1996年秋帽儿山迁徙鸟类环志研究

１９９６年秋５０天连续环志鸟类２８种７７８只,红胁蓝尾鸲、黄喉、灰头３种占总量８０％。５天左右一个迁徙高峰,最高峰在迁徙期后半。今秋山丁子、黄檗树不结实,斑鸫和北朱雀甚少。原地当季重捕率１５４％。捕到去秋或今春环志鸟９只,全是黄喉。旅鸟停歇最长记录１７天。日落后半小时至１小时加一次网,能减少网上死鸟。     

帽儿山候鸟环志初报

１９９５年秋季环志斑鸫、灰背鸫、北朱雀、红胁蓝尾鸲、黄喉等候鸟１７种３９９只,原地重捕３种７只。     

吉林珲春2002～2005年鸟类环志监测报告

2002～2005年在吉林省珲春市马川子乡依力南沟开展了鸟类环志研究工作。这里为长白山低山丘陵区,是候鸟经长白山脉、乌苏里江迁徙途中重要的停歇地和食物补给站。4年共环志鸟类126种63386只,其中春季103种9761只,秋季113种53625只,隶属13目37科。春季以黄喉鸦（Emberiza elegans）为迁徙鸟类的优势种,环志2467只,占春季环志数量的25．3％;秋季以黄喉鸦、红胁蓝尾鸲（Tarsiger cyanurus）、黄眉柳莺（Phylloscopus inornatus）、大山雀（Parus major）、白眉鸦（E.tristrami）、极北柳莺（P.borealis）等6种为迁徙鸟类的优势种,累计33586只,占秋季环志数量的62．6％;通过环志研究发现,被认为是留鸟的大山雀、银喉长尾山雀（Aeghhalos caudatus）、沼泽山雀（P.palustris）、长尾雀（Uragus sibiricus）具有定期的迁徙行为。     

1996年秋帽儿山迁徒岛类环志研究

１９９６年秋５０天连续环志鸟类２８种７７８只,红胁蓝尾鸲,黄喉葵３种占总量８０％。５天左右一个迁徒高峰,最高峰在迁徒期后半。今秋山丁子,黄檗树不结实,斑鸫和北朱雀甚少。原地当季重捕率１５．４％,捕到去秋或今春环志鸟９只,全是黄喉巫。旅鸟停歇最长记录１７天。日落后半小时至１小时加遛一次网,能减少网上死鸟。     

帽儿山候鸟环环志初报

１９９５年秋季环志斑鸫,北朱雀,红胁蓝尾鸲,黄喉等候鸟１７种３９９只,原地重捕３种７只。     

帽儿山实验林场1998年春季鸟类环志报道

１９９８年３月２８日￣５月１６日（５０天）共环志鸟类１０１９只,日均２０．４只,最高日６５只,最低日６只。原地回收鸟５种８２只,是几年来回收种类、数量最多的一季。     

帽儿山1999年春季鸟类环志

1999年春季60天环志鸟类70种3111只,日均51．8只。蓝尾鸲数量在往年第一位降到第四位,田鸟升到第二位。估计黄唯鸟、灰头鸟的寿命为4－5年。在环志做法上有几点改进。     

黑龙江省鸟类新纪录——灰斑鸠

2005年秋季环志期间,捕到1只灰斑鸠,鉴定为指名亚种（Streptopelia decaocto decaocto）。该鸟量度见表1。     

新疆鸟类环志与回收

2001年8月与14－26日在新疆阿勒泰,吐鲁番的8个环志地点进行了秋际鸟类环志工作。环志鸟类233只,约33种,多以莺亚科（Sylviinae)和鸫亚科（Turdinae)的种类为主。其中的新疆歌鸲（Luscinia sp.)等17种鸟类均属于中国首次环志种类,占环志种数的53％。     

黑龙江省嫩江高峰林区2004年度鸟类环志监测报告

2004年在黑龙江省嫩江县高峰林场开展了鸟类环志研究工作,共环志鸟类100种22 194只,其中春季84种10 256只,秋季82种11 938只,隶属10目30科。春季以棕眉山岩鹨(Prunella montanella)、田(Emberiza rustica)、红胁蓝尾鸲(Tarsiger cyanurus)、燕雀(Fringilla montifringilla)、小(E.pusilla)、黄眉柳莺(Phylloscopus inornatus)、栗(E.rutila)等7种为迁徙鸟类的优势种(数量大于500只),环志7534只,占春季环志数量的73.5%;秋季以黄眉柳莺、燕雀、田、银喉长尾山雀(Aegithalos caudatus)、黄雀(Carduelis spinus)、白腰朱顶雀(C.flammea)等6种为迁徙鸟类的优势种,环志8764只,占秋季环志数量的73.4%;通过环志,发现两种黑龙江省鸟类新记录———姬鹬(Lymnocryptes minimus)、棕眉柳莺(P.armandi-i),高峰林场环志的白腰朱顶雀、红喉姬(Ficedula parva)分别被挪威、俄罗斯回收。这是欧洲国家...     

