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Heat shock factor 1 over-expression protects against exposure of hydrophobic residues on mutant SOD1 and early mortality in a mouse model of amyotrophic lateral sclerosis
Authors:Pei-Yi?Lin  Sharotka?M?Simon  Won?Kyun?Koh  Oluwarotimi?Folorunso  C?Samuel?Umbaugh  Email author" target="_blank">Anson?PierceEmail author
Institution:1.Department of Biochemistry & Molecular Biology,University of Texas Medical Branch,Galveston,USA;2.Department of Neuroscience and Cell Biology,University of Texas Medical Branch,Galveston,USA;3.Sealy Center for Vaccine Development,University of Texas Medical Branch,Galveston,USA;4.George and Cynthia Woods Mitchell Center for Neurodegenerative Diseases,University of Texas Medical Branch,Galveston,USA;5.Rosensteil Basic Medical Sciences Research Center,Brandeis University,Waltham,USA;6.Barshop Institute for Longevity and Aging Studies,University of Texas Health Science Center,San Antonio,USA
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