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Secretion of inflammatory factors from chondrocytes by layilin signaling
Authors:Kota Asano  Mitsumi Arito  Manae S. Kurokawa  Kazuki Omoteyama  Kazuki Okamoto  Naoya Suematsu  Kazuo Yudoh  Hiroshi Nakamura  Moroe Beppu  Tomohiro Kato
Affiliation:1. Clinical Proteomics and Molecular Medicine, St. Marianna University Graduate School of Medicine, 2-16-1 Sugao, Miyamae, Kawasaki, Kanagawa 216-8511, Japan;2. Disease Biomarker Analysis and Molecular Regulation, St. Marianna University Graduate School of Medicine, 2-16-1 Sugao, Miyamae, Kawasaki, Kanagawa 216-8511, Japan;3. Institute of Medical Science, St. Marianna University Graduate School of Medicine, 2-16-1 Sugao, Miyamae, Kawasaki, Kanagawa 216-8512, Japan;4. Department of Orthopedic Surgery, Nippon Medical School, 1-1-5 Sendagi, Bunkyo-ku, Tokyo 113-8603, Japan;5. Department of Orthopaedic Surgery, St. Marianna University School of Medicine, 2-16-1 Sugao, Miyamae, Kawasaki, Kanagawa 216-8511, Japan
Abstract:Layilin (LAYN) is thought to be involved in reorganization of cytoskeleton structures, interacting with merlin, radixin, and talin. Also, LAYN is known to be one of the receptors for hyaluronic acid (HA).
Keywords:Chondrocytes   Complement5   Inflammation   Layilin   Rheumatoid arthritis   TNF-α
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