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Gene-expression patterns reveal underlying biological processes in Kawasaki disease |
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| Authors: | Stephen J Popper Chisato Shimizu Hiroko Shike John T Kanegaye Jane W Newburger Robert P Sundel Patrick O Brown Jane C Burns David A Relman |
| Affiliation: | (1) Departments of Microbiology and Immunology, and Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA;(2) Department of Pediatrics, University of California San Diego School of Medicine, San Diego, CA 92123, USA;(3) Departments of Cardiology and Pediatrics, Children's Hospital Boston, and Harvard Medical School, Boston, MA 02115, USA;(4) Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305, USA;(5) Veterans Affairs Palo Alto Health Care System, Palo Alto, CA 94304, USA |
| Abstract: | BackgroundKawasaki disease (KD) is an acute self-limited vasculitis and the leading cause of acquired heart disease in children in developed countries. No etiologic agent(s) has been identified, and the processes that mediate formation of coronary artery aneurysms and abatement of fever following treatment with intravenous immunoglobulin (IVIG) remain poorly understood. |
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