Changes in lactate dehydrogenase activity in chicken tissues in hyperthyreosis in ontogenesis

The lactate dehydrogenase activity in reactions of lactate oxidation and synthesis was studied in subfractions of the chicken brain, heart and liver at the embryonal, early postembryonal and adult stages of development after thyroxine administration. It has been shown that during embryogenesis thyroxine predominantly enhanced the rate of lactate oxidation in the mitochondrial tissues. A marked increase in the lactate synthesis was found in cytoplasm of the adult chicken tissues. Specificity of enzyme activity alterations was detected in the chicken brain during ontogenesis after thyroxine administration.     

Role for dopamine in malonate-induced damage in vivo in striatum and in vitro in mesencephalic cultures

Defects in mitochondrial energy metabolism have been implicated in the pathology of several neurodegenerative disorders. In addition, the reactive metabolites generated from the metabolism and oxidation of the neurotransmitter dopamine (DA) are thought to contribute to the damage to neurons of the basal ganglia. We have previously demonstrated that infusions of the metabolic inhibitor malonate into the striata of mice or rats produce degeneration of DA nerve terminals. In the present studies, we demonstrate that an intrastriatal infusion of malonate induces a substantial increase in DA efflux in awake, behaving mice as measured by in vivo microdialysis. Furthermore, pretreatment of mice with tetrabenazine (TBZ) or the TBZ analogue Ro 4-1284 (Ro-4), compounds that reversibly inhibit the vesicular storage of DA, attenuates the malonate-induced DA efflux as well as the damage to DA nerve terminals. Consistent with these findings, the damage to both DA and GABA neurons in mesencephalic cultures by malonate exposure was attenuated by pretreatment with TBZ or Ro-4. Treatment with these compounds did not affect the formation of free radicals or the inhibition of oxidative phosphorylation resulting from malonate exposure alone. Our data suggest that DA plays an important role in the neurotoxicity produced by malonate. These findings provide direct evidence that inhibition of succinate dehydrogenase causes an increase in extracellular DA levels and indicate that bioenergetic defects may contribute to the pathogenesis of chronic neurodegenerative diseases through a mechanism involving DA.     

Scurvy in capybaras bred in captivity in Argentine

In order to determine if the absence of vitamin C in the diet of capybaras (Hydrochoerus hydrochaeris) causes scurvy, a group of seven young individuals were fed food pellets without ascorbic acid, while another group of eight individuals received the same food with 1 g of ascorbic acid per animal per day. Animals in the first group developed signs of scurvy-like gingivitis, breaking of the incisors and death of one animal. Clinical signs appeared between 25 and 104 days from the beginning of the trial in all individuals. Growth rates of individuals deprived of vitamin C was considerably less than those observed in the control group. Deficiency of ascorbic acid had a severe effect on reproduction of another population of captive capybaras. We found that the decrease in ascorbic acid content in the diet affected pregnancy, especially during the first stages. The results obtained suggest that it is necessary to supply a suitable quantity of vitamin C in the diet of this species in captivity.     

SSTR5 ablation in islet results in alterations in glucose homeostasis in mice

Somatostatin (SST) peptide is a potent inhibitor of insulin secretion and its effect is mediated via somatostatin receptor 5 (SSTR5) in the endocrine pancreas. To investigate the consequences of gene ablation of SSTR5 in the mouse pancreas, we have generated a mouse model in which the SSTR5 gene was specifically knocked down in the pancreatic beta cells (betaSSTR5Kd) using the Cre-lox system. Immunohistochemistry analysis showed that SSTR5 gene expression was absent in beta cells at three months of age. At the time of gene ablation, betaSSTR5Kd mice demonstrated glucose intolerance with lack of insulin response and significantly reduced serum insulin levels. Insulin tolerance test demonstrated a significant increase of insulin clearance in vivo at the same age. In vitro studies demonstrated an absence of response to SST-28 stimulation in the betaSSTR5Kd mouse islet, which was associated with a significantly reduced SST expression level in betaSSTR5Kd mice pancreata. In addition, betaSSTR5Kd mice had significantly reduced serum glucose levels and increased serum insulin levels at 12 months of age. Glucose tolerance test at an older age also indicated a persistently higher insulin level in betaSSTR5Kd mice. Further studies of betaSSTR5Kd mice had revealed elevated serum C-peptide levels at both 3 and 12 months of age, suggesting that these mice are capable of producing and releasing insulin to the periphery. These results support the hypothesis that SSTR5 plays a pivotal role in the regulation of insulin secretion in the mouse pancreas.