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| 细胞自噬与凋亡的交互作用 | |
| 引用本文： | 付晶晶,段君凯,李红. 细胞自噬与凋亡的交互作用[J]. 生命的化学, 2014, 0(5): 649-653 |
| 作者姓名： | 付晶晶 段君凯 李红 |
| 作者单位： | 江西省儿童医院中心实验室,南昌330006 |
| 基金项目： | 江西省自然科学基金项目（20114BAB215031）;江西省卫生厅科技计划项目（20141152） |
| 摘 要： | 细胞自唾又称Ⅱ型程序性细胞死亡,参与了多种疾病的发生和发展。自唆与凋亡之间存在着复杂的交互调控——二者能被多种应激刺激共同激活、共享多个调节分子,甚至互相协调转化等。全面深入研究自噬与凋亡之间的交互作用机制,将为肿瘤等疾病的认知及治疗带来突破性进展。 |
| 关 键 词： | 自遂 凋亡 交互作用 程序性细胞死亡 肺瘤 |
The crosstalk between autophagy and apoptosis |
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| FU Jingjing,DUAN Junkai,LI Hong. The crosstalk between autophagy and apoptosis[J]. Chemistry of Life, 2014, 0(5): 649-653 | |
| Authors: | FU Jingjing DUAN Junkai LI Hong |
| Affiliation: | (The Central Laboratory of Jiangxi Provincial Children＇s Hospital, Nanchang 330006, China) |
| Abstract: | Autophagy is also known as programmed contribute to the occurrence and development of many type II cell death. And autophagy has been shown to diseases. More and more studies have suggested that the molecular regulation of autophagy and apoptosis pathways are inter-connected -- numerous death stimuli are capable of activating either pathway, and both pathways share several genes, they can even transform with each other. A comprehensive study of the crosstalk between autophagy and apoptosis will bring a break- through on the cognition of the treatment of cancer and other diseases. |
| Keywords: | autophagy apoptosis crosstalk programmed cell death cancer |
| 本文献已被 CNKI 维普 等数据库收录！ | |