新疆倍叉(虫责)属一新种记述(襀翅目:叉(虫责)科)

本文记述了采自新疆喀纳斯的倍叉[虫责]属Amphinemura 1新种：环齿倍叉[虫责]Amphinemura tricintusidens Wang＆Zhu,sp．nov。根据肛侧突中叶端部有3或4个环绕的小齿和肛上突背骨片端部形成的圆形腔可将该新种与其它种类相区别。正模♂,新疆喀纳斯,2005-Ⅷ—01,朱江艳等采。副模1♂,采集资料同正模。新种的模式标本保存在扬州大学昆虫标本馆。     

中国叉(虫责)属二新种(襀翅目,叉(虫责)科)

记述我国叉(虫责)属Nemoura 2新种:吉林叉(虫责)N.ijlinensis sp.nov.、妙峰山叉(虫责)N.miaofengshanensis sp.nov.,描述了其形态特征并与近似种做了比较.     

中国倍叉Zhe属三新种（Zhe翅目：叉Zhe科）

记述我国倍叉Zhe属Amphinemura 3新种：尖突倍叉Zhe Amphinemura acutata sp.nov.、双突倍叉Zhe Amphinemura didyma sp.nov.、单突倍叉ZheAmphinemura singularis sp.nov.。     

西藏倍叉属(襀翅目:叉科)三新种(英文)

记述我国西藏倍叉属Amphinemura 3新种 :杨氏倍叉Amphinemurayangisp .nov .(模式产地 :鲁郎 )、李氏倍叉Amphinemuraliisp .nov .(模式产地 :米林 )、西藏倍叉Amphinemuratibetensissp .nov .(模式产地 :波密 )。杨氏倍叉A .yangisp .nov .与a .wittmeriZwick&Sivec近似 ,但肛侧突中叶末端小而缘且有 3— 5个粗刺 ;李氏倍叉A .liisp .nov .与A .lebeziSivec近似 ,但肛侧突内叶不对称 ;西藏倍叉A .tibetensissp .nov .与江苏倍A .kiangsiensis(Wu)近似 ,但内叶短于中叶且末端呈 2瓣状。模式标本存中国农业大学昆虫标本馆。     

中国瘤(虫责)属一新种记述(襀翅目:(虫责)科)

记述采自中国陕西佛龙的瘤(虫责)属Tyloperla 1新种:双凹瘤(虫责)Tyloperla bihypodroma Du, sp. nov.,该种与尖突瘤(虫责)Tyloperla attenuata很相近,但前者第8背板近后缘形成1个隆起脊、其中部有1凹陷,后者则在背板中部形成1近圆形的隆起、其上的锥状感觉器比前者多;前者第9背板中央微凹、其前缘骨化明显、极小的锥状感觉器稀少,后者中部有1丛明显的锥状感觉器;前者翅透明,后者翅微烟褐色.新种的模式标本保存在扬州大学昆虫标本馆.     

贵州钩(虫责)属一新种记述((襀)翅目:(虫责)科)

记述采自贵州宽阔水保护区的钩(虫责)属l新种:巨刺钩(虫责)Kamimuria grandispinata Du ＆Sun,sp.nov.,该新种的阳茎特征与长刺钩虫责K.longispina Wu相似,但长刺钩(虫责)阳茎囊背面近端部有1膜质突起,上面生有粒状微刺,而新种阳茎囊背面近端部是1个近似三角形的刺斑,无突起...     

贵州新虫责属二新种记述1(襀翅目:虫责科:虫责亚科)

记述采自中国贵州的新虫责属 2新种 ,即双瘤新虫责 Neoperla bituberculata,sp.nov.和曲囊新虫责 N eoperla flexiscrotata,sp.nov.。模式标本存扬州大学植物保护系昆虫标本室 ( YU)     

中国缺叉多距石蛾属六新种记述(毛翅目,多距石蛾科)

研究了中国毛翅目多距石蛾科缺叉多距石蛾属种类,并报道了其中亲缘关系较近的6新种,即端截缺叉多距石蛾Polyplectropus truncatufus sp.nov.,端突缺叉多距石蛾Polyplectropus convexus sp.nov.,斧端缺叉多距石蛾Polyplectropus dolabriformis sp.nov.,颚状缺叉多距石蛾Polyplectropus mandibuliformis sp.nov.,距枝缺叉多距石蛾Polyptectropus rectangulus sp.nov.,喙端缺叉多距石蛾Polyplectropus rostriformis sp.nov..新种模式标本保存在南京农业大学昆虫标本馆.     

中国钮虫责属种类记述(襀翅目:鰿科:钮虫责亚科)

记述中国钮虫责属 Acroneuria Pictet昆虫 4种 ,其中包括 2新种 :莫氏钮虫责Acroneuria morsei,sp.nov.和多锥钮虫责 Acroneuria multiconata,sp.nov.。模式标本存扬州大学植物保护系昆虫标本室 ( YU)和西北农林科技大学昆虫博物馆 ( NW-STUAF)。     

四川诺(虫责)属(襀翅目,卷(虫责)科)一新种记述

记述采自四川1新种,李氏诺Rhopalopsole lii sp.nov.,并讨论了其与近似种的区别.模式标本保存在中国农业大学标本馆.     

Entwicklungsgeschichte und Ultrastruktur von Pollenkitt und Exine bei nahe verwandten entomophilen und anemophilen Angiospermensippen derAlismataceae,Liliaceae, Juncaceae,Cyperaceae, Poaceae undAraceae

Some closely related members of the monocotyledonous familiesAlismataceae, Liliaceae, Juncaceae, Cyperaceae, Poaceae andAraceae with variable modes of pollination (insect- and wind-pollination) were studied in relation to the ultrastructure of pollenkitt and exine (amount, consistency and distribution of pollenkitt on the surface of pollen grains). The character syndromes of pollen cementing in entomophilous, anemophilous and intermediate (ambophilous or amphiphilous) monocotyledons are the same in principal as in dicotyledons. Comparing present with former results one can summarize: 1) The pollenkitt is always produced in the same manner by the anther tapetum in all angiosperm sub-classes. 2) The variable stickiness of entomophilous and anemophilous pollen always depends on the particular distribution and consistency of the pollenkitt, but not its amount on the pollen surface. 3) The mostly dry and powdery pollen of anemophilous plants always contains a variable amount of inactive pollenkitt in its exine cavities. 4) A step-by step change of the pollen cementing syndrome can be observed from entomophily towards anemophily. 5) From the omnipresence of pollenkitt in all wind-pollinated angiosperms studied one can conclude that the ancestors of anemophilous angiosperms probably have been zoophilous (i.e. entomophilous) throughout.

     

Identification and Characterization of the First Equine Parainfluenza Virus 5

正Dear Editor,Parainfluenza virus 5 (PIV5), known as canine parainfluenza virus in the veterinary field, is a negative-sense,nonsegmented, single-stranded RNA virus belonging to the Paramyxoviridae family (Chen 2018). The virus was first reported in primary monkey kidney cells in 1954 (Hsiung1972), then it has been frequently discovered in various     

Pathogenic Characterization and Full Length Genome Sequence of a Reassortant Infectious Bursal Disease Virus Newly Isolated in Pakistan

<正>Dear Editor,Infectious bursal disease (IBD) is one of the most important diseases of the poultry. The IBD virus (IBDV), a nonenveloped virus belonging to the Birnaviridae family with a genome consisting of two segments of double-stranded RNA (segments A and B), targets B lymphocytes of bursa of Fabricious leading to immunosuppression. In Pakistan,poultry farming is the second biggest industry and IBD is the second biggest disease threating the poultry sector.However, there is limited genome information of IBDV     

Mink Circovirus Can Infect Minks,Foxes and Raccoon Dogs

正Dear Editor,Mink circovirus (MiCV), which is clustered in the genus Circovirus of the family Circoviridae, was first described in minks from farms in Dalian, China in 2013 (Lian et al.2014). The complete single-stranded circular genome of the virus is 1,753 nucleotides long and contains two major open reading frames (ORFs), designated ORF1 (Rep gene)and ORF2 (Cap gene)(Lian et al. 2014; Ge et al. 2018).Sequence analysis has shown that MiCV is most closely     

CypA Regulates AIP4-Mediated M1 Ubiquitination of Influenza A Virus

Cyclophilin A (CypA) is a peptidyl-prolyl cis/trans isomerase that interacts with the matrix protein (M1) of influenza A virus (IAV) and restricts virus replication by regulating the ubiquitin–proteasome-mediated degradation of M1. However,the mechanism by which CypA regulates M1 ubiquitination remains unknown. In this study, we reported that E3 ubiquitin ligase AIP4 promoted K48-linked ubiquitination of M1 at K102 and K104, and accelerated ubiquitin–proteasome-mediated degradation of M1. The recombinant IAV with mutant M1 (K102 R/K104 R) could not be rescued, suggesting that the ubiquitination of M1 at K102/K104 was essential for IAV replication. Furthermore, CypA inhibited AIP4-mediated M1 ubiquitination by impairing the interaction between AIP4 and M1. More importantly, both the mutations of M1 (K102 R/K104 R) and CypA inhibited the nuclear export of M1, indicating that CypA regulates the cellular localization of M1 via inhibition of AIP4-mediated M1 ubiquitination at K102 and K104, which results in the reduced replication of IAV.Collectively, our findings reveal a novel ubiquitination-based mechanism by which CypA regulates the replication of IAV.