892, USA

Abstract:

Keywords:	BM, basement membrane ECM, extracellular membrane EMT, epithelial mesenchymal transition FAK, focal adhesion kinase MAPK, mitogen-activated protein kinase LOH, loss of heterozygosity NCOR1, nuclear receptor co-repressor 1 MMP, matrix metalloproteinase mTOR, mammalian target of rapamycin PAX8, paired box gene 8 PIP2, phosphatidylinositol-4,5-biphosphate PIP3, phosphatidylinositol-3,4,5-triphosphate PI3K, phosphatidylinositol 3-kinase PPARγ, peroxisome proliferator-activated receptor γ PPRE, peroxisome proliferator responsive element PPTG, pituitary tumor-transforming gene PTEN, phosphatase and tensin homologue deleted from chromosome 10 RTH, resistance to thyroid hormone RXRα, retinoid X receptor α SH, Src homology SKY, spectral karyotyping analysis SMRT, silencing mediator of retinoid and thyroid hormone receptors SRC, steroid receptor co-activator TREs, thyroid hormone response elements TRs, thyroid hormone nuclear receptors T3, triiodothyronine TSH, thyroid stimulating hormone
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