河北省有瓣蝇类研究(一)(双翅目:蝇科)

整理近年来采自河北省蝇科标本,发现阳蝇属Helina R.-D.二新种和棘蝇属Phaonia R.-D一新种,特此描述。模式标本存于沈阳师范学院生物系。 1.黑古阳蝇Helina nigricnnosa Xue et Zhao,新种(图1—4)     

新疆阳蝇属四新种(双翅目:蝇科)

报告采自新疆蝇科阳蝇属Helina R.-D.4新种,即眼鬃阳蝇Helina ocellijuba sp.nov.,粘叶阳蝇Helina mallocerca sp.nov.,狭跗阳蝇Helina stenotarsis sp.nov.和板刺阳蝇Helina sterniteoacaena sp.nov.。模式标本保存于中国科学院动物研究所。     

青海省阳蝇属四新种(双翅目,蝇科)

报告采自青海省蝇科阳蝇属Helina R—D．4新种,分别命名为高居阳蝇Helina alpigenus sp．nov．,尖尾阳蝇Helina apicicauda sp．nov．,二刺阳蝇Helina bispina sp．nov．和大毁阳蝇Helina largideleta sp．nov．。模式标本保存于中国科学院动物研究所。     

中国四川省阳蝇属五新种(双翅目:蝇科)

报道采自中国四川省阳蝇属Helina R．-D．,1830 5新种：腹刺阳蝇Helina sternitoscola,sp.nov．;华西阳蝇Helina sinoccidentala,sp．nov．;亚蜜阳蝇Helina subfica,sp．nov．;双叉阳蝇Helina didicrocerca,sp．nov．：四川阳蝇Helina sichuanica,sp．nov．。前4种模式标本保存在中国科学院上海生命科学研究院植物生理生态研究所：最后1种保存于北京军事医学科学院微生物流行病研究所医学昆虫标本馆。     

四川省阳蝇属五新种（双翅目：蝇科）

记述采自中国四川西部山区阳蝇属Helina R.-D.,1980的5新种,分别命名为金阳蝇Helina aureolicolorata,sp.nov.;羽胫阳蝇Helina plumipostitibia,sp.nov.;茂汶阳蝇Helina maowenna,sp.nov.;雨阳蝇Helina hyeta,sp.nov.;类介阳蝇Helina mimintermedia,sp.nov.。模式标本存北京军事医学科学院微生物流行病研究所医学昆虫标本馆。     

中国阳蝇属国上新种（双翅目：蝇科）

报道采自黑龙江和甘两省的蝇科阳蝇属Helina R.-D.4新各上,分别命名为胡氏阳蝇Helina huae sp.nov.,小兴安阳蝇Heliona xiaoxinganna sp.nov.,舟曲阳蝇Helina zhouquensis sp.nov和圆板阳蝇Helina ampyxocerca sp.nov.。模式标本保存于沈阳师范大学昆虫研究所。     

长白山阳蝇属三新种(双翅目:蝇科)

报道采自吉林省长白山蝇科阳蝇属Helina R.-D.3新种,即密胡阳蝇Helina densibarbata sp.nov。,吉林阳蝇Helina jilinensis sp.nov.和小毁阳蝇Helina minutideleta sp.nov.,模式标本保存于沈阳师范大学昆虫研究所。     

新疆阳蝇属Helina R.—D.二新种（双翅目：蝇科）

记述采自新疆蝇属Helina R.-D．2新种,托木尔阳蝇Helina tuomuerra,sp,nov。和新疆阳蝇Helina xinjiangensis,sp.nov.模式标本保存于中国科学院动物研究所。     

中国四川阳蝇属五新种(双翅目:蝇科)

报告采自四川西部山区的蝇科阳蝇属HelinaR.-D.五新种,即：亚喜密阳蝇Helina subevecta sp.nov,阳阳蝇Helina solata sp.nov,用公山阳蝇Helina zhougongshanna sp.nov,康定阳蝇Helina kangdingen-sis sp.nov和赘脉阳蝇Helina appendicivena sp.nov。模式标本保存于中国军事医学科学院微生物流行病研究所医学昆虫标本馆。     

中国阳蝇属四新种(双翅目: 蝇科)

报道采自黑龙江和甘肃两省的蝇科阳蝇属Helina R. -D. 残轮?分别命名为胡氏阳蝇Helina huae sp. nov., 小兴安阳蝇Heliona xiaoxinganna sp. nov., 舟曲阳蝇Helina zhouquensis sp. nov. 和圆板阳蝇Helina ampyxocerca sp. nov..模式标本保存于沈阳师范大学昆虫研究所.     

