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An atypical hyperacidic Grp78/BiP isoform is upregulated by TLR2 agonism or bacterial infection but not by endoplasmic reticulum stress
Authors:Cristian JA Asensio  Li Jun Tang  Stefania Zampieri  Maria G Pittis  Rodolfo C Garcia
Institution:1. Leukocyte Biology Unit, International Centre for Genetic Engineering and Biotechnology (ICGEB), Area Science Park, Padriciano 99, 34012, Trieste, Italy

Kennedy Institute of Rheumatology, Imperial College London, 1 Aspenlea Road, Hammersmith W6 8LH, London, U.K;2. Leukocyte Biology Unit, International Centre for Genetic Engineering and Biotechnology (ICGEB), Area Science Park, Padriciano 99, 34012, Trieste, Italy

Molecular Biology Research Center, School of Biological Science and Technology, Central South University, Xiangya Road 110, 410078, Changsha, Hunan, P.R.China;3. Leukocyte Biology Unit, International Centre for Genetic Engineering and Biotechnology (ICGEB), Area Science Park, Padriciano 99, 34012, Trieste, Italy

Abstract:
Keywords:
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