中国大叶蝉科一新记录属及五新种(同翅目：叶蝉总科)

中国大叶蝉科一新记录属及五新种（同翅目：叶蝉总科）蔡平,葛钟麟（浙江农业大学杭州３１００２９）（安徽农业大学合肥２３００３６）透大叶蝉属ＮａｎａｔｋａＹｏｕｎｇ,１９８６,隶属于同翅目（Ｈｏｍｏｐｔｅｒａ）叶蝉总科（Ｃｉｃａｄｅｌｌｏｉｄｅａ）大叶蝉...     

大叶蝉科一新属二新种（同翅目：叶蝉总科）

本文记述大叶蝉科一新属——肖大叶蝉属Paracicadella与一新种——红条肖大叶蝉P.rubrovitta及大叶蝉属一新种——烟翅大叶蝉Cicadella fumosa。新属新种发现于西藏,大叶蝉属新种发现于云南。     

云南森林叶蝉九新种(同翅目:叶蝉总科)

本文记述的九个新种,均属于同翅目(Homoptera),分隶于四个科,即带叶蝉属(Scaphoideus Uhler,1889)与纹翅叶蝉属(Nakaharanus Ishihara,1953)属殃叶蝉科(Euscelidae);凹大叶蝉属(Bothrogonia China,1933)与笏头叶蝉属(Namsangia Distant,1908)属大叶蝉科(Tettigellidae);癫叶蝉属(Moonia Dis-tant,1908)属窄颊叶蝉科(Ulopidae),匙顶叶蝉属(Glossocratus Fieber,1866)属铲头叶蝉科(Hecalidae)。其中带叶蝉属、凹大叶蝉属与匙顶叶蝉属所包含的种数     

突大叶蝉属三新种：（同翅目：叶蝉总科）

本文记述同翅目Ｈｏｍｏｐｔｅｒａ叶蝉总科Ｃｉｃａｄｅｌｌｏｉｄｅａ大叶蝉科Ｃｉｃａｄｅｌｌｉｄａｅ突大叶蝉属Ｇｕｎｕｎｇｉｄｉａ三新种。红颜突大叶蝉Ｇｕｎｕｎｇｉｄｉａ ｒｕｎｕｎｇｉｄｉａ ｒｕｂｅｓｃｅｎｓ ｓｐ．ｎｏｖ．,锈面突大叶蝉Ｇ．ｒｕｂｉｇｉｎｏｓｅ ｓｐ．ｎｏｖ．与白翅突大叶蝉Ｇ．ａｌｂａｔａ ｓｐ．ｎｏｖ．。采自中国南方。     

斑大叶蝉属三新种：同翅目：叶蝉总科

斑大叶蝉属Anatkina Young,1986隶属于叶蝉总科Cicadelloidea,大叶蝉科Cicadellidae,大叶蝉亚科Cicadellinae,分布于东洋区。据报道全世界现有36种,我国有7种。本文报道3新种。模式标本存放于安徽农学院植保系昆虫标本室。     

福建大叶蝉科一新记录属及三新种（同翅目：叶蝉总科）

本文记述大叶蝉科１中国新记录属──暗大叶蝉属ＳｅａｓｏｇｏｎｉａＹｏｕｎｇ与１新种──红暗大叶蝉Ｓ．ｓａｎｇｕｉｎｅａＫｕｏｈｅｔｚｈｕｏ,以及条大叶蝉属ＡｔｋｉｎｓｏｎｉｅｌｌａＤｉｓｔａｎｔ２新种──黑背条大叶蝉Ａ．ｎｉｇｒｉｄｏｒｓｕｍＫｕｏｈｅｔｚｈｕｏ与红脉条大叶蝉Ａ．ｒｕｂｒｉｖｅｎｏｓａＫｕｏｈｅｔＺｈｕｏ。均采自福建。     

中国大叶蝉科一新记录属及一新种一新组合（同翅目：叶蝉总科）

记述大叶科１中国新记录属－长冠叶蝉属Ｓｔｅｎａｔｋｉｎａ Ｙｏｕｎｇ及１新种－双斑长冠叶蝉Ｓｔｅｎａｔｋｉｎａ ｂｉｍａｃｕｌａｔａ,ｓｐ,ｎｏｖ．和１新种组合－角突长冠叶蝉Ｓｔｅｎａｔｋｉｎａ ａｎｇｕｓｔａｔａ,ｃｏｍｂ,ｎｏｖ．模式标本保存在贵州大学昆虫研究所。     

中国边大叶蝉属的分类研究 (同翅目：大叶蝉科

研究了中国大叶蝉科边大叶蝉属Kolla Distant,描述了3新种：五斑边大叶蝉K.pentistigma新种、黑条边大叶蝉K.nigrifascia新种、棒突边大叶蝉K.rhabdoma新种,编制出种检索表。模式标本保存在贵州大学昆虫研究所。     

云南条大叶蝉属五新种一新纪录：同翅目：叶蝉总科：大叶蝉科

本文记述大叶蝉科条大叶蝉属ＡｔｋｉｎｓｏｎｉｅｌｌａＤｉｓｔａｎｔ５新种及一中国新纪录种：黄条大叶蝉Ａ．ｘａｎｔｈｏｖｉｔｔａ、黄斑条大叶蝉Ａ．ｘａｎｔｈｏｎｏｔａ、黑体条大叶蝉Ａ．ｎｉｇｒａ、红斑条大叶蝉Ａ．ｒｕｂｒａ、污黄条大叶蝉Ａ．ｌａｃｔｅａ及披纹条大叶蝉Ａ．ｍａｌａｓｉｅｉＹｏｕｎ。标本采自我国云南省。文中描述了新种、新纪录种的外部形态和雄性外生殖器构造特征,以及与近似种的区别。     