Shapes of curves of pH-dependence of reactions

A simple case is considered in which the rate of a two-step reaction depends on pH because the intermediate formed in the first step has to gain (or lose) a proton before it can react in the second step, and in which the rate-determining step therefore changes with pH. The curves of reaction rate against pH are shown to be symmetrical, and the sharpest peak possible has a width at half its height of 1.53pH units, i.e. of 2log(3+2 radical2). Any particular curve for this situation proves to be identical with a curve that could be generated for the pH-dependence of a single-step reaction in which the rate is proportional to the concentration of a particular ionic form of a reactant. Curves for the latter situation, however, can have forms impossible for the former case in which the rate-determining step changes, but only if the protonations that activate and deactivate the reactant are co-operative. The peak can then become even sharper, and its width at half its height can fall to 1.14pH units, i.e. to 2log(2+ radical3).     

Overview of mechanisms of action of lycopene

Dietary intakes of tomatoes and tomato products containing lycopene have been shown to be associated with decreased risk of chronic diseases such as cancer and cardiovascular diseases in numerous studies. Serum and tissue lycopene levels have also been inversely related to the risk of lung and prostate cancers. Lycopene functions as a very potent antioxidant, and this is clearly a major important mechanism of lycopene action. In this regard, lycopene can trap singlet oxygen and reduce mutagenesis in the Ames test. However, evidence is accumulating for other mechanisms as well. Lycopene at physiological concentrations can inhibit human cancer cell growth by interfering with growth factor receptor signaling and cell cycle progression specifically in prostate cancer cells without evidence of toxic effects or apoptosis of cells. Studies using human and animal cells have identified a gene, connexin 43, whose expression is upregulated by lycopene and which allows direct intercellular gap junctional communication (GJC). GJC is deficient in many human tumors and its restoration or upregulation is associated with decreased proliferation. The combination of low concentrations of lycopene with 1,25-dihydroxyvitamin D3 exhibits a synergistic effect on cell proliferation and differentiation and an additive effect on cell cycle progression in the HL-60 promyelocytic leukemia cell line, suggesting some interaction at a nuclear or subcellular level. The combination of lycopene and lutein synergistically interact as antioxidants, and this may relate to specific positioning of different carotenoids in membranes. This review will focus on the growing body of evidence that carotenoids have unexpected biologic effects in experimental systems, some of which may contribute to their cancer preventive properties in models of carcinogenesis. Consideration of solubility in vitro, comparison with doses achieved in humans by dietary means, interactions with other phytochemicals, and other potential mechanisms such as stimulation of xenobiotic metabolism, inhibition of cholesterogenesis, modulation of cyclooxygenase pathways, and inhibition of inflammation will be considered. This review will point out areas for future research where more evidence is needed on the effects of lycopene on the etiology of chronic disease.     

Determination of stoichiometry of radioiodination of proteins

A sensitive method for the detection of small quantities of hydrophobic antioxidant free radical scavengers such as butylatedhydroxytoluene (BHT) and butylatedhydroxyanisole (BHA) in aqueous samples is described. The procedure involves extraction of the hydrophobic free radical scavenger into an organic solvent phase, followed by the subsequent reaction of an aliquot of this extract with the stable cation radical tris(p-bromophenyl)amminium hexachloroantimonate (TBACA). In experiments with BHT and BHA, the loss of TBACA absorbance at 730 nm was found to be linearly proportional to the amount of antioxidant added, with quantities of BHT as small as 200 pmol being easily detectable. In aqueous suspensions of dimyristoylphosphatidylcholine vesicles, assays of the aqueous BHT concentration showed that BHT partitioned strongly into the membrane phase, achieving very high BHT/phospholipid ratios. For a given concentration of BHT, partitioning into the membrane phase was greater in large, multilamellar liposomes than in either small, single-walled vesicles or in purified rat brain synaptic vesicle membranes. Direct assay of BHT and BHA in phospholipid membranes, however, was complicated by a nonspecific interaction between TBACA and the phospholipid.