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The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid
Authors:Birgit Steinmetz  Hubert Hackl  Eva Slabáková  Ilse Schwarzinger  Monika Smějová  Andreas Spittler
Affiliation:1. Department of Medicine I;2. Medical University of Vienna;3. W?hringer Gürtel, Vienna, Austria;4. Comprehensive Cancer Center;5. Biocenter;6. Division of Bioinformatics;7. Innsbruck Medical University;8. Innrain, Innsbruck, Austria;9. Department of Cytokinetics;10. Institute of Biophysics;11. Academy of Sciences of the Czech Republic;12. Královopolská, Brno, Czech Republic;13. Center of Biomolecular and Cellular Engineering;14. International Clinical Research Center;15. St. Anne's University Hospital Brno;16. Peka?ská, Brno, Czech Republic;17. Department of Laboratory Medicine;18. Masaryk University;19. Faculty of Science;20. Department of Biochemistry;21. Kotlá?ská, Brno, Czech Republic;22. Core Facility Flow Cytometry &23. Surgical Research Laboratories
Abstract:
Keywords:apoptosis  cell cycle  EVI1  all-trans retinoic acid  GDF15  gene expression profiling  myeloid differentiation
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