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Cardiac fibroblast miR-27a may function as an endogenous anti-fibrotic by negatively regulating Early Growth Response Protein 3 (EGR3)
Authors:Lifeng Teng  Yubing Huang  Jun Guo  Bin Li  Jin Lin  Lining Ma  Yudai Wang  Cong Ye  Qianqian Chen
Affiliation:1. Department of Cardiology, Hainan General Hospital, Haikou, China

Contribution: Conceptualization (equal), Data curation (equal);2. Department of Cardiology, Hainan General Hospital, Haikou, China

Contribution: Formal analysis (equal), ​Investigation (equal);3. Department of Cardiology, The First Affiliated Hospital of Jinan University, GuangZhou, China;4. Department of Cardiology, Hainan General Hospital, Haikou, China

Contribution: Data curation (equal), Formal analysis (equal), Software (equal);5. Department of Cardiology, Hainan General Hospital, Haikou, China

Contribution: Data curation (equal), Resources (equal), Validation (equal);6. Department of Cardiology, Hainan General Hospital, Haikou, China

Contribution: Formal analysis (equal), Validation (equal), Visualization (equal);7. Nursing Department, Hainan Maternal and Child Health Hospital, Haikou, China

Contribution: Data curation (equal), Resources (equal), Validation (equal)

Abstract:
Keywords:cardiac fibrosis  cardiac remodelling  EGR3  miR-27a  TGF-β
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