福建大叶蝉科-新记录属及三新种（同翅目：叶蝉总科）

本记述大叶蝉科1中国新记录属——晴大叶蝉属Seasogonin与1新种——红暗大叶蝉S．sanguinea Kuoh et Zhuo,及条大叶蝉属Atkinomieffa Distant新种——黑背条大叶蝉A．nigridorsurn Kuoh et Zhuo与红脉条大叶蝉A．rubricerosa Kuoh et Zhuo。均采自福建。     

A homozygous female hemophilia A

BACKGROUND:

Hemophilia A (HA), being an X-linked recessive disorder, females are rarely affected, although they can be carriers.

AIMS:

To study the mutation in F8 gene in an extended family with a homozygous female HA.

MATERIALS AND METHODS:

All the seven affected members (six males and one female) were initially screened by Conformation Sensitive Gel Electrophoresis (CSGE) and direct DNA sequencing.

RESULTS:

A homozygous missense mutation c.1315G>A (p.Gly420Ser) was identified in exon 9 of F8 gene in homozygous state in the affected female born of 1° consanguinous marriage and in all the affected male members of the family. Her factor VIII levels was found to be 5.5%, vWF:Ag 120%.

CONCLUSION:

In India, as consanguineous marriages are very common in certain communities (up to 30%), the likelihood of encountering female hemophilia is higher, although this is the first case of HA out of 1600 hemophilia families registered in our Comprehensive Haemophilia Care Center. Genetic diagnosis in such cases is not necessary as all the male children will be affected and daughters obligatory carriers.     

Aminopeptidase A is angiotensinase A

Summary Quantitative histochemical measurements of aminopeptidase A (APA; E.C.3.4.11.7) were done kinetically in the kidney glomeruli of rat and mouse with an instrumental setup consisting of a microdensitometer and a computer-supported morphometric system. The histochemical demonstration of APA was carried out using the simultaneous azo coupling technique (purest-grade Fast Blue B as coupling agent and -l-glutamic acid-4-methoxy-2-naphthylamide as substrate). The methodological studies show that APA activity is calcium-ion-dependent and increases linearly with the thickness of the tissue section (3–12 m) and that the time-course of APA activity as determined by linear regression is linear only for the first 1 to 2 min of the reaction. — Kinetic measurements indicate a 40% decrease in APA activities when -l-glutamic acid-4-methoxy-2-naphthylamide (-l-Glu-MNA) is replaced by -l-aspartic acid-4-methoxy-2-naphthylamide. When -l-Glu-MNA is replaced with l-alanine-4-methoxy-2-naphthylamide, which is a substrate of aminopeptidase M (APM) only very low reaction rates are measurable (about 1.4% of those with -l-Glu-MNA). 100 and 130 mM NaCl in the incubation medium increase APA activities by approximately 16%–17%. — To clarify the functional importance of APA in the kidney, their activities were measured under the influence of angiotensins. The glomerulus was selected as the measuring site, for besides APA it contains no APM or other peptidases that could degrade angiotensins (the glomerular dipeptidyl peptidase IV is not inhibited by angiotensin II). Using the Lineweaver-Burk plot, we determined a K _m of 0.16 mM for the APA in rat glomeruli and 0.14 mM in mouse glomeruli. The V _max in mouse glomeruli is 1.6 times higher than in rat glomeruli. Ang iotensin I, II and III competitively inhibit APA in the rat and mouse glomeruli. — With quantitative histochemical techniques it was possible to show that APA is equivalent to angiotensinase A (splitting off the N-terminal aspartic acid from angiotensin I and II).Supported by the Deutsche Forschungsgemeinschaft (SFB 105)     

A glucoside of urospermal A

The aerial parts of Urospermum picroides afforded, in addition to urospermal A a p-hydroxylphenyl acetate of a glucoside of urospermal A.     

Quadruplexes of human telomere DNA analogs designed to contain G:A:G:A,G:G:A:A,and A:A:A:A tetrads

Replacement of two to four guanines by adenines in the human telomere DNA repeat dG₃(TTAG₃)₃ did not hinder the formation of quadruplexes if the substitutions took place in the terminal tetrad bridged by the diagonal loop of the intramolecular antiparallel three‐tetrad scaffold, as proved by CD and PAGE in both Na⁺ and K⁺ solutions. Thermodynamic data showed that, in Na⁺ solution, the dG₃(TTAG₃)₃ quadruplex was destabilized, the least by the two G:A:G:A tetrads, the most by the G:G:A:A tetrad in which the adenosines replaced syn‐guanosines. In physiological K⁺ solution, the highest destabilization was caused by the 4A tetrad. In K⁺, only the unmodified dG₃(TTAG₃)₃ quadruplex rearranged into a K⁺‐dependent quadruplex form, none of the multiple adenine‐modified structures did so. This may imply biological consequences for nonrepaired A‐for‐G mutations. © 2010 Wiley Periodicals, Inc. Biopolymers 93: 880–886, 2